Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration - Wikidata - wikimedia - TriplyDB

Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration

Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration

http://www.wikidata.org/entity/Q24617620

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Small RNA and transcriptional upregulation Upregulation of Cyclin B1 by miRNA and its implications in cancer Cancer Stem Cell Hierarchy in Glioblastoma Multiforme Regulation of mammalian transcription and splicing by Nuclear RNAi The Krüppel-like factors in female reproductive system pathologies MiR-145 negatively regulates Warburg effect by silencing KLF4 and PTBP1 in bladder cancer cells A novel KLF4/LDHA signaling pathway regulates aerobic glycolysis in and progression of pancreatic cancer Cell-to-cell signaling influences the fate of prostate cancer stem cells and their potential to generate more aggressive tumors.Critical and reciprocal regulation of KLF4 and SLUG in transforming growth factor β-initiated prostate cancer epithelial-mesenchymal transition Fibulin-1 is epigenetically down-regulated and related with bladder cancer recurrence Formulation of Small Activating RNA Into Lipidoid Nanoparticles Inhibits Xenograft Prostate Tumor Growth by Inducing p21 Expression.Gene Expression Profile Changes After Short-activating RNA-mediated Induction of Endogenous Pluripotency Factors in Human Mesenchymal Stem Cells Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression.The varied roles of nuclear argonaute-small RNA complexes and avenues for therapy.Krüppel-like factor 4 induces apoptosis and inhibits tumorigenic progression in SK-BR-3 breast cancer cells.Inducing gene expression by targeting promoter sequences using small activating RNAs.saRNA-guided Ago2 targets the RITA complex to promoters to stimulate transcription Enhancing DPYSL3 gene expression via a promoter-targeted small activating RNA approach suppresses cancer cell motility and metastasis.KLF4 overexpression and apigenin treatment down regulated anti-apoptotic Bcl-2 proteins and matrix metalloproteinases to control growth of human malignant neuroblastoma SK-N-DZ and IMR-32 cells MALAT1 promotes colorectal cancer cell proliferation/migration/invasion via PRKA kinase anchor protein 9.Attenuation of krüppel-like factor 4 facilitates carcinogenesis by inducing g1/s phase arrest in clear cell renal cell carcinoma Acetylation-dependent regulation of essential iPS-inducing factors: a regulatory crossroad for pluripotency and tumorigenesis MicroRNA miR-324-3p induces promoter-mediated expression of RelA gene Krüppel-like factor 4 (KLF4) activates the transcription of the gene for the platelet isoform of phosphofructokinase (PFKP) in breast cancer.down-regulation of Kruppel-like factor-4 (KLF4) by microRNA-143/145 is critical for modulation of vascular smooth muscle cell phenotype by transforming growth factor-beta and bone morphogenetic protein 4 Intelectin 1 suppresses the growth, invasion and metastasis of neuroblastoma cells through up-regulation of N-myc downstream regulated gene 2 RNAa-mediated overexpression of WT1 induces apoptosis in HepG2 cells.Small RNA and its application in andrology and urology Targeted p21WAF1/CIP1 activation by RNAa inhibits hepatocellular carcinoma cells.F-box protein FBXO22 mediates polyubiquitination and degradation of KLF4 to promote hepatocellular carcinoma progression.Downregulation of KLF4 and the Bcl-2/Bax ratio in advanced epithelial ovarian cancer.Molecular profiling of prostatic acinar morphogenesis identifies PDCD4 and KLF6 as tissue architecture-specific prognostic markers in prostate cancer.Small activating RNA binds to the genomic target site in a seed-region-dependent manner.Loss of epigenetic Kruppel-like factor 4 histone deacetylase (KLF-4-HDAC)-mediated transcriptional suppression is crucial in increasing vascular endothelial growth factor (VEGF) expression in breast cancer.Demystifying the mechanistic and functional aspects of p21 gene activation with double-stranded RNAs in human cancer cells.KLF4 functions as an activator of the androgen receptor through reciprocal feedback.KLF4α stimulates breast cancer cell proliferation by acting as a KLF4 antagonist.KLF4 downregulates hTERT expression and telomerase activity to inhibit lung carcinoma growth.Krüppel-like factors in cancer progression: three fingers on the steering wheel.Transcription factor decoy: a pre-transcriptional approach for gene downregulation purpose in cancer.

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Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration

http://www.wikidata.org/entity/Q24617620

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Prognostic value and function ...... umor cell growth and migration

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Clara E Magyar

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Emily J Noonan

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Ji Wang

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Jiaoti Huang

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Long-Cheng Li

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Robert F Place

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Vera Huang

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Xiaoling Wang

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P2860

RNAa is conserved in mammalian cells

Identification and characterization of a gene encoding a gut-enriched Krüppel-like factor expressed during growth arrest

Transcriptional profiling of Krüppel-like factor 4 reveals a function in cell cycle regulation and epithelial differentiation

Overexpression of Krüppel-like factor 4 in the human colon cancer cell line RKO leads to reduced tumorigenecity

MicroRNA-373 induces expression of genes with complementary promoter sequences

Kruppel-like factor 4 mediates p53-dependent G1/S cell cycle arrest in response to DNA damage

Small dsRNAs induce transcriptional activation in human cells

Downregulation and growth inhibitory effect of epithelial-type Krüppel-like transcription factor KLF4, but not KLF5, in bladder cancer

p57KIP2, a structurally distinct member of the p21CIP1 Cdk inhibitor family, is a candidate tumor suppressor gene

Krüppel-like factor 4 (gut-enriched Krüppel-like factor) inhibits cell proliferation by blocking G1/S progression of the cell cycle

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24

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