about
Analysis of the genome to personalize therapy for melanomaMelanoma: oncogenic drivers and the immune systemMelanoma: new insights and new therapiesRas oncogenes: split personalitiesRationale for RAS mutation-tailored therapies.Activation of ras oncogene in aflatoxin-induced rat liver carcinogenesis.The dual RAF/MEK inhibitor CH5126766/RO5126766 may be a potential therapy for RAS-mutated tumor cells.Combinatorial treatments that overcome PDGFRβ-driven resistance of melanoma cells to V600EB-RAF inhibition.Molecular bases of cutaneous and uveal melanomas.Relevance of ultraviolet-induced N-ras oncogene point mutations in development of primary human cutaneous melanomaN-ras mutations in human cutaneous melanoma from sun-exposed body sites.Models and mechanisms in malignant melanoma.Applications of genomics in melanoma oncogene discoveryHuman cutaneous melanoma; a review of NRAS and BRAF mutation frequencies in relation to histogenetic subclass and body site.Structural alteration in the MYB protooncogene and deletion within the gene encoding alpha-type protein kinase C in human melanoma cell lines.Melanoma biomarkers: current status and utility in diagnosis, prognosis, and response to therapy.Measurement of constitutive MAPK and PI3K/AKT signaling activity in human cancer cell linesMutation-driven drug development in melanoma.Molecular mechanisms of ultraviolet radiation carcinogenesis.Genetic alterations and personalized medicine in melanoma: progress and future prospects.A novel multi-CDK inhibitor P1446A-05 restricts melanoma growth and produces synergistic effects in combination with MAPK pathway inhibitors.Cyclobutane thymine dimers in a ras proto-oncogene hot spot activate the gene by point mutation.Point mutation in codon 61 of N-RAS genes in human myeloma cell linesHuman malignant melanoma. A genetic disease?Mutational analysis of the human p53 gene in malignant melanoma.Ras gene mutation and amplification in human nonmelanoma skin cancers.Transformation of murine melanocytes by basic fibroblast growth factor cDNA and oncogenes and selective suppression of the transformed phenotype in a reconstituted cutaneous environmentInfluence of Ki-ras-driven oncogenic transformation on the protein network of murine fibroblasts.Lack of evidence of Ki-ras codon 12 mutations in melanocytic lesions.H-ras ribozyme-mediated alteration of the human melanoma phenotype.Both N-ras and c-myc are activated in the SHAC human stomach fibrosarcoma cell line.Genetic recombination in human melanoma and astrocytoma cell lines involves oncogenes and growth factor genes.Proto-oncogene expression in dermal naevi and melanomas
P2860
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P2860
description
1985 nî lūn-bûn
@nan
1985 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1985 թվականի մարտին հրատարակված գիտական հոդված
@hy
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
name
Activation of N-ras in a human melanoma cell line
@ast
Activation of N-ras in a human melanoma cell line
@en
Activation of N-ras in a human melanoma cell line
@nl
type
label
Activation of N-ras in a human melanoma cell line
@ast
Activation of N-ras in a human melanoma cell line
@en
Activation of N-ras in a human melanoma cell line
@nl
prefLabel
Activation of N-ras in a human melanoma cell line
@ast
Activation of N-ras in a human melanoma cell line
@en
Activation of N-ras in a human melanoma cell line
@nl
P2093
P2860
P356
P1476
Activation of N-ras in a human melanoma cell line
@en
P2093
G A Currie
N C Barrass
P2860
P356
10.1128/MCB.5.3.582
P407
P577
1985-03-01T00:00:00Z