A non-additive interaction of a functional MAO-A VNTR and testosterone predicts antisocial behavior - Wikidata - wikimedia - TriplyDB

A non-additive interaction of a functional MAO-A VNTR and testosterone predicts antisocial behavior

A non-additive interaction of a functional MAO-A VNTR and testosterone predicts antisocial behavior

http://www.wikidata.org/entity/Q24646593

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A population-specific HTR2B stop codon predisposes to severe impulsivity The genetic basis of addictive disorders The dominance behavioral system and psychopathology: evidence from self-report, observational, and biological studies Genetics of impulsive behaviour Candidate genes for aggression and antisocial behavior: a meta-analysis of association studies of the 5HTTLPR and MAOA-uVNTR Autism severity is associated with child and maternal MAOA genotypes Gonadectomy and hormone replacement exert region- and enzyme isoform-specific effects on monoamine oxidase and catechol-O-methyltransferase activity in prefrontal cortex and neostriatum of adult male rats Developmental changes in genetic and environmental influences on rule-breaking and aggression: age and pubertal development Early life stress, MAOA, and gene-environment interactions predict behavioral disinhibition in children.Functional variation of the dopamine D2 receptor gene is associated with emotional control as well as brain activity and connectivity during emotion processing in humans.The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review.The 'antisocial' person: an insight in to biology, classification and current evidence on treatment Gene x environment interactions in complex behavior: First, build a telescope.Gene-sex interactions in schizophrenia: focus on dopamine neurotransmission.The heterogeneity of disruptive behavior disorders - implications for neurobiological research and treatment Neural mechanisms of the testosterone-aggression relation: the role of orbitofrontal cortex.The inclination to evil and the punishment of crime - from the bible to behavioral genetics.Association of low-activity MAOA allelic variants with violent crime in incarcerated offenders Influence of Altered Hormonal Status on Platelet 5-HT and MAO-B Activity in Cigarette Smokers.Child abuse and neglect, MAOA, and mental health outcomes: a prospective examination.Expanding our lens: female pathways to antisocial behavior in adolescence and adulthood.Interaction between FKBP5 and childhood trauma and risk of aggressive behavior.Does MAOA increase susceptibility to prenatal stress in young children?Monoamine oxidase A genotype predicts human serotonin 1A receptor availability in vivo.Genetic approaches to addiction: genes and alcohol.Habits of the heart: life history and the developmental neuroendocrinology of emotion Impulsive alcohol-related risk-behavior and emotional dysregulation among individuals with a serotonin 2B receptor stop codon Cortical enlargement in autism is associated with a functional VNTR in the monoamine oxidase A gene MAOA, childhood maltreatment, and antisocial behavior: meta-analysis of a gene-environment interaction.Effects of MAOA-genotype, alcohol consumption, and aging on violent behavior.The psychological neuroscience of depression: implications for understanding effects of deep brain stimulation.Studying longitudinal trajectories in animal models of psychiatric illness and their translation to the human condition Lack of association between MAOA-uVNTR variants and excessive daytime sleepiness.Sex Differences in Sources of Resilience and Vulnerability to Risk for Delinquency.Type A and B monoamine oxidases distinctly modulate signal transduction pathway and gene expression to regulate brain function and survival of neurons.A functional polymorphism of the MAOA gene is associated with neural responses to induced anger control.Blunted insula activation reflects increased risk and reward seeking as an interaction of testosterone administration and the MAOA polymorphism.The c.1460C>T polymorphism of MAO-A is associated with the risk of depression in postmenopausal women.Early-life adversity-induced long-term epigenetic programming associated with early onset of chronic physical aggression: Studies in humans and animals.Monoamine Oxidase A in Antisocial Personality Disorder and Borderline Personality Disorder.

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P2860

A non-additive interaction of a functional MAO-A VNTR and testosterone predicts antisocial behavior

http://www.wikidata.org/entity/Q24646593

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2008 nî lūn-bûn

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2008年の論文

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A non-additive interaction of ...... e predicts antisocial behavior

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A non-additive interaction of ...... e predicts antisocial behavior

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A non-additive interaction of ...... e predicts antisocial behavior

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A non-additive interaction of ...... e predicts antisocial behavior

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Neuropsychopharmacology

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A non-additive interaction of ...... e predicts antisocial behavior

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Christina S Barr

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Francesca Ducci

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Liliana Dell'osso

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Matti Virkkunen

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Rickard L Sjöberg

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Timothy K Newman

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P2860

X-inactivation profile reveals extensive variability in X-linked gene expression in females

Abnormal behavior associated with a point mutation in the structural gene for monoamine oxidase A

Glucocorticoid and androgen activation of monoamine oxidase A is regulated differently by R1 and Sp1

Role of genotype in the cycle of violence in maltreated children

Cloning, after cloning, knock-out mice, and physiological functions of MAO A and B

A functional polymorphism in the monoamine oxidase A gene promoter

MAOA, maltreatment, and gene–environment interaction predicting children's mental health: new evidence and a meta-analysis

Cerebrospinal fluid and behavioral changes after methyltestosterone administration: preliminary findings.

Hormones in male sexual development.

Sex differences in the neural basis of emotional memories.

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10.1038/SJ.NPP.1301417

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2

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33

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1014698673

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17429405

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2665792

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