Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
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Cytokine and Growth Factor Activation In Vivo and In Vitro after Spinal Cord InjuryAlzheimer's Disease: Exploring the Role of Inflammation and Implications for TreatmentMicroglia protect against brain injury and their selective elimination dysregulates neuronal network activity after stroke.Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agentsLupus brain fog: a biologic perspective on cognitive impairment, depression, and fatigue in systemic lupus erythematosusIntrahippocampal administration of a domain antibody that binds aggregated amyloid-β reverses cognitive deficits produced by diet-induced obesity.Dealing with Danger in the CNS: The Response of the Immune System to Injury.Ubiquilin-2 drives NF-κB activity and cytosolic TDP-43 aggregation in neuronal cells.Nonconventional MRI biomarkers for in vivo monitoring of pathogenesis in multiple sclerosis.High-Throughput Sequencing and Co-Expression Network Analysis of lncRNAs and mRNAs in Early Brain Injury Following Experimental Subarachnoid HaemorrhageTNF-α promotes extracellular vesicle release in mouse astrocytes through glutaminasePathogenic Roles of Glutamic Acid Decarboxylase 65 Autoantibodies in Cerebellar AtaxiasGABAergic/glutamatergic imbalance relative to excessive neuroinflammation in autism spectrum disorders.Cytokine correlations in youth with tic disorders.Proinflammatory cytokines upregulate sympathoexcitatory mechanisms in the subfornical organ of the rat.Valproic Acid Neuroprotection in the 6-OHDA Model of Parkinson's Disease Is Possibly Related to Its Anti-Inflammatory and HDAC Inhibitory Properties.Subventricular zone neural progenitors reverse TNF-alpha effects in cortical neurons.Microarray Profiling and Co-Expression Network Analysis of LncRNAs and mRNAs in Neonatal Rats Following Hypoxic-ischemic Brain DamagePentoxifylline Neuroprotective Effects Are Possibly Related to Its Anti-Inflammatory and TNF-Alpha Inhibitory Properties, in the 6-OHDA Model of Parkinson's Disease.Excitotoxins, Mitochondrial and Redox Disturbances in Multiple Sclerosis.Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS)Label-Free Proteomic Analysis of Protein Changes in the Striatum during Chronic Ethanol Use and Early WithdrawalCerebrovascular and microglial states are not altered by functional neuroinflammatory gene variant.Neuroinflammation in glaucoma: soluble tumor necrosis factor alpha and the connection with excitotoxic damage.Intraperitoneally administered IgG from patients with amyotrophic lateral sclerosis or from an immune-mediated goat model increase the levels of TNF-α, IL-6, and IL-10 in the spinal cord and serum of mice.Classical and Alternative Activation of Cyanobacterium Oscillatoria sp. Lipopolysaccharide-Treated Rat Microglia in vitroNF-κB and IRF1 Induce Endogenous Retrovirus K Expression via Interferon-Stimulated Response Elements in Its 5' Long Terminal RepeatSex differences in innate immunity and its impact on opioid pharmacology.Methylmercury induces the expression of TNF-α selectively in the brain of mice.TNFα Affects Ciliary Beat Response to Increased Viscosity in Human Pediatric Airway EpitheliumOpposing Functions of Microglial and Macrophagic TNFR2 in the Pathogenesis of Experimental Autoimmune Encephalomyelitis.Particulate matter exposure is associated with inflammatory gene methylation in obese subjectsNano-Sized Secondary Organic Aerosol of Diesel Engine Exhaust Origin Impairs Olfactory-Based Spatial Learning Performance in Preweaning Mice.Selective induction of alternatively spliced FynT isoform by TNF facilitates persistent inflammatory responses in astrocytes.Blockade of Glutamine Synthetase Enhances Inflammatory Response in Microglial Cells.Inflammatory signature of cerebellar neurodegeneration during neonatal hyperbilirubinemia in Ugt1 -/- mouse model.Sulfhydryl-mediated redox signaling in inflammation: role in neurodegenerative diseases.Functionality of Intrinsic Disorder in Tumor Necrosis Factor-α and its Receptors.The proinflammatory cytokine tumor necrosis factor-α excites subfornical organ neurons.Resveratrol modulates GSH system in C6 astroglial cells through heme oxygenase 1 pathway.
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P2860
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
description
2014 nî lūn-bûn
@nan
2014 թուականին հրատարակուած գիտական յօդուած
@hyw
2014 թվականին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@ast
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en-gb
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@nl
type
label
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@ast
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en-gb
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@nl
prefLabel
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@ast
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en-gb
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@nl
P2860
P921
P3181
P356
P1476
Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicity
@en
P2093
Jerònia Lladó
P2860
P304
P3181
P356
10.1155/2014/861231
P407
P50
P577
2014-05-21T00:00:00Z