Increased insulin action in SKIP heterozygous knockout mice
about
Silencer of death domains (SODD) inhibits skeletal muscle and kidney enriched inositol 5-phosphatase (SKIP) and regulates phosphoinositide 3-kinase (PI3K)/Akt signaling to the actin cytoskeletonInositol polyphosphate 5-phosphatases; new players in the regulation of cilia and ciliopathiesPhosphoinositides: tiny lipids with giant impact on cell regulationPhosphoinositides: Key modulators of energy metabolismThe inositol Inpp5k 5-phosphatase affects osmoregulation through the vasopressin-aquaporin 2 pathway in the collecting systemMutations in INPP5K, Encoding a Phosphoinositide 5-Phosphatase, Cause Congenital Muscular Dystrophy with Cataracts and Mild Cognitive ImpairmentGlucose-regulated protein 78 (GRP78) binds directly to PIP3 phosphatase SKIP and determines its localization.Regulation of insulin signaling by the phosphatidylinositol 3,4,5-triphosphate phosphatase SKIP through the scaffolding function of Pak1Regulation of insulin signaling and glucose transporter 4 (GLUT4) exocytosis by phosphatidylinositol 3,4,5-trisphosphate (PIP3) phosphatase, skeletal muscle, and kidney enriched inositol polyphosphate phosphatase (SKIP).Sac3 is an insulin-regulated phosphatidylinositol 3,5-bisphosphate phosphatase: gain in insulin responsiveness through Sac3 down-regulation in adipocytesThe structure of phosphoinositide phosphatases: Insights into substrate specificity and catalysis.Lowered Expression of Tumor Suppressor Candidate MYO1C Stimulates Cell Proliferation, Suppresses Cell Adhesion and Activates AKT.Differential SKIP expression in PTEN-deficient glioblastoma regulates cellular proliferation and migration.Phosphatidylinositolphosphate phosphatase activities and cancer.Role of phosphatidylinositol 3,4,5-trisphosphate (PIP3) 5-phosphatase skeletal muscle- and kidney-enriched inositol polyphosphate phosphatase (SKIP) in myoblast differentiation.Phosphatidylinositol 3,4,5-Trisphosphate Phosphatase SKIP Links Endoplasmic Reticulum Stress in Skeletal Muscle to Insulin Resistance.Inpp5e suppresses polycystic kidney disease via inhibition of PI3K/Akt-dependent mTORC1 signaling.The inositol 5-phosphatase INPP5K participates in the fine control of ER organization
P2860
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P2860
Increased insulin action in SKIP heterozygous knockout mice
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Increased insulin action in SKIP heterozygous knockout mice
@ast
Increased insulin action in SKIP heterozygous knockout mice
@en
Increased insulin action in SKIP heterozygous knockout mice
@en-gb
Increased insulin action in SKIP heterozygous knockout mice
@nl
type
label
Increased insulin action in SKIP heterozygous knockout mice
@ast
Increased insulin action in SKIP heterozygous knockout mice
@en
Increased insulin action in SKIP heterozygous knockout mice
@en-gb
Increased insulin action in SKIP heterozygous knockout mice
@nl
prefLabel
Increased insulin action in SKIP heterozygous knockout mice
@ast
Increased insulin action in SKIP heterozygous knockout mice
@en
Increased insulin action in SKIP heterozygous knockout mice
@en-gb
Increased insulin action in SKIP heterozygous knockout mice
@nl
P2093
P2860
P921
P3181
P356
P1476
Increased insulin action in SKIP heterozygous knockout mice
@en
P2093
Allan Bradley
Kiyohito Mizutani
Sanshiro Tateya
Tadaomi Takenawa
Y Eugene Yu
Yoshikazu Tamori
P2860
P304
P3181
P356
10.1128/MCB.01990-06
P407
P577
2008-09-01T00:00:00Z