The extent of heterocellular communication mediated by gap junctions is predictive of bystander tumor cytotoxicity in vitro - Wikidata - wikimedia - TriplyDB

The extent of heterocellular communication mediated by gap junctions is predictive of bystander tumor cytotoxicity in vitro

The extent of heterocellular communication mediated by gap junctions is predictive of bystander tumor cytotoxicity in vitro

http://www.wikidata.org/entity/Q24564192

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Prodrugs for Gene-Directed Enzyme-Prodrug Therapy (Suicide Gene Therapy)Connexins in neurons and glia: targets for intervention in disease and injury Therapeutic efficacy of human hepatocyte transplantation in a SCID/uPA mouse model with inducible liver disease Control of Kaposi's sarcoma-associated herpesvirus reactivation induced by multiple signals Tanshinone IIA increases the bystander effect of herpes simplex virus thymidine kinase/ganciclovir gene therapy via enhanced gap junctional intercellular communication The engineered thymidylate kinase (TMPK)/AZT enzyme-prodrug axis offers efficient bystander cell killing for suicide gene therapy of cancer Targeting of suicide gene delivery in pancreatic cancer cells via FGF receptors.Combination gene delivery of the cell cycle inhibitor p27 with thymidine kinase enhances prodrug cytotoxicity Prodrug activation enzymes in cancer gene therapy.Plant thymidine kinase 1: a novel efficient suicide gene for malignant glioma therapy Phase IB study of gene-mediated cytotoxic immunotherapy adjuvant to up-front surgery and intensive timing radiation for malignant glioma.Gap junctions and tumour progression.Sharing signals: connecting lung epithelial cells with gap junction channels.Proposed mechanisms of action for prostate cancer vaccines.Gene therapy of hepatocarcinoma: a long way from the concept to the therapeutical impact.Gap junction coupling is required for tumor cell migration through lymphatic endothelium Gene therapy for malignant glioma.TK gene combined with mIL-2 and mGM-CSF genes in treatment of gastric cancer Monitoring the Bystander Killing Effect of Human Multipotent Stem Cells for Treatment of Malignant Brain Tumors.Oxidative stress, lens gap junctions, and cataracts Connexin43 increases the sensitivity of prostate cancer cells to TNFalpha-induced apoptosis.Strategies in gene therapy for glioblastoma.Glioma virus therapies between bench and bedside.Gap junctions and cancer: communicating for 50 years.Cytotoxic immunotherapy strategies for cancer: mechanisms and clinical development.Connexins: key mediators of endocrine function.Involvement of Fas (CD95/APO-1) and Fas ligand in apoptosis induced by ganciclovir treatment of tumor cells transduced with herpes simplex virus thymidine kinase.Micro-PET/CT monitoring of herpes thymidine kinase suicide gene therapy in a prostate cancer xenograft: the advantage of a cell-specific transcriptional targeting approach.Relationship between the expression of connexin43 and bystander effect of suicide gene therapy in ovarian cancer.Direct evidence for the absence of intercellular trafficking of VP22 fused to GFP or to the herpes simplex virus thymidine kinase.Hapten-directed targeting to single-chain antibody receptors.Selective inhibition of hepatoma cells using diphtheria toxin A under the control of the promoter/enhancer region of the human alpha-fetoprotein gene.Antitumor activity with the HSV-tk-gene-modified cell line PA-1-STK in malignant mesothelioma.Mind the Gaps in Tumor Immunity: Impact of Connexin-Mediated Intercellular Connections.Increased cytotoxicity and bystander effect of 5-fluorouracil and 5-deoxy-5-fluorouridine in human colorectal cancer cells transfected with thymidine phosphorylase.Gene- and viral-based therapies for brain tumors Stimulation of intercellular communication of poor-communicating cells by gap-junction-competent cells enhances the HSV-TK/GCV bystander effect in vitro.Induction of a bystander effect in HeLa cells by using a bigenic vector carrying viral thymidine kinase and connexin32 genes.Bystander effect of purine nucleoside analogues in HSV-1 tk suicide gene therapy is superior to that of pyrimidine nucleoside analogues.Connexin 43-mediated bystander effect in two rat glioma cell models.

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The extent of heterocellular communication mediated by gap junctions is predictive of bystander tumor cytotoxicity in vitro

http://www.wikidata.org/entity/Q24564192

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1995 nî lūn-bûn

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1995 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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1995年の論文

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The extent of heterocellular c ...... er tumor cytotoxicity in vitro

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The extent of heterocellular c ...... er tumor cytotoxicity in vitro

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The extent of heterocellular c ...... er tumor cytotoxicity in vitro

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The extent of heterocellular c ...... er tumor cytotoxicity in vitro

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Proceedings of the National Academy of Sciences of the United States of America

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The extent of heterocellular c ...... er tumor cytotoxicity in vitro

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E M Westphale

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F G Barker

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J Fick

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M A Israel

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P2860

Connexin43: a protein from rat heart homologous to a gap junction protein from liver

In vitro evidence that metabolic cooperation is responsible for the bystander effect observed with HSV tk retroviral gene therapy.

Development of anti-tumor immunity following thymidine kinase-mediated killing of experimental brain tumors

Gene therapy of rat 9L gliosarcoma tumors by transduction with selectable genes does not require drug selection.

Specificity of gap junction communication among human mammary cells and connexin transfectants in culture.

Regression of established macroscopic liver metastases after in situ transduction of a suicide gene.

Hypoxanthine-guanine phosphoribosyltransferase deficiency: chemical agents selective for mutant or normal cultured fibroblasts in mixed and heterozygote cultures.

'Bystander killing' induces apoptosis and is inhibited by forskolin.

The "bystander effect": tumor regression when a fraction of the tumor mass is genetically modified.

Selective elimination of recombinant genes in vivo with a suicide retroviral vector.

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11071-11075

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15719040

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40573

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