about
Grayanotoxin poisoning: 'mad honey disease' and beyondA systematic evaluation of protein kinase A-A-kinase anchoring protein interaction motifs.The anti-protozoal drug pentamidine blocks KIR2.x-mediated inward rectifier current by entering the cytoplasmic pore region of the channel.Foetal and adult cardiomyocyte progenitor cells have different developmental potentialRobust anti-arrhythmic efficacy of verapamil and flunarizine against dofetilide-induced TdP arrhythmias is based upon a shared and a different mode of action.Dominant missense mutations in ABCC9 cause CantĂș syndrome.Comparison of the IKr blockers moxifloxacin, dofetilide and E-4031 in five screening models of pro-arrhythmia reveals lack of specificity of isolated cardiomyocytesMolecular aspects of adrenergic modulation of cardiac L-type Ca2+ channels.A small novel A-kinase anchoring protein (AKAP) that localizes specifically protein kinase A-regulatory subunit I (PKA-RI) to the plasma membrane.Molecular aspects of adrenergic modulation of the transient outward current.Fraud and misconduct in science: the stem cell seduction: Implications for the peer-review process.Selectivity in enrichment of cAMP-dependent protein kinase regulatory subunits type I and type II and their interactors using modified cAMP affinity resins.Rewiring the heart: stem cell therapy to restore normal cardiac excitability and conduction.Cardiac cell-cell junctions in health and disease: Electrical versus mechanical coupling.Regulatory roles of the ubiquitin-proteasome system in cardiomyocyte apoptosis.Toward specific cardiac I(K1) modulators for in vivo application: old drugs point the way.Beat-to-beat variability of repolarization as a new biomarker for proarrhythmia in vivo.Inhibition of cardiac inward rectifier currents by cationic amphiphilic drugs.Cardiac ion channel trafficking defects and drugs.Barium toxicity and the role of the potassium inward rectifier current.A 2015 focus on preventing drug-induced arrhythmias.Pharmacological exploration of the resting membrane potential reserve: Impact on atrial fibrillation.A Heart too Drunk to Drive; AV Block following Acute Alcohol Intoxication.Epigenetics: DNA demethylation promotes skeletal myotube maturation.Human cardiomyocyte progenitor cell-derived cardiomyocytes display a maturated electrical phenotype.Cardiomyocytes from human and mouse embryonic stem cells.Connexin43 repression following epithelium-to-mesenchyme transition in embryonal carcinoma cells requires Snail1 transcription factor.Regulated expression of the X. tropicalis connexin43 promoter.Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent.Drugs and trafficking of ion channels: a new pro-arrhythmic threat on the horizon?Inhibiting the clathrin-mediated endocytosis pathway rescues K(IR)2.1 downregulation by pentamidine.Lysosome mediated Kir2.1 breakdown directly influences inward rectifier current density.Maximal epidermal growth-factor-induced cytosolic phospholipase A2 activation in vivo requires phosphorylation followed by an increased intracellular calcium concentration.Class III antiarrhythmic drugs amiodarone and dronedarone impair KIR 2.1 backward trafficking.The actin binding domain of the epidermal growth factor receptor is required for EGF-stimulated tissue invasion.Identification of an intracellular domain of the EGF receptor required for high-affinity binding of EGF.Epidermal growth factor-induced activation and translocation of c-Src to the cytoskeleton depends on the actin binding domain of the EGF-receptor.Anesthesia and arrhythmogenesis in the chronic atrioventricular block dog model.Insights in KIR2.1 channel structure and function by an evolutionary approach; cloning and functional characterization of the first reptilian inward rectifier channel KIR2.1, derived from the California kingsnake (Lampropeltis getula californiae).Reduced plakoglobin immunoreactivity in arrhythmogenic cardiomyopathy: methodological considerations.
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description
researcher
@en
wetenschapper
@nl
name
M A van der Heijden
@en
M A van der Heijden
@nl
type
label
M A van der Heijden
@en
M A van der Heijden
@nl
prefLabel
M A van der Heijden
@en
M A van der Heijden
@nl
P106
P31
P496
0000-0002-4225-7942