PU. 1 is not essential for early myeloid gene expression but is required for terminal myeloid differentiation.
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The LKLF transcription factor is required for normal tunica media formation and blood vessel stabilization during murine embryogenesisPhysical interactions between Ets and NF-kappaB/NFAT proteins play an important role in their cooperative activation of the human immunodeficiency virus enhancer in T cellsMicroglia development and functionPU.1 is essential for p47(phox) promoter activity in myeloid cellsSpi-B can functionally replace PU.1 in myeloid but not lymphoid developmentTissue macrophages act as cellular chaperones for vascular anastomosis downstream of VEGF-mediated endothelial tip cell induction.Pearson correlation analysis of microarray data allows for the identification of genetic targets for early B-cell factor.Marrow stem cells shift gene expression and engraftment phenotype with cell cycle transitLevel of RUNX1 activity is critical for leukemic predisposition but not for thrombocytopenia.Identification of markers that distinguish monocyte-derived fibrocytes from monocytes, macrophages, and fibroblastsDownregulation of PU.1 leads to decreased expression of Dectin-1 in alveolar macrophages during Pneumocystis pneumonia.Normal myeloid development requires both the glutamine-rich transactivation domain and the PEST region of transcription factor PU.1 but not the potent acidic transactivation domain.BSAP can repress enhancer activity by targeting PU.1 function.Defective B cell receptor-mediated responses in mice lacking the Ets protein, Spi-B.PU.1 regulates both cytokine-dependent proliferation and differentiation of granulocyte/macrophage progenitors.Innate immune regulation by STAT-mediated transcriptional mechanisms.Activated Fes protein tyrosine kinase induces terminal macrophage differentiation of myeloid progenitors (U937 cells) and activation of the transcription factor PU.1.Murine hematopoietic stem cells and multipotent progenitors express truncated intracellular form of c-kit receptor.Multilineage embryonic hematopoiesis requires hypoxic ARNT activityFunctional PU.1 in macrophages has a pivotal role in NF-κB activation and neutrophilic lung inflammation during endotoxemia.PML-retinoic acid receptor alpha inhibits PML IV enhancement of PU.1-induced C/EBPepsilon expression in myeloid differentiation.Signaling networks that control the lineage commitment and differentiation of bone cells.FES-Cre targets phosphatidylinositol glycan class A (PIGA) inactivation to hematopoietic stem cells in the bone marrowInterferon (IFN) consensus sequence-binding protein, a transcription factor of the IFN regulatory factor family, regulates immune responses in vivo through control of interleukin 12 expression.The Ets transcription factor ERM is Th1-specific and induced by IL-12 through a Stat4-dependent pathway.Cross-lineage expression of Ig-beta (B29) in thymocytes: positive and negative gene regulation to establish T cell identity.Nuclear receptor steroidogenic factor 1 directs embryonic stem cells toward the steroidogenic lineagespib is required for primitive myeloid development in XenopusExpression of Scl in mesoderm rescues hematopoiesis in the absence of Oct-4Identification of the microRNA networks contributing to macrophage differentiation and functionThe role of PU.1 and GATA-1 transcription factors during normal and leukemogenic hematopoiesis.Macrophage polarization: an opportunity for improved outcomes in biomaterials and regenerative medicineRegulation of monocyte differentiation by specific signaling modules and associated transcription factor networks.Macrophage subsets and osteoimmunology: tuning of the immunological recognition and effector systems that maintain alveolar bone.Transcriptional regulation of the stem cell leukemia gene by PU.1 and Elf-1.The transcription factor PU.1 is involved in macrophage proliferationNext generation transcriptomics and genomics elucidate biological complexity of microglia in health and disease.Macrophage heterogeneity in the context of rheumatoid arthritis.A developmentally controlled competitive STAT5-PU.1 DNA binding mechanism regulates activity of the Ig κ E3' enhancer.Use of RDA analysis of knockout mice to identify myeloid genes regulated in vivo by PU.1 and C/EBPalpha
P2860
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P2860
PU. 1 is not essential for early myeloid gene expression but is required for terminal myeloid differentiation.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh-hant
name
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@en
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@nl
type
label
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@en
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@nl
prefLabel
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@en
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@nl
P2093
P1433
P1476
PU. 1 is not essential for ear ...... minal myeloid differentiation.
@en
P2093
P304
P356
10.1016/1074-7613(95)90060-8
P407
P577
1995-12-01T00:00:00Z