HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol - Wikidata - wikimedia - TriplyDB

HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol

HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol

http://www.wikidata.org/entity/Q24555784

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Developments in the scientific and clinical understanding of gout Toxic epidermal necrolysis and Stevens-Johnson syndrome Genetic testing for prevention of severe drug-induced skin rash Peptide-binding motifs and characteristics for HLA -B*13:01 molecule 2012 American College of Rheumatology guidelines for management of gout. Part 1: systematic nonpharmacologic and pharmacologic therapeutic approaches to hyperuricemia Clinical Pharmacogenetics Implementation Consortium guidelines for human leukocyte antigen-B genotype and allopurinol dosing The genetics of hyperuricaemia and gout Immunotoxicogenomics: gene expression and immunotoxicology in the age of genomics Treatment of hyperuricemia in gout: current therapeutic options, latest developments and clinical implications Drug induced exfoliative dermatitis: state of the art Toxic epidermal necrolysis Impact of New Genomic Technologies on Understanding Adverse Drug Reactions Evolving models of the immunopathogenesis of T cell-mediated drug allergy: The role of host, pathogens, and drug response.Association of HLA-B*5801 allele and allopurinol-induced Stevens Johnson syndrome and toxic epidermal necrolysis: a systematic review and meta-analysis Abacavir pharmacogenetics--from initial reports to standard of care Pharmacogenomics of adverse drug reactions Mechanistic insights into xanthine oxidoreductase from development studies of candidate drugs to treat hyperuricemia and gout Genotypic and phenotypic spectrum in attenuated variants of Lesch-Nyhan disease Drug reaction with Eosinophilia and Systemic Symptoms (DRESS) / Drug-induced Hypersensitivity Syndrome (DIHS): a review of current concepts Harvesting candidate genes responsible for serious adverse drug reactions from a chemical-protein interactome Drug hypersensitivity caused by alteration of the MHC-presented self-peptide repertoire Fever, rash, and systemic symptoms: understanding the role of virus and HLA in severe cutaneous drug allergy.Severe Cutaneous Adverse Reactions: The Pharmacogenomics from Research to Clinical Implementation The Importance of Patient-Specific Factors for Hepatic Drug Response and Toxicity Relationship between human leucocyte antigen class I and class II and chronic idiopathic urticaria associated with aspirin and/or NSAIDs hypersensitivity Exploring off-targets and off-systems for adverse drug reactions via chemical-protein interactome--clozapine-induced agranulocytosis as a case study ABC transporters and the proteasome complex are implicated in susceptibility to Stevens-Johnson syndrome and toxic epidermal necrolysis across multiple drugs Genome-Wide Association Study in Immunocompetent Patients with Delayed Hypersensitivity to Sulfonamide Antimicrobials What Taiwan contributes to the world of allergy and clinical immunology?Genome-wide association study of Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis in Europe Human leucocyte antigen-adverse drug reaction associations: from a perspective of ethnicity.Drug reaction with eosinophilia and systemic symptoms (DRESS): incidence, pathogenesis and management.Allopurinol: insights from studies of dose-response relationships.HLA-DRB1*07:01 is associated with a higher risk of asparaginase allergies.Pharmacogenomics of off-target adverse drug reactions.Japan PGx Data Science Consortium Database: SNPs and HLA genotype data from 2994 Japanese healthy individuals for pharmacogenomics studies.DRESS syndrome: cerebral vasculitic-like presentation.Influence of human leukocyte antigen (HLA) alleles and killer cell immunoglobulin-like receptors (KIR) types on heparin-induced thrombocytopenia (HIT).Genotyping for severe drug hypersensitivity.Tumor lysis syndrome: new challenges and recent advances.

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HLA-B*5801 allele as a genetic marker for severe cutaneous adverse reactions caused by allopurinol

http://www.wikidata.org/entity/Q24555784

description

2005 nî lūn-bûn

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2005 թուականի Մարտին հրատարակուած գիտական յօդուած

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2005 թվականի մարտին հրատարակված գիտական հոդված

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2005年の論文

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HLA-B*5801 allele as a genetic ...... eactions caused by allopurinol

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HLA-B*5801 allele as a genetic ...... eactions caused by allopurinol

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HLA-B*5801 allele as a genetic ...... eactions caused by allopurinol

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HLA-B*5801 allele as a genetic ...... eactions caused by allopurinol

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Proceedings of the National Academy of Sciences of the United States of America

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Cathy S J Fann

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Chen-Chung Chu

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Chia-Yu Chu

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Chien-Hsiun Chen

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Hong-Shang Hong

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Hsien-Ping Huang

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Jer-Yuarn Wu

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Joung-Liang Lan

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Kuo-Hsien Wang

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Li-Cheng Yang

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P2860

Stevens-Johnson syndrome induced by methazolamide treatment

FLAME-1, a novel FADD-like anti-apoptotic molecule that regulates Fas/TNFR1-induced apoptosis

Medical genetics: a marker for Stevens-Johnson syndrome

Relation between adverse events associated with allopurinol and renal function in patients with gout

dbSNP: a database of single nucleotide polymorphisms

An integrated haplotype map of the human major histocompatibility complex.

Ankylosing spondylitis and HL-A 27.

Medication use and the risk of Stevens-Johnson syndrome or toxic epidermal necrolysis.

Human herpesvirus 6 infection as a risk factor for the development of severe drug-induced hypersensitivity syndrome.

Association between presence of HLA-B*5701, HLA-DR7, and HLA-DQ3 and hypersensitivity to HIV-1 reverse-transcriptase inhibitor abacavir.

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102

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Yuan-Tsong Chen

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2005-03-02T00:00:00Z

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7989995

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