TAR-independent transactivation by Tat in cells derived from the CNS: a novel mechanism of HIV-1 gene regulation
about
The octamer-binding proteins Oct-1 and Oct-2 repress the HIV long terminal repeat promoter and its transactivation by TatAbsolute dependence on kappa B responsive elements for initiation and Tat-mediated amplification of HIV transcription in blood CD4 T lymphocytesInhibition of HIV-1 Tat-mediated LTR transactivation and HIV-1 infection by anti-Tat single chain intrabodiesHuman immunodeficiency virus type 1 Tat upregulates interleukin-2 secretion in activated T cellsRegulation of human immunodeficiency virus type 1 and cytokine gene expression in myeloid cells by NF-kappa B/Rel transcription factorsTransdominant mutants of I kappa B alpha block Tat-tumor necrosis factor synergistic activation of human immunodeficiency virus type 1 gene expression and virus multiplicationDiminished production of human immunodeficiency virus type 1 in astrocytes results from inefficient translation of gag, env, and nef mRNAs despite efficient expression of Tat and RevRole for human immunodeficiency virus type 1 Tat protein in suppression of viral reverse transcriptase activity during late stages of viral replication.In vivo selection of randomly mutated retroviral genomes.Interplay of Rad51 with NF-κB pathway stimulates expression of HIV-1.Interactions of the transcription factor AP-1 with the long terminal repeat of different human immunodeficiency virus type 1 strains in Jurkat, glial, and neuronal cellsTranscription of the JC virus archetype late genome: importance of the kappa B and the 23-base-pair motifs in late promoter activity in glial cellsCooperation between herpes simplex virus type 1-encoded ICP0 and Tat to support transcription of human immunodeficiency virus type 1 long terminal repeat in vivo can occur in the absence of the TAR binding site.Inhibition of human immunodeficiency virus type 1 and type 2 Tat function by transdominant Tat protein localized to both the nucleus and cytoplasm.A model system for human cytomegalovirus-mediated modulation of human immunodeficiency virus type 1 long terminal repeat activity in brain cells.HIV-1 transactivator protein Tat induces proliferation and TGF beta expression in human articular chondrocytes.The emergence and evolution of the research fronts in HIV/AIDS research.Central nervous system-derived cells express a kappa B-binding activity that enhances human immunodeficiency virus type 1 transcription in vitro and facilitates TAR-independent transactivation by Tat.The full-length Tat protein is required for TAR-independent, posttranscriptional trans activation of human immunodeficiency virus type 1 env gene expression.HIV-1 Tat mediates degradation of RON receptor tyrosine kinase, a regulator of inflammation.Regulation of HIV-1 latency by T-cell activation.Activation of expression of genes coding for extracellular matrix proteins in Tat-producing glioblastoma cells.HIV-1 Tat directly binds to NFkappaB enhancer sequence: role in viral and cellular gene expression.Human immunodeficiency virus tat gene transfer to the murine central nervous system using a replication-defective herpes simplex virus vector stimulates transforming growth factor beta 1 gene expression.Evolution of a disrupted TAR RNA hairpin structure in the HIV-1 virus.The HIV-1 Tat protein has a versatile role in activating viral transcriptionThe tumor suppressor protein p53 strongly alters human immunodeficiency virus type 1 replicationPermanent occupancy of the human immunodeficiency virus type 1 enhancer by NF-kappa B is needed for persistent viral replication in monocytes.Cooperative roles of NF-κB and NFAT4 in polyomavirus JC regulation at the KB control element.The Tat protein of human immunodeficiency virus type 1 (HIV-1) can promote placement of tRNA primer onto viral RNA and suppress later DNA polymerization in HIV-1 reverse transcription.Evidence for regulation of long terminal repeat transcription by Wnt transcription factor TCF-4 in human astrocytic cells.Regulation of I kappa B alpha and p105 in monocytes and macrophages persistently infected with human immunodeficiency virus.The Ras-Raf pathway is activated in human immunodeficiency virus-infected monocytes and particpates in the activation of NF-kappa B.Differential role of long terminal repeat control elements for the regulation of basal and Tat-mediated transcription of the human immunodeficiency virus in stimulated and unstimulated primary human macrophages.TAR loop-dependent human immunodeficiency virus trans activation requires factors encoded on human chromosome 12.TAR-independent replication of human immunodeficiency virus type 1 in glial cells.Cross-interaction between JC virus agnoprotein and human immunodeficiency virus type 1 (HIV-1) Tat modulates transcription of the HIV-1 long terminal repeat in glial cells.
P2860
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P2860
TAR-independent transactivation by Tat in cells derived from the CNS: a novel mechanism of HIV-1 gene regulation
description
1992 nî lūn-bûn
@nan
1992 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
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1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@ast
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@en
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@nl
type
label
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@ast
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@en
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@nl
prefLabel
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@ast
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@en
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@nl
P2093
P2860
P1433
P1476
TAR-independent transactivatio ...... anism of HIV-1 gene regulation
@en
P2093
J P Taylor
J Rappaport
M Chowdhury
R Pomerantz
P2860
P304
P407
P577
1992-09-01T00:00:00Z