about
Central chronic apelin infusion decreases energy expenditure and thermogenesis in mice.Regulation of skeletal muscle lipolysis and oxidative metabolism by the co-lipase CGI-58Skeletal muscle mitochondrial capacity and insulin resistance in type 2 diabetesMyotubes from severely obese type 2 diabetic subjects accumulate less lipids and show higher lipolytic rate than myotubes from severely obese non-diabetic subjects.Beta-adrenergic and atrial natriuretic peptide interactions on human cardiovascular and metabolic regulationNatriuretic peptides enhance the oxidative capacity of human skeletal muscle.Exercise-like effects by Estrogen-related receptor-gamma in muscle do not prevent insulin resistance in db/db mice.Determinants of intramyocellular triglyceride turnover: implications for insulin sensitivity.G0/G1 Switch Gene 2 controls adipose triglyceride lipase activity and lipid metabolism in skeletal muscleControl of lipolysis by natriuretic peptides and cyclic GMP.Association of β-2 adrenergic agonist and corticosteroid injection in the treatment of lipomas.Perilipin 5 fine-tunes lipid oxidation to metabolic demand and protects against lipotoxicity in skeletal muscle.Effect of Human Myotubes-Derived Media on Glucose-Stimulated Insulin Secretion.Does Insulin Resistance Trigger Natriuretic Peptide Deficiency?Natriuretic peptides and cGMP signaling control of energy homeostasis.Dynamics of skeletal muscle lipid pools.Natriuretic peptide control of energy balance and glucose homeostasis.Intramyocellular fat storage in metabolic diseases.Fatty acids from fat cell lipolysis do not activate an inflammatory response but are stored as triacylglycerols in adipose tissue macrophages.Targeting cardiac natriuretic peptides in the therapy of diabetes and obesity.Attenuated atrial natriuretic peptide-mediated lipolysis in subcutaneous adipocytes of obese type 2 diabetic men.White-to-brite conversion in human adipocytes promotes metabolic reprogramming towards fatty acid anabolic and catabolic pathways.Adipogenic progenitors from obese human skeletal muscle give rise to functional white adipocytes that contribute to insulin resistance.Defective Natriuretic Peptide Receptor Signaling in Skeletal Muscle Links Obesity to Type 2 Diabetes.Natriuretic peptides: new players in energy homeostasis.Impaired atrial natriuretic peptide-mediated lipolysis in obesity.Acute exposure to long-chain fatty acids impairs {alpha}2-adrenergic receptor-mediated antilipolysis in human adipose tissue.Adipocyte Exosomes Promote Melanoma Aggressiveness through Fatty Acid Oxidation: A Novel Mechanism Linking Obesity and Cancer.Comment on: Sitnick et al. Skeletal muscle triacylglycerol hydrolysis does not influence metabolic complications of obesity. Diabetes 2013;62:3350-3361.Comment on: Lazo et al. NH2-terminal pro-brain natriuretic peptide and risk of diabetes. Diabetes 2013;62:3189-3193.Endurance exercise training up-regulates lipolytic proteins and reduces triglyceride content in skeletal muscle of obese subjects.Atrial natriuretic peptide contributes to physiological control of lipid mobilization in humans.[Natriuretic peptides: a new lipolytic pathway in human fat cells].Lipid mobilization with physiological atrial natriuretic peptide concentrations in humans.Exercise-induced lipid mobilization in subcutaneous adipose tissue is mainly related to natriuretic peptides in overweight men.Training enhances ANP lipid-mobilizing action in adipose tissue of overweight men.Atrial natriuretic peptide stimulates lipid mobilization during repeated bouts of endurance exercise.Lipid oxidation according to intensity and exercise duration in overweight men and women.Sex differences in lipolysis-regulating mechanisms in overweight subjects: effect of exercise intensity.Caloric Restriction and Diet-Induced Weight Loss Do Not Induce Browning of Human Subcutaneous White Adipose Tissue in Women and Men with Obesity.
P50
Q27316188-F34BAA4B-361B-4800-A4E5-0F56D2753B34Q28592284-684674B3-BC79-4F9E-8F53-571005B391A4Q34758533-8C5AA7A0-2BCC-4A7E-A25E-9BDC4822B784Q35195718-A2749AB5-E047-4117-99F8-68E07601E461Q36131187-61B170F5-9DBB-47DB-AF31-89BF45A92F52Q36498070-C5C26496-18AF-4178-8146-936BCFD38725Q36933850-7E824898-2D3B-4011-9154-64D3E277B359Q37001119-73EA3032-2BB0-4F16-AB6D-C386324632DEQ37039708-C483AD93-5C19-4B4B-9902-B8C60E4589FEQ37109051-47859D60-8118-425E-8107-13D4B08FDEF9Q37166419-E7019D54-AEDA-44CE-B804-1457F9F1F538Q37476109-40F5325F-5459-4AD8-8D45-9331BA6F4FE1Q37670818-8FBB07D4-73BE-4CF6-9CD6-69CBFE4CED78Q37714308-D3FA0B07-B3BC-4E61-A32F-B33630EBD838Q38064374-62420650-E3CB-41B8-8219-78A529775178Q38132526-7848CA63-3632-4E4E-BF1B-DF85F8F6DC5BQ38513704-871ECACB-E443-48AB-9B2D-1AD62D89A702Q38689278-47A7415C-28D9-4C0B-BA63-5668067FF9A0Q38846145-BD265B47-8125-46A6-BB98-32B2D12EB131Q38992403-70986417-7645-4487-B754-2B82F1F3B8D4Q39809555-CA5D6F1E-4E93-4EFF-A5C5-FD1FD161660EQ40368059-BD1CFC13-B856-4DF3-B591-4340E0E37DA9Q40512963-3D7007F3-BD91-4819-BC2A-9E8374BDA8FFQ40647072-BFA5469C-175C-4C3D-A2D1-9E49A41A24EFQ42019825-D8A69B18-C2D4-4E93-A17B-3A5D62061775Q42484201-A6B01A9E-9740-4CF4-80D4-1A0F0E9255D4Q42515724-73987D65-99FB-4DAE-B0F0-12713E9BDEDBQ42810435-B349A09E-B8AC-4C8B-888C-7819A42CB512Q42873207-EF02BCBC-10A3-4ABB-A9BF-A7AB3F620A2FQ42873241-8039C881-85DD-4B92-8F0B-921808F6776DQ43450611-07307F66-A618-4CA3-B00C-B77887F59189Q44807389-3C9E35F0-F6C8-4544-8AD6-7D6C9739B2AFQ45216072-2930CAC4-8104-4BE2-AE2D-BFC4BD3ABEE2Q45291593-F6990C37-0898-443B-B50E-574DF3CF0BDAQ46535371-17420AE6-A444-465A-809B-63D5AA5F7135Q46598946-E6A8F88B-6526-4F12-BD30-51224FCD90DCQ46807508-407904BE-859D-4867-8FB0-72E69F9596D2Q46985476-8FA0A783-C923-4691-AD7D-20FDDFCAB531Q46985482-58A1BE61-9E3D-4093-9C8E-371E7D3F037AQ47554080-8E99F63C-B769-4AC2-BD3B-EFAA40D73873
P50
description
investigador
@es
researcher
@en
name
Cédric Moro
@en
Cédric Moro
@nl
type
label
Cédric Moro
@en
Cédric Moro
@nl
prefLabel
Cédric Moro
@en
Cédric Moro
@nl
P31
P496
0000-0003-4294-0597