Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily - Wikidata - wikimedia - TriplyDB

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

http://www.wikidata.org/entity/Q24558912

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A role for the receptor for advanced glycation end products in idiopathic pulmonary fibrosis The Relationship between Dyslipidemia and Acute Axonal Function in Type 2 Diabetes Mellitus In Vivo Oxidative stress and diabetic complications Large scale isolation and purification of soluble RAGE from lung tissue.Diabetic neuropathy: mechanisms to management Controlling the receptor for advanced glycation end-products to conquer diabetic vascular complications The HMGB1-RAGE axis mediates traumatic brain injury-induced pulmonary dysfunction in lung transplantation.Neuropathy, retinopathy, and glucose-lowering treatments.New therapeutic and biomarker discovery for peripheral diabetic neuropathy: PARP inhibitor, nitrotyrosine, and tumor necrosis factor-{alpha}Different roles of 12/15-lipoxygenase in diabetic large and small fiber peripheral and autonomic neuropathies.Soluble RAGE: therapy and biomarker in unraveling the RAGE axis in chronic disease and aging Up-regulation of the receptor for advanced glycation end products in the skin biopsy specimens of patients with severe diabetic neuropathy.Baicalein alleviates diabetic peripheral neuropathy through inhibition of oxidative-nitrosative stress and p38 MAPK activation.Evaluation of PMI-5011, an ethanolic extract of Artemisia dracunculus L., on peripheral neuropathy in streptozotocin-diabetic mice.Paradoxical function for the receptor for advanced glycation end products in mouse models of pulmonary fibrosis.PARP inhibition alleviates diabetes-induced systemic oxidative stress and neural tissue 4-hydroxynonenal adduct accumulation: correlation with peripheral nerve function.RAGE regulates immune cell infiltration and angiogenesis in choroidal neovascularization.Morphological Changes and Immunohistochemical Expression of RAGE and its Ligands in the Sciatic Nerve of Hyperglycemic Pig (Sus Scrofa)Regulation of RAGE for attenuating progression of diabetic vascular complications.Inactivation of TNF-α ameliorates diabetic neuropathy in mice.BTBR ob/ob mice as a novel diabetic neuropathy model: Neurological characterization and gene expression analyses Posttranslationally modified proteins as mediators of sustained intestinal inflammation.Early- and advanced non-enzymatic glycation in diabetic vascular complications: the search for therapeutics.Triglyceride, nonesterified fatty acids, and prediabetic neuropathy: role for oxidative-nitrosative stress.Role of advanced glycation endproducts and glyoxalase I in diabetic peripheral sensory neuropathy.Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation.Metanx alleviates multiple manifestations of peripheral neuropathy and increases intraepidermal nerve fiber density in Zucker diabetic fatty rats.The HMGB1 receptor RAGE mediates ischemic brain damage.Advanced glycation end product receptor-mediated cellular dysfunction.Emerging Targets for Therapeutic Development in Diabetes and Its Complications: The RAGE Signaling Pathway.Exercise alleviates hypoalgesia and increases the level of calcitonin gene-related peptide in the dorsal horn of the spinal cord of diabetic rats.Diabetic peripheral neuropathy: should a chaperone accompany our therapeutic approach?A novel function of the receptor for advanced glycation end-products (RAGE) in association with tumorigenesis and tumor differentiation of HCC Critical role of RAGE and HMGB1 in inflammatory heart disease.RAGE deficiency improves postinjury sciatic nerve regeneration in type 1 diabetic mice.Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes.Sulfasalazine blocks the development of tactile allodynia in diabetic rats.RAGE and soluble RAGE: potential therapeutic targets for cardiovascular diseases.Receptor for AGEs (RAGE) blockade may exert its renoprotective effects in patients with diabetic nephropathy via induction of the angiotensin II type 2 (AT2) receptor.RAGE-induced cytosolic ROS promote mitochondrial superoxide generation in diabetes.

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P2860

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

http://www.wikidata.org/entity/Q24558912

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2004 nî lūn-bûn

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2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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Journal of Clinical Investigation

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Loss of pain perception in dia ...... the immunoglobulin superfamily

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P2093

Ahmed A R Sayed

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Angelika Bierhaus

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Ann-Marie Schmidt

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Bernd Arnold

http://www.w3.org/2001/XMLSchema#string

Bernhard Neundörfer

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Birgit Liliensiek

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David M Stern

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Dieter Heuss

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Erwin Schleicher

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Hans Tritschler

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P2860

A role for ASIC3 in the modulation of high-intensity pain stimuli

Biochemistry and molecular cell biology of diabetic complications

Diabetes-associated sustained activation of the transcription factor nuclear factor-kappaB

Reversal of experimental diabetic neuropathy by VEGF gene transfer

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Glucose-induced oxidative stress and programmed cell death in diabetic neuropathy.

Diabetic neuropathies.

C-peptide prevents and improves chronic Type I diabetic polyneuropathy in the BB/Wor rat.

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1741-51

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scholarly article

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2309951

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10.1172/JCI18058

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12

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114

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2004-12-01T00:00:00Z

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15599399

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535062

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