about
The mechanistic classification of addictive drugsGroup 1 mGluR-dependent synaptic long-term depression: mechanisms and implications for circuitry and diseaseDrug-evoked synaptic plasticity causing addictive behaviorCell-Type Specific Insertion of GluA2-Lacking AMPARs with Cocaine Exposure Leading to Sensitization, Cue-Induced Seeking, and Incubation of Craving.Optogenetically inspired deep brain stimulation: linking basic with clinical researchDrug-driven AMPA receptor redistribution mimicked by selective dopamine neuron stimulationSHANK3 controls maturation of social reward circuits in the VTANeural bases for addictive properties of benzodiazepines.D1R/GluN1 complexes in the striatum integrate dopamine and glutamate signalling to control synaptic plasticity and cocaine-induced responsesEmerging roles for G protein-gated inwardly rectifying potassium (GIRK) channels in health and disease.Methamphetamine-evoked depression of GABA(B) receptor signaling in GABA neurons of the VTA.Absence and rescue of morphine withdrawal in GIRK/Kir3 knock-out mice.Firing modes of dopamine neurons drive bidirectional GIRK channel plasticity.Synaptic plasticity and addiction: learning mechanisms gone awry.Drug-evoked plasticity: do addictive drugs reopen a critical period of postnatal synaptic development?Cocaine-evoked synaptic plasticity of excitatory transmission in the ventral tegmental area.Pathological circuit function underlying addiction and anxiety disorders.Temporally precise labeling and control of neuromodulatory circuits in the mammalian brain.Optogenetic dissection of neural circuitry: from synaptic causalities to blue prints for novel treatments of behavioral diseases.VTA Projection Neurons Releasing GABA and Glutamate in the Dentate Gyrus.NMDA receptor-dependent long-term potentiation and long-term depression (LTP/LTD).Bi-directional effect of increasing doses of baclofen on reinforcement learning.GABA neurons of the VTA drive conditioned place aversion.Cocaine-evoked synaptic plasticity: persistence in the VTA triggers adaptations in the NAc.Glutamate receptors on dopamine neurons control the persistence of cocaine seeking.[Impulse control disorders and Parkinson's disease]Cocaine-evoked synaptic plasticity: a key to addiction?RGS2 modulates coupling between GABAB receptors and GIRK channels in dopamine neurons of the ventral tegmental area.Ribosomal Protein S6 Phosphorylation Is Involved in Novelty-Induced Locomotion, Synaptic Plasticity and mRNA Translation.Hippocampal Somatostatin Interneurons Control the Size of Neuronal Memory Ensembles.Cocaine Exposure Enhances the Activity of Ventral Tegmental Area Dopamine Neurons via Calcium-Impermeable NMDARs.Convergence of Reinforcing and Anhedonic Cocaine Effects in the Ventral Pallidum.Sufficiency of Mesolimbic Dopamine Neuron Stimulation for the Progression to Addiction.Addiction therapy. Refining deep brain stimulation to emulate optogenetic treatment of synaptic pathology.Short pulse width in subthalamic stimulation in Parkinson's disease: a randomized, double-blind study.Expression of cocaine-evoked synaptic plasticity by GluN3A-containing NMDA receptors.Cocaine disinhibits dopamine neurons by potentiation of GABA transmission in the ventral tegmental area.Dominique Muller (1956–2015).A deeply superficial brain stimulation.Ventral tegmental area GABA projections pause accumbal cholinergic interneurons to enhance associative learning.
P50
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P50
description
investigador
@es
researcher
@en
name
Christian Lüscher
@en
Christian Lüscher
@nl
type
label
Christian Lüscher
@en
Christian Lüscher
@nl
prefLabel
Christian Lüscher
@en
Christian Lüscher
@nl
P31
P496
0000-0001-7917-4596