Respiratory syncytial virus and TNF alpha induction of chemokine gene expression involves differential activation of Rel A and NF-kappa B1 - Wikidata - wikimedia - TriplyDB

Respiratory syncytial virus and TNF alpha induction of chemokine gene expression involves differential activation of Rel A and NF-kappa B1

Respiratory syncytial virus and TNF alpha induction of chemokine gene expression involves differential activation of Rel A and NF-kappa B1

http://www.wikidata.org/entity/Q21093328

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Normal and cystic fibrosis airway surface liquid homeostasis. The effects of phasic shear stress and viral infections Respiratory syncytial virus inhibits ciliagenesis in differentiated normal human bronchial epithelial cells: effectiveness of N-acetylcysteine.Respiratory syncytial virus represses glucocorticoid receptor-mediated gene activation.Cholesterol-rich microdomains as docking platforms for respiratory syncytial virus in normal human bronchial epithelial cells Role of human beta-defensin-2 during tumor necrosis factor-alpha/NF-kappaB-mediated innate antiviral response against human respiratory syncytial virus Respiratory syncytial virus (RSV) suppression of glucocorticoid receptor phosphorylation does not account for repression of transactivation Regulation of Monocyte Chemotactic Protein-1 secretion by the Two-Pore-Domain Potassium (K2P) channel TREK-1 in human alveolar epithelial cells Cigarette smoke condensate enhances respiratory syncytial virus-induced chemokine release by modulating NF-kappa B and interferon regulatory factor activation.The respiratory syncytial virus (RSV) nonstructural proteins mediate RSV suppression of glucocorticoid receptor transactivation.Autophagy-mediated dendritic cell activation is essential for innate cytokine production and APC function with respiratory syncytial virus responses.Respiratory syncytial virus-mediated NF-kappa B p65 phosphorylation at serine 536 is dependent on RIG-I, TRAF6, and IKK beta.Adenovirus type 7 induces interleukin-8 in a lung slice model and requires activation of Erk.Protein kinase C-alpha activity is required for respiratory syncytial virus fusion to human bronchial epithelial cells.LMP-420, a small-molecule inhibitor of TNF-alpha, reduces replication of HIV-1 and Mycobacterium tuberculosis in human cells.RNA-based drug screening using automated RNA purification and real-time RT-PCR1.Redox Biology of Respiratory Viral Infections

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Respiratory syncytial virus and TNF alpha induction of chemokine gene expression involves differential activation of Rel A and NF-kappa B1

http://www.wikidata.org/entity/Q21093328

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2002 nî lūn-bûn

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BMC Infectious Diseases

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James N Moy

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Kenneth A Roebuck

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Laura R Carpenter

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P2860

IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

Nuclear factor-kappaB-dependent induction of interleukin-8 gene expression by tumor necrosis factor alpha: evidence for an antioxidant sensitive activating pathway distinct from nuclear translocation

Essential involvement of interleukin-8 (IL-8) in acute inflammation

Respiratory syncytial virus-induced RANTES production from human bronchial epithelial cells is dependent on nuclear factor-kappa B nuclear binding and is inhibited by adenovirus-mediated expression of inhibitor of kappa B alpha

Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

NF-kappa B subunit-specific regulation of the interleukin-8 promoter

Tumor necrosis factor alpha and interleukin 1 stimulate the human immunodeficiency virus enhancer by activation of the nuclear factor kappa B

Stimulus-specific regulation of chemokine expression involves differential activation of the redox-responsive transcription factors AP-1 and NF-kappaB.

Multiple signals converging on NF-kappaB.

Regulation of interleukin-8 gene expression.

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10.1186/1471-2334-2-5

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2002-03-28T00:00:00Z

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11442114

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1044578336

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11922866

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infectious disease

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102322

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