Cyanobacterial blooms and the occurrence of the neurotoxin, beta-N-methylamino-l-alanine (BMAA), in South Florida aquatic food webs - Wikidata - wikimedia - TriplyDB

Cyanobacterial blooms and the occurrence of the neurotoxin, beta-N-methylamino-l-alanine (BMAA), in South Florida aquatic food webs

Cyanobacterial blooms and the occurrence of the neurotoxin, beta-N-methylamino-l-alanine (BMAA), in South Florida aquatic food webs

http://www.wikidata.org/entity/Q24618334

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Dietary BMAA exposure in an amyotrophic lateral sclerosis cluster from southern France Maternal transfer of the cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) via milk to suckling offspring The emerging science of BMAA: do cyanobacteria contribute to neurodegenerative disease?The cyanobacteria derived toxin Beta-N-methylamino-L-alanine and amyotrophic lateral sclerosis Cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) in shark fins Dietary exposure to an environmental toxin triggers neurofibrillary tangles and amyloid deposits in the brain Unraveling 50-Year-Old Clues Linking Neurodegeneration and Cancer to Cycad Toxins: Are microRNAs Common Mediators?Searching for a link between the L-BMAA neurotoxin and amyotrophic lateral sclerosis: a study protocol of the French BMAALS programme Mapping amyotrophic lateral sclerosis lake risk factors across northern New England Cyanobacterial Neurotoxin BMAA and Mercury in Sharks Marine harmful algal blooms, human health and wellbeing: challenges and opportunities in the 21st century β-N-Methylamino-L-Alanine Toxicity in PC12: Excitotoxicity vs. Misincorporation.Neonatal exposure to the cyanobacterial toxin BMAA induces changes in protein expression and neurodegeneration in adult hippocampus Current pathways for epidemiological research in amyotrophic lateral sclerosis.High resolution metabolite imaging in the hippocampus following neonatal exposure to the environmental toxin BMAA using ToF-SIMS Diatoms: a novel source for the neurotoxin BMAA in aquatic environments.Biotransfer of β-N-methylamino-L-alanine (BMAA) in a eutrophicated freshwater lake.Cyanotoxins: bioaccumulation and effects on aquatic animals Exposure to environmental toxicants and pathogenesis of amyotrophic lateral sclerosis: state of the art and research perspectives.New Typical Vector of Neurotoxin β-N-Methylamino-l-Alanine (BMAA) in the Marine Benthic Ecosystem.The John Walton Muscular Dystrophy Research Centre in the University of Newcastle and the BMAA Theory of Motor Neuron Disease Co-occurrence of the cyanotoxins BMAA, DABA and anatoxin-a in Nebraska reservoirs, fish, and aquatic plants Presence of the neurotoxin BMAA in aquatic ecosystems: what do we really know?Challenges and trends in the determination of selected chemical contaminants and allergens in food.Does α-amino-β-methylaminopropionic acid (BMAA) play a role in neurodegeneration?Good mass spectrometry and its place in good science.Is exposure to cyanobacteria an environmental risk factor for amyotrophic lateral sclerosis and other neurodegenerative diseases?The effects of the toxic cyanobacterium Limnothrix (strain AC0243) on Bufo marinus larvae.Assessing Environmental Exposure to β-N-Methylamino-L-Alanine (BMAA) in Complex Sample Matrices: a Comparison of the Three Most Popular LC-MS/MS Methods.A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.Assessing Cyanobacterial Harmful Algal Blooms as Risk Factors for Amyotrophic Lateral Sclerosis.Analysis of Neurotoxic Amino Acids from Marine Waters, Microbial Mats, and Seafood Destined for Human Consumption in the Arabian Gulf.Overview of the potent cyanobacterial neurotoxin β-methylamino-L-alanine (BMAA) and its analytical determination.Quantification of neurotoxin BMAA (β-N-methylamino-L-alanine) in seafood from Swedish markets.Cyanobacterial Neurotoxins: Their Occurrence and Mechanisms of Toxicity.Studies of Environmental Risk Factors in Amyotrophic Lateral Sclerosis (ALS) and a Phase I Clinical Trial of L-Serine.Assessment of the non-protein amino acid BMAA in Mediterranean mussel Mytilus galloprovincialis after feeding with estuarine cyanobacteria.Seafood sold in Sweden contains BMAA: A study of free and total concentrations with UHPLC-MS/MS and dansyl chloride derivatization.Environmental neurotoxin interaction with proteins: Dose-dependent increase of free and protein-associated BMAA (β-N-methylamino-L-alanine) in neonatal rat brain.Intrathecal infusion of BMAA induces selective motor neuron damage and astrogliosis in the ventral horn of the spinal cord.

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Cyanobacterial blooms and the occurrence of the neurotoxin, beta-N-methylamino-l-alanine (BMAA), in South Florida aquatic food webs

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2010 nî lūn-bûn

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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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Angela Compton

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P2860

Human fatalities from cyanobacteria: chemical and biological evidence for cyanotoxins

Biomagnification of cycad neurotoxins in flying foxes: implications for ALS-PDC in Guam

Cycad neurotoxins, consumption of flying foxes, and ALS-PDC disease in Guam

Diverse taxa of cyanobacteria produce beta-N-methylamino-L-alanine, a neurotoxic amino acid

BMAA selectively injures motor neurons via AMPA/kainate receptor activation

Neurotoxicity of beta-N-methylamino-L-alanine (BMAA) and beta-N-oxalylamino-L-alanine (BOAA) on cultured cortical neurons

Beta-N-methylamino-L-alanine enhances neurotoxicity through multiple mechanisms

A mechanism for slow release of biomagnified cyanobacterial neurotoxins and neurodegenerative disease in Guam

Occurrence of beta-methylamino-l-alanine (BMAA) in ALS/PDC patients from Guam

Beta-N-methylamino-L-alanine (BMAA) in novel South African cyanobacterial isolates

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