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Replication rates of Mycobacterium tuberculosis in human macrophages do not correlate with mycobacterial antibiotic susceptibilityHuman macrophages infected with a high burden of ESAT-6-expressing M. tuberculosis undergo caspase-1- and cathepsin B-independent necrosisMidkine is expressed and differentially processed during chronic obstructive pulmonary disease exacerbations and ventilator-associated pneumonia associated with Staphylococcus aureus infection.IL-1 Receptor Antagonist Treatment Aggravates Staphylococcal Septic Arthritis and Sepsis in Mice.RAGE Deficiency Impairs Bacterial Clearance in Murine Staphylococcal Sepsis, but Has No Significant Impact on Staphylococcal Septic ArthritisHuman gene variants linked to enhanced NLRP3 activity limit intramacrophage growth of Mycobacterium tuberculosis.Inside or outside the phagosome? The controversy of the intracellular localization of Mycobacterium tuberculosis.Deficiency of the Complement Component 3 but Not Factor B Aggravates Staphylococcus aureus Septic Arthritis in Mice.Incorporation of Mycobacterium tuberculosis lipoarabinomannan into macrophage membrane rafts is a prerequisite for the phagosomal maturation block.Validation of a medium-throughput method for evaluation of intracellular growth of Mycobacterium tuberculosis.Vitamin D enhances IL-1β secretion and restricts growth of Mycobacterium tuberculosis in macrophages from TB patients.Galectin-3 Is a Target for Proteases Involved in the Virulence of Staphylococcus aureus.Comparative Proteomics of Purified Pathogen Vacuoles Correlates Intracellular Replication of Legionella pneumophila with the Small GTPase Ras-related protein 1 (Rap1).Eosinophils from eosinophilic oesophagitis patients have T cell suppressive capacity and express FOXP3.CTLA4 Immunoglobulin but Not Anti-Tumor Necrosis Factor Therapy Promotes Staphylococcal Septic Arthritis in Mice.Importance of phagosomal functionality for growth restriction of Mycobacterium tuberculosis in primary human macrophages.Survivin co-ordinates formation of follicular T-cells acting in synergy with Bcl-6.Structural insights into Legionella RidL-Vps29 retromer subunit interaction reveal displacement of the regulator TBC1D5.Formation of the Legionella Replicative Compartment at the Crossroads of Retrograde Trafficking.ER remodeling by the large GTPase atlastin promotes vacuolar growth of Legionella pneumophila.Quantitative imaging flow cytometry of Legionella-infected Dictyostelium reveals the impact of retrograde trafficking on pathogen vacuole composition.A neutrophil inhibitory pepducin derived from FPR1 expected to target FPR1 signaling hijacks the closely related FPR2 instead
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Amanda Welin
@en
Amanda Welin
@nl
type
label
Amanda Welin
@en
Amanda Welin
@nl
prefLabel
Amanda Welin
@en
Amanda Welin
@nl
P31
P496
0000-0001-8460-5952