SCAI acts as a suppressor of cancer cell invasion through the transcriptional control of beta1-integrin
about
Redox modification of nuclear actin by MICAL-2 regulates SRF signalingJunctional actin assembly is mediated by Formin-like 2 downstream of Rac1.Nuclear F-actin formation and reorganization upon cell spreading.Nuclear actin modulates cell motility via transcriptional regulation of adhesive and cytoskeletal genes.Formin-like 2 drives amoeboid invasive cell motility downstream of RhoCProtein complex interactor analysis and differential activity of KDM3 subfamily members towards H3K9 methylationCdc42 promotes transendothelial migration of cancer cells through β1 integrin.RCP-driven α5β1 recycling suppresses Rac and promotes RhoA activity via the RacGAP1-IQGAP1 complexMiR-425-5p promotes invasion and metastasis of hepatocellular carcinoma cells through SCAI-mediated dysregulation of multiple signaling pathwaysActivation and repression of cellular immediate early genes by serum response factor cofactors.Shape-induced terminal differentiation of human epidermal stem cells requires p38 and is regulated by histone acetylation.Linking actin dynamics and gene transcription to drive cellular motile functions.Involvement of the serum response factor coactivator megakaryoblastic leukemia (MKL) in the activin-regulated dendritic complexity of rat cortical neuronsEmerging roles of the myocardin family of proteins in lipid and glucose metabolism.Mechanical signaling through the cytoskeleton regulates cell proliferation by coordinated focal adhesion and Rho GTPase signaling.Functional interaction of SCAI with the SWI/SNF complex for transcription and tumor cell invasionLMO7 mediates cell-specific activation of the Rho-myocardin-related transcription factor-serum response factor pathway and plays an important role in breast cancer cell migration.MicroRNA-1290 promotes esophageal squamous cell carcinoma cell proliferation and metastasis.Rho/MRTF-A-Induced Integrin Expression Regulates Angiogenesis in Differentiated Multipotent Mesenchymal Stem CellsMiR-1228 promotes breast cancer cell growth and metastasis through targeting SCAI protein.SCAI promotes DNA double-strand break repair in distinct chromosomal contextsMiR-625-3p promotes cell migration and invasion via inhibition of SCAI in colorectal carcinoma cells.Androgen-responsive serum response factor target genes regulate prostate cancer cell migration.Proteomic signatures of acquired letrozole resistance in breast cancer: suppressed estrogen signaling and increased cell motility and invasiveness.HPV16 integration probably contributes to cervical oncogenesis through interrupting tumor suppressor genes and inducing chromosome instability.SAP domain-dependent Mkl1 signaling stimulates proliferation and cell migration by induction of a distinct gene set indicative of poor prognosis in breast cancer patients.Vascular smooth muscle cell differentiation-2010.Rho, nuclear actin, and actin-binding proteins in the regulation of transcription and gene expression.Nuclear roles for actin.Envisioning metastasis as a transdifferentiation phenomenon clarifies discordant results on cancer.Filamin A interacts with the coactivator MKL1 to promote the activity of the transcription factor SRF and cell migration.Mechanism of irradiation-induced mammary cancer metastasis: A role for SAP-dependent Mkl1 signaling.The kinase LMTK3 promotes invasion in breast cancer through GRB2-mediated induction of integrin β₁.Rho GTPases and cancer.Distinct roles of AKT isoforms in regulating β1-integrin activity, migration, and invasion in prostate cancer.MRTFs- master regulators of EMT.Transient α-helices in the disordered RPEL motifs of the serum response factor coactivator MKL1.Myocardin-related transcription factor A (MRTF-A) activity-dependent cell adhesion is correlated to focal adhesion kinase (FAK) activity.Comparative proteomic analysis of the function and network mechanisms of MASPIN in human lung cells.Injury induced expression of caveolar proteins in human kidney tubules - role of megakaryoblastic leukemia 1.
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SCAI acts as a suppressor of cancer cell invasion through the transcriptional control of beta1-integrin
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@ast
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en-gb
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@nl
type
label
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@ast
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en-gb
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@nl
prefLabel
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@ast
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en-gb
SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@nl
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SCAI acts as a suppressor of c ...... onal control of beta1-integrin
@en
P2093
Christian Baarlink
Dominique T Brandt
Peter Nollau
Thomas M Kitzing
P2860
P2888
P304
P3181
P356
10.1038/NCB1862
P407
P577
2009-05-01T00:00:00Z
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P6179
1043827056