N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis - Wikidata - wikimedia - TriplyDB

N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis

N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis

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N-acetylglucosamine Regulates Virulence Properties in Microbial Pathogens Multiple sclerosis: molecular mechanisms and therapeutic opportunities Oral Supplementation of Glucosamine Fails to Alleviate Acute Kidney Injury in Renal Ischemia-Reperfusion Damage Encoding asymmetry of the N-glycosylation motif facilitates glycoprotein evolution The Potential Role of T Helper Cell 22 and IL-22 in Immunopathogenesis of Multiple Sclerosis Glycolysis and glutaminolysis cooperatively control T cell function by limiting metabolite supply to N-glycosylation.Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.Family studies of type 1 diabetes reveal additive and epistatic effects between MGAT1 and three other polymorphisms N-glycan remodeling on glucagon receptor is an effector of nutrient sensing by the hexosamine biosynthesis pathway Sialylation of outer membrane porin protein D: a mechanistic basis of antibiotic uptake in Pseudomonas aeruginosa.Neurology of inherited glycosylation disorders.Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: An update for 2011-2012.Mannose metabolism: more than meets the eye.Metabolite profiling in posttraumatic stress disorder.Interleukin-2, Interleukin-7, T cell-mediated autoimmunity, and N-glycosylation.Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants.An inhibitor of chondroitin sulfate proteoglycan synthesis promotes central nervous system remyelination.Hyaluronan anchored to activated CD44 on central nervous system vascular endothelial cells promotes lymphocyte extravasation in experimental autoimmune encephalomyelitis.N-acetylglucosamine (GlcNAc) functions in cell signaling Understanding human glycosylation disorders: biochemistry leads the charge.The regulatory power of glycans and their binding partners in immunity Aberrant glycosylation of plasma proteins in severe preeclampsia promotes monocyte adhesion.Glycobiology of immune responses Pathogenesis of multiple sclerosis via environmental and genetic dysregulation of N-glycosylation.PFA-fixed Hsp60sp-loaded dendritic cells as a vaccine for the control of mouse experimental allergic encephalomyelitis.Self-antigen presentation by dendritic cells in autoimmunity.Glycans of myelin proteins.Immunometabolism in early and late stages of rheumatoid arthritis.Density-dependent lectin-glycan interactions as a paradigm for conditional regulation by posttranslational modifications.Comparative Phenotype and Genome Analysis of Cellvibrio sp. PR1, a Xylanolytic and Agarolytic Bacterium from the Pearl River.Perhaps a wee bit of sugar would help.Assessment of protective effects of glucosamine and N-acetyl glucosamine against DNA damage induced by hydrogen peroxide in human lymphocytes.Collagen-induced arthritis: severity and immune response attenuation using multivalent N-acetyl glucosamine.Glycolysis and the Hexosamine Biosynthetic Pathway as Novel Targets for Upper and Lower Airway Inflammation.Sugar or Fat?-Metabolic Requirements for Immunity to Viral Infections.The Modulatory Roles of N-glycans in T-Cell-Mediated Autoimmune Diseases.Glycyrrhizin Protects Mice Against Experimental Autoimmune Encephalomyelitis by Inhibiting High-Mobility Group Box 1 (HMGB1) Expression and Neuronal HMGB1 Release.

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N-Acetylglucosamine Inhibits T-helper 1 (Th1)/T-helper 17 (Th17) Cell Responses and Treats Experimental Autoimmune Encephalomyelitis

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Molecularly cloned mammalian glucosamine-6-phosphate deaminase localizes to transporting epithelium and lacks oscillin activity

Assembly of asparagine-linked oligosaccharides

A placebo-controlled trial of oral fingolimod in relapsing multiple sclerosis

Oral fingolimod or intramuscular interferon for relapsing multiple sclerosis

The Tim-3 ligand galectin-9 negatively regulates T helper type 1 immunity

MGAT5 alters the severity of multiple sclerosis

Risk alleles for multiple sclerosis identified by a genomewide study

Cloning and functional characterization of GNPI2, a novel human homolog of glucosamine-6-phosphate isomerase/oscillin.

Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation.

Genome, epigenome and RNA sequences of monozygotic twins discordant for multiple sclerosis.

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