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1
AAC.01052-10
AAC.01052-10
http://dx.doi.org/10.1128/AAC.01052-10
Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.
P356
Q39642654-5C85E3B5-53A1-4435-83F7-4CB689E5D6A9
P356
AAC.01052-10
http://dx.doi.org/10.1128/AAC.01052-10