Novel monoamine oxidase A knock out mice with human-like spontaneous mutation - Wikidata - wikimedia - TriplyDB

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

http://www.wikidata.org/entity/Q24606127

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Monoamine oxidase A and A/B knockout mice display autistic-like features.Valproic acid induces monoamine oxidase A via Akt/forkhead box O1 activation Behavioral disinhibition and reduced anxiety-like behaviors in monoamine oxidase B-deficient mice The aggression and behavioral abnormalities associated with monoamine oxidase A deficiency are rescued by acute inhibition of serotonin reuptake.Monoamine-sensitive developmental periods impacting adult emotional and cognitive behaviors.Animal models of tic disorders: a translational perspective.Early postnatal inhibition of serotonin synthesis results in long-term reductions of perseverative behaviors, but not aggression, in MAO A-deficient mice Behavioral and pharmacogenetics of aggressive behavior.Brain serotonin receptors and transporters: initiation vs. termination of escalated aggression Pharmacological insights into the role of P2X4 receptors in behavioural regulation: lessons from ivermectin.Social deficits and perseverative behaviors, but not overt aggression, in MAO-A hypomorphic mice Behavioral outcomes of monoamine oxidase deficiency: preclinical and clinical evidence.Creatine transporter (CrT; Slc6a8) knockout mice as a model of human CrT deficiency Monoamine oxidase inactivation: from pathophysiology to therapeutics De novo microdeletion of Xp11.3 exclusively encompassing the monoamine oxidase A and B genes in a male infant with episodic hypotonia: a genomics approach to personalized medicine.Dopamine and serotonin signaling during two sensitive developmental periods differentially impact adult aggressive and affective behaviors in mice.20 ans après: a second mutation in MAOA identified by targeted high-throughput sequencing in a family with altered behavior and cognition.Monoamine oxidases as sources of oxidants in the heart Serotonin, genetic variability, behaviour, and psychiatric disorders--a review Neurogenetics of aggressive behavior: studies in rodents.Prenatal exposure to urban air nanoparticles in mice causes altered neuronal differentiation and depression-like responses.A transient placental source of serotonin for the fetal forebrain A medium density genetic map and QTL for behavioral and production traits in Japanese quail.Transcriptional regulation and multiple functions of MAO genes.Altered expression of tyrosine hydroxylase in the locus coeruleus noradrenergic system in citalopram neonatally exposed rats and monoamine oxidase a knock out mice.Insights into the genetic foundation of aggression in Papio and the evolution of two length-polymorphisms in the promoter regions of serotonin-related genes (5-HTTLPR and MAOALPR) in Papionini.Lower Monoamine Oxidase-A Total Distribution Volume in Impulsive and Violent Male Offenders with Antisocial Personality Disorder and High Psychopathic Traits: An [(11)C] Harmine Positron Emission Tomography Study NMDARs mediate the role of monoamine oxidase A in pathological aggression.Monoamine oxidase A regulates neural differentiation of murine embryonic stem cells Maladaptive defensive behaviours in monoamine oxidase A-deficient mice.Altered gene expression in early postnatal monoamine oxidase A knockout mice.The role of the serotonergic system at the interface of aggression and suicide Regulation of monoamine oxidase A by the SRY gene on the Y chromosome.Monoamine oxidases in development.From depression to neurodegeneration and heart failure: re-examining the potential of MAO inhibitors.Olfaction and olfactory-mediated behaviour in psychiatric disease models.Modulation of monoamine oxidase (MAO) expression in neuropsychiatric disorders: genetic and environmental factors involved in type A MAO expression.Serotonin receptor 6 mediates defective brain development in monoamine oxidase A-deficient mouse embryos.The neurobiology of aggression and violence.Aggression in non-human vertebrates: Genetic mechanisms and molecular pathways.

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P2860

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

http://www.wikidata.org/entity/Q24606127

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2008 թվականի մայիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

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Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@en

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@nl

type

label

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@ast

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@en

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@nl

prefLabel

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@ast

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@en

Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

@nl

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Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

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P2093

Anna L Scott

http://www.w3.org/2001/XMLSchema#string

Jean C Shih

http://www.w3.org/2001/XMLSchema#string

Kevin Chen

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Marco Bortolato

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P2860

Abnormal behavior associated with a point mutation in the structural gene for monoamine oxidase A

Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA

cDNA cloning of human liver monoamine oxidase A and B: molecular basis of differences in enzymatic properties

The deamination of dopamine by human brain monoamine oxidase. Specificity for the two enzyme forms in seven brain regions.

Increased stress response and beta-phenylethylamine in MAOB-deficient mice

Site-directed mutagenesis of monoamine oxidase A and B: role of cysteines

Specification of the phenotype required for men with monoamine oxidase type A deficiency

Genetic mouse models of parkinsonism: strengths and limitations.

Monoamine oxidase in neuropsychiatry and behavior.

A spontaneous point mutation produces monoamine oxidase A/B knock-out mice with greatly elevated monoamines and anxiety-like behavior.

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