Apoptosis: a mechanism of cell killing by influenza A and B viruses
about
Influenza virus PB1-F2 protein induces cell death through mitochondrial ANT3 and VDAC1.Protection against fatal Sindbis virus encephalitis by beclin, a novel Bcl-2-interacting proteinLung epithelial cells resist influenza A infection by inducing the expression of cytochrome c oxidase VIc which is modulated by miRNA 4276PD-L1 expression induced by the 2009 pandemic influenza A(H1N1) virus impairs the human T cell responseNonvirion protein of novirhabdovirus suppresses apoptosis at the early stage of virus infectionClonogenic assay of type a influenza viruses reveals noninfectious cell-killing (apoptosis-inducing) particlesAdapting the Stress Response: Viral Subversion of the mTOR Signaling PathwayRole of Autophagy and Apoptosis in the Postinfluenza Bacterial PneumoniaAntiapoptotic but not antiviral function of human bcl-2 assists establishment of Japanese encephalitis virus persistence in cultured cellsLa Crosse Bunyavirus Nonstructural Protein NSs Serves To Suppress the Type I Interferon System of Mammalian HostsTemporal changes in chromatin, intracellular calcium, and poly(ADP-ribose) polymerase during Sindbis virus-induced apoptosis of neuroblastoma cellsEffect of enforced expression of human bcl-2 on Japanese encephalitis virus-induced apoptosis in cultured cellsGlycoprotein Erns of pestiviruses induces apoptosis in lymphocytes of several speciesNovel packages of viral and self-antigens are generated during apoptosisCompounds with anti-influenza activity: present and future of strategies for the optimal treatment and management of influenza. Part II: Future compounds against influenza virusA review of mathematical models of influenza A infections within a host or cell culture: lessons learned and challenges aheadHost defense mechanism-based rational design of live vaccineEarly host responses of seasonal and pandemic influenza A viruses in primary well-differentiated human lung epithelial cellsMulti-organ lesions in suckling mice infected with SARS-associated mammalian reovirus linked with apoptosis induced by viral proteins μ1 and σ1Drosophila RNAi screen identifies host genes important for influenza virus replicationH5N1 avian influenza virus induces apoptotic cell death in mammalian airway epithelial cells.2009 H1N1 influenza.Pandemic Influenza A (H1N1) Virus Infection Increases Apoptosis and HIV-1 Replication in HIV-1 Infected Jurkat Cells.Networks of Host Factors that Interact with NS1 Protein of Influenza A VirusAttenuated Bordetella pertussis protects against highly pathogenic influenza A viruses by dampening the cytokine storm.Highly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.Influenza A virus replication induces cell cycle arrest in G0/G1 phaseHigher level of replication efficiency of 2009 (H1N1) pandemic influenza virus than those of seasonal and avian strains: kinetics from epithelial cell culture and computational modelingAfrican green monkey kidney (Vero) cells provide an alternative host cell system for influenza A and B virusesThe use of Random Homozygous Gene Perturbation to identify novel host-oriented targets for influenza.RNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cellsInduction of apoptosis in murine coronavirus-infected cultured cells and demonstration of E protein as an apoptosis inducer.Mechanisms of viral pathogenesis in rheumatic diseaseInfluenza virus infection decreases tracheal mucociliary velocity and clearance of Streptococcus pneumoniaeTransmissible gastroenteritis coronavirus induces programmed cell death in infected cells through a caspase-dependent pathway.Highly pathogenic avian influenza A virus H5N1 NS1 protein induces caspase-dependent apoptosis in human alveolar basal epithelial cellsAlpha/beta interferons potentiate virus-induced apoptosis through activation of the FADD/Caspase-8 death signaling pathwayHuman immunodeficiency virus type 1 vpr induces apoptosis through caspase activationDose-dependent changes in influenza virus-infected dendritic cells result in increased allogeneic T-cell proliferation at low, but not high, doses of virus.Competing death programs in poliovirus-infected cells: commitment switch in the middle of the infectious cycle.
P2860
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P2860
Apoptosis: a mechanism of cell killing by influenza A and B viruses
description
1994 nî lūn-bûn
@nan
1994 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@ast
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@en
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@nl
type
label
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@ast
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@en
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@nl
prefLabel
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@ast
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@en
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@nl
P2093
P2860
P3181
P1433
P1476
Apoptosis: a mechanism of cell killing by influenza A and B viruses
@en
P2093
N Dybdahl-Sissoko
V S Hinshaw
P2860
P304
P3181
P407
P577
1994-06-01T00:00:00Z