DNase-seq: a high-resolution technique for mapping active gene regulatory elements across the genome from mammalian cells - Wikidata - wikimedia - TriplyDB

DNase-seq: a high-resolution technique for mapping active gene regulatory elements across the genome from mammalian cells

DNase-seq: a high-resolution technique for mapping active gene regulatory elements across the genome from mammalian cells

http://www.wikidata.org/entity/Q24633746

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Current bioinformatic approaches to identify DNase I hypersensitive sites and genomic footprints from DNase-seq data Chromatin accessibility data sets show bias due to sequence specificity of the DNase I enzyme Uncovering correlated variability in epigenomic datasets using the Karhunen-Loeve transform Classification of human genomic regions based on experimentally determined binding sites of more than 100 transcription-related factors Discovery of active enhancers through bidirectional expression of short transcripts A comparison of peak callers used for DNase-Seq data High-resolution genome-wide in vivo footprinting of diverse transcription factors in human cells Chromatin: constructing the big picture Next generation sequencing technology and genomewide data analysis: Perspectives for retinal research Next-Generation Sequencing Approaches in Cancer: Where Have They Brought Us and Where Will They Take Us?Transcriptional Enhancers in the Regulation of T Cell Differentiation Chromatin Dynamics in the Regulation of CFTR Expression Epigenetics, chromatin and genome organization: recent advances from the ENCODE project Cancer genome sequencing: understanding malignancy as a disease of the genome, its conformation, and its evolution.Integrating Epigenomics into the Understanding of Biomedical Insight Statistical Methods in Integrative Genomics Epigenomic comparison reveals activation of "seed" enhancers during transition from naive to primed pluripotency Combining Hi-C data with phylogenetic correlation to predict the target genes of distal regulatory elements in human genome.Removing reference mapping biases using limited or no genotype data identifies allelic differences in protein binding at disease-associated loci.DNA-protein interactions: methods for detection and analysis.Identifying peaks in *-seq data using shape information.Cistrome Data Browser: a data portal for ChIP-Seq and chromatin accessibility data in human and mouse Combining ATAC-seq with nuclei sorting for discovery of cis-regulatory regions in plant genomes.Folate deficiency facilitates recruitment of upstream binding factor to hot spots of DNA double-strand breaks of rRNA genes and promotes its transcription TargetOrtho: a phylogenetic footprinting tool to identify transcription factor targets.Chromosomal dynamics predicted by an elastic network model explains genome-wide accessibility and long-range couplings Application of a systems approach to study developmental gene regulation.Dynamic chromatin accessibility modeled by Markov process of randomly-moving molecules in the 3D genome Prediction of Chromatin Accessibility in Gene-Regulatory Regions from Transcriptomics Data.Open chromatin defined by DNaseI and FAIRE identifies regulatory elements that shape cell-type identity.Using formaldehyde-assisted isolation of regulatory elements (FAIRE) to isolate active regulatory DNA.Global mapping of cell type-specific open chromatin by FAIRE-seq reveals the regulatory role of the NFI family in adipocyte differentiation.Epigenetic landscape during osteoblastogenesis defines a differentiation-dependent Runx2 promoter region.Microbiota modulate transcription in the intestinal epithelium without remodeling the accessible chromatin landscape.Interrogation of allelic chromatin states in human cells by high-density ChIP-genotyping.Dual role of FoxA1 in androgen receptor binding to chromatin, androgen signalling and prostate cancer The bone-specific Runx2-P1 promoter displays conserved three-dimensional chromatin structure with the syntenic Supt3h promoter.Deletions of chromosomal regulatory boundaries are associated with congenital disease.CNV-guided multi-read allocation for ChIP-seq Unbiased, genome-wide in vivo mapping of transcriptional regulatory elements reveals sex differences in chromatin structure associated with sex-specific liver gene expression

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DNase-seq: a high-resolution technique for mapping active gene regulatory elements across the genome from mammalian cells

http://www.wikidata.org/entity/Q24633746

description

2010 nî lūn-bûn

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2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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Cold Spring Harbor Protocols

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DNase-seq: a high-resolution t ...... he genome from mammalian cells

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Lingyun Song

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Identification and analysis of functional elements in 1% of the human genome by the ENCODE pilot project

DNase-chip: a high-resolution method to identify DNase I hypersensitive sites using tiled microarrays

High-resolution mapping and characterization of open chromatin across the genome

Identification and characterization of cell type-specific and ubiquitous chromatin regulatory structures in the human genome.

Nuclease hypersensitive sites in chromatin.

The chromatin structure of specific genes: II. Disruption of chromatin structure during gene activity.

DNase I hypersensitive sites in Drosophila chromatin occur at the 5' ends of regions of transcription.

Global mapping of protein-DNA interactions in vivo by digital genomic footprinting.

Genome-scale mapping of DNase I sensitivity in vivo using tiling DNA microarrays.

Mapping regulatory elements by DNaseI hypersensitivity chip (DNase-Chip).

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pdb.prot5384

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539336

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10.1101/PDB.PROT5384

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2010

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Gregory E Crawford

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41424558

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20150147

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3627383

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