Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus - Wikidata - wikimedia - TriplyDB

Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

http://www.wikidata.org/entity/Q21090530

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Inhibition of cyclophilins alters lipid trafficking and blocks hepatitis C virus secretion Regulation of hepatitis C virus translation and infectious virus production by the microRNA miR-122 Metabolic alterations and hepatitis C: From bench to bedside Roles of lipoprotein receptors in the entry of hepatitis C virus Assembly and release of infectious hepatitis C virus involving unusual organization of the secretory pathway Roles for endocytic trafficking and phosphatidylinositol 4-kinase III alpha in hepatitis C virus replication The association of CD81 with tetraspanin-enriched microdomains is not essential for Hepatitis C virus entry Role of Oxysterol Binding Protein in Hepatitis C Virus infection Towards a small animal model for hepatitis C Temporal Proteome and Lipidome Profiles Reveal Hepatitis C Virus-Associated Reprogramming of Hepatocellular Metabolism and Bioenergetics Identification of GBF1 as a Cellular Factor Required for Hepatitis C Virus RNA Replication Ethanol Enhances Hepatitis C Virus Replication through Lipid Metabolism and Elevated NADH/NAD+Lipid dysregulation in hepatitis C virus, and impact of statin therapy upon clinical outcomes A novel small molecule inhibitor of hepatitis C virus entry Tetraspanin CD81 is required for Listeria monocytogenes invasion.The missing pieces of the HCV entry puzzle.Complementary transcriptomic, lipidomic, and targeted functional genetic analyses in cultured Drosophila cells highlight the role of glycerophospholipid metabolism in Flock House virus RNA replication Gene expression profiling indicates the roles of host oxidative stress, apoptosis, lipid metabolism, and intracellular transport genes in the replication of hepatitis C virus.A new model to produce infectious hepatitis C virus without the replication requirement.Viral MicroRNAs Repress the Cholesterol Pathway, and 25-Hydroxycholesterol Inhibits Infection.Lipoprotein lipase inhibits hepatitis C virus (HCV) infection by blocking virus cell entry Interferon-lambda genotype and low serum low-density lipoprotein cholesterol levels in patients with chronic hepatitis C infection.Antisense gets a grip on miR-122 in chimpanzees.Associations between serum lipids and hepatitis C antiviral treatment efficacy.Identification of an overabundant cholesterol precursor in hepatitis B virus replicating cells by untargeted lipid metabolite profiling Barriers of hepatitis C virus interspecies transmission Gains of ubiquitylation sites in highly conserved proteins in the human lineage.Protein kinase D negatively regulates hepatitis C virus secretion through phosphorylation of oxysterol-binding protein and ceramide transfer protein Up-regulation of the ATP-binding cassette transporter A1 inhibits hepatitis C virus infection.The effect of antiviral therapy on serum cholesterol levels in chronic hepatitis C Antiretroviral Effects on Host Lipoproteins Are Associated With Changes in Hepatitis C Virus (HCV) RNA Levels in Human Immunodeficiency Virus/HCV Coinfected Individuals.Baseline serum cholesterol is associated with a response to pegylated interferon alfa-2b and ribavirin therapy for chronic hepatitis C genotype 2.Transient activation of the PI3K-AKT pathway by hepatitis C virus to enhance viral entry.Hepatitis C virus G1b infection decreases the number of small low-density lipoprotein particles Retrospective study of the associations between hepatitis C virus infection and metabolic factors.Activity of hexokinase is increased by its interaction with hepatitis C virus protein NS5A.Hepatitis C virus stimulates low-density lipoprotein receptor expression to facilitate viral propagation.Advances and challenges in studying hepatitis C virus in its native environment.Cholesterol and chronic hepatitis C virus infection.Antiviral therapy for hepatitis C virus: beyond the standard of care.

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

http://www.wikidata.org/entity/Q21090530

description

2007 nî lūn-bûn

@nan

2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի օգոստոսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of Host Cholesterol Metabolic Pathways for the Life Cycle of Hepatitis C Virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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Reliance of host cholesterol metabolic pathways for the life cycle of hepatitis C virus

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P2860

Binding of hepatitis C virus to CD81

Identification of FBL2 as a geranylgeranylated cellular protein required for hepatitis C virus RNA replication

The human scavenger receptor class B type I is a novel candidate receptor for the hepatitis C virus.

Mechanisms of inhibition by apolipoprotein C of apolipoprotein E-dependent cellular metabolism of human triglyceride-rich lipoproteins through the low density lipoprotein receptor pathway

Hepatitis C virus nonstructural proteins inhibit apolipoprotein B100 secretion

Clinical significance of hepatitis C virus genotypes

Expression of hepatitis C virus proteins induces distinct membrane alterations including a candidate viral replication complex

Scavenger receptor BI and BII expression levels modulate hepatitis C virus infectivity

Infectious hepatitis C virus pseudo-particles containing functional E1-E2 envelope protein complexes

Initiation of hepatitis C virus infection is dependent on cholesterol and cooperativity between CD81 and scavenger receptor B type I

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