Response gene to complement 32 interacts with Smad3 to promote epithelial-mesenchymal transition of human renal tubular cells
about
Response gene to complement 32 is essential for fibroblast activation in renal fibrosisSingle-nucleotide polymorphisms of the dopamine D2 receptor increase inflammation and fibrosis in human renal proximal tubule cells.Response gene to complement 32 regulates the G2/M phase checkpoint during renal tubular epithelial cell repair.Smad2 and myocardin-related transcription factor B cooperatively regulate vascular smooth muscle differentiation from neural crest cells.Response gene to complement 32 promotes vascular lesion formation through stimulation of smooth muscle cell proliferation and migration.Smad2 and PEA3 cooperatively regulate transcription of response gene to complement 32 in TGF-β-induced smooth muscle cell differentiation of neural crest cells.Dedicator of cytokinesis 2, a novel regulator for smooth muscle phenotypic modulation and vascular remodeling.Response gene to complement 32 (RGC-32) expression on M2-polarized and tumor-associated macrophages is M-CSF-dependent and enhanced by tumor-derived IL-4.Response gene to complement 32 deficiency causes impaired placental angiogenesis in mice.Role of C5b-9 complement complex and response gene to complement-32 (RGC-32) in cancer.Genetics of breast cancer bone metastasis: a sequential multistep pattern.DOCK2 deficiency mitigates HFD-induced obesity by reducing adipose tissue inflammation and increasing energy expenditure.Down-regulated miR-15a mediates the epithelial-mesenchymal transition in renal tubular epithelial cells promoted by high glucose.Snail and kidney fibrosis.Overexpression of response gene to complement 32 (RGC32) promotes cell invasion and induces epithelial-mesenchymal transition in lung cancer cells via the NF-κB signaling pathway.Connections in chronic kidney disease: connexin 43 and connexin 37 interaction.RGC-32 Promotes Th17 Cell Differentiation and Enhances Experimental Autoimmune Encephalomyelitis.RGC-32 (Response Gene to Complement 32 Protein) Deficiency Protects Endothelial Cells From Inflammation and Attenuates Atherosclerosis.Response Gene to Complement 32 in Vascular Diseases
P2860
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P2860
Response gene to complement 32 interacts with Smad3 to promote epithelial-mesenchymal transition of human renal tubular cells
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Response gene to complement 32 ...... n of human renal tubular cells
@ast
Response gene to complement 32 ...... n of human renal tubular cells
@en
Response gene to complement 32 ...... n of human renal tubular cells
@en-gb
Response gene to complement 32 ...... n of human renal tubular cells
@nl
type
label
Response gene to complement 32 ...... n of human renal tubular cells
@ast
Response gene to complement 32 ...... n of human renal tubular cells
@en
Response gene to complement 32 ...... n of human renal tubular cells
@en-gb
Response gene to complement 32 ...... n of human renal tubular cells
@nl
prefLabel
Response gene to complement 32 ...... n of human renal tubular cells
@ast
Response gene to complement 32 ...... n of human renal tubular cells
@en
Response gene to complement 32 ...... n of human renal tubular cells
@en-gb
Response gene to complement 32 ...... n of human renal tubular cells
@nl
P2093
P2860
P921
P1476
Response gene to complement 32 ...... n of human renal tubular cells
@en
P2093
Pedro A Jose
Shi-You Chen
P2860
P304
P356
10.1152/AJPCELL.00204.2010
P407
P577
2011-06-01T00:00:00Z