Molecular mechanisms of cocaine reward: combined dopamine and serotonin transporter knockouts eliminate cocaine place preference - Wikidata - wikimedia - TriplyDB

Molecular mechanisms of cocaine reward: combined dopamine and serotonin transporter knockouts eliminate cocaine place preference

Molecular mechanisms of cocaine reward: combined dopamine and serotonin transporter knockouts eliminate cocaine place preference

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Depletion of serotonin and selective inhibition of 2B receptor suppressed tumor angiogenesis by inhibiting endothelial nitric oxide synthase and extracellular signal-regulated kinase 1/2 phosphorylation Neurotensin receptor antagonist administered during cocaine withdrawal decreases locomotor sensitization and conditioned place preference Kappa opioids as potential treatments for stimulant dependence Animal models of depression in dopamine, serotonin, and norepinephrine transporter knockout mice: prominent effects of dopamine transporter deletions Multiple ADH genes modulate risk for drug dependence in both African- and European-Americans RNA-binding proteins, neural development and the addictions Reward processing by the dorsal raphe nucleus: 5-HT and beyond Modulation of the endocannabinoid system: vulnerability factor and new treatment target for stimulant addiction Altered reward circuitry in the norepinephrine transporter knockout mouse Altered neurocircuitry in the dopamine transporter knockout mouse brain NrCAM in addiction vulnerability: positional cloning, drug-regulation, haplotype-specific expression, and altered drug reward in knockout mice Dopamine, learning and motivation Classic Studies on the Interaction of Cocaine and the Dopamine Transporter Serotonin/dopamine interactions in a hyperactive mouse: reduced serotonin receptor 1B activity reverses effects of dopamine transporter knockout Serotonergic involvement in the amelioration of behavioral abnormalities in dopamine transporter knockout mice by nicotine.Deletion of Go2alpha abolishes cocaine-induced behavioral sensitization by disturbing the striatal dopamine system How the serotonin story is being rewritten by new gene-based discoveries principally related to SLC6A4, the serotonin transporter gene, which functions to influence all cellular serotonin systems.Dramatically decreased cocaine self-administration in dopamine but not serotonin transporter knock-out mice.Activation of the kappa opioid receptor in the dorsal raphe nucleus mediates the aversive effects of stress and reinstates drug seeking.NrCAM-regulating neural systems and addiction-related behaviors.Characterization of Highper, an ENU-induced mouse mutant with abnormal psychostimulant and stress responses.Dorsal raphe neurons signal reward through 5-HT and glutamate.Cyclin-Dependent Kinase Inhibitor 1a (p21) Modulates Response to Cocaine and Motivated Behaviors.The addicted human brain: insights from imaging studies.Functional neuroimaging of genetic variation in serotonergic neurotransmission.Cocaine modulates locomotion behavior in C. elegans.Distinct Roles of Opioid and Dopamine Systems in Lateral Hypothalamic Intracranial Self-Stimulation.Drosophila, a genetic model system to study cocaine-related behaviors: a review with focus on LIM-only proteins.The cannabinoid CB2 receptor is necessary for nicotine-conditioned place preference, but not other behavioral effects of nicotine in mice Using In Vivo Electrochemistry to Study the Physiological Effects of Cocaine and Other Stimulants on the Drosophila melanogaster Dopamine Transporter.Boron-Doped Diamond Microelectrodes Reveal Reduced Serotonin Uptake Rates in Lymphocytes from Adult Rhesus Monkeys Carrying the Short Allele of the 5-HTTLPR.Behavioral screening for cocaine sensitivity in mutagenized zebrafish.Dietary restriction mitigates cocaine-induced alterations of olfactory bulb cellular plasticity and gene expression, and behavior.DARPP-32 mediates serotonergic neurotransmission in the forebrain.Dissociation of rewarding and dopamine transporter-mediated properties of amphetamine.Identification and characterization of a novel monoamine transporter in the human brain.Implications of genome wide association studies for addiction: are our a priori assumptions all wrong?Genetic susceptibility to substance dependence.The Nociceptin Receptor as an Emerging Molecular Target for Cocaine Addiction Sugar addiction: the state of the science.

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Molecular mechanisms of cocaine reward: combined dopamine and serotonin transporter knockouts eliminate cocaine place preference

http://www.wikidata.org/entity/Q24603526

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2001 nî lūn-bûn

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2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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Molecular mechanisms of cocain ...... inate cocaine place preference

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Molecular mechanisms of cocain ...... inate cocaine place preference

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Molecular mechanisms of cocain ...... inate cocaine place preference

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Molecular mechanisms of cocain ...... inate cocaine place preference

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Molecular mechanisms of cocain ...... inate cocaine place preference

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Proceedings of the National Academy of Sciences of the United States of America

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Molecular mechanisms of cocain ...... inate cocaine place preference

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A M Andrews

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C Wichems

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P2860

Hyperlocomotion and indifference to cocaine and amphetamine in mice lacking the dopamine transporter

Cocaine reward models: conditioned place preference can be established in dopamine- and in serotonin-transporter knockout mice

Anterior pituitary hypoplasia and dwarfism in mice lacking the dopamine transporter

Altered brain serotonin homeostasis and locomotor insensitivity to 3, 4-methylenedioxymethamphetamine ("Ecstasy") in serotonin transporter-deficient mice

Absence of opiate rewarding effects in mice lacking dopamine D2 receptors

Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats

RU 24969, a 5-HT1A/1B agonist, elevates brain stimulation reward thresholds: an effect reversed by GR 127935, a 5-HT1B/1D antagonist.

Cocaine self-administration in dopamine-transporter knockout mice.

Addiction and its reward process through polymorphisms of the D2 dopamine receptor gene: a review.

[3H]nisoxetine: a new radioligand for norepinephrine uptake sites in brain.

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