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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

A rationale for lowering the blood lead action level from 10 to 2 microg/dL

http://www.wikidata.org/entity/Q24647248

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Lead bullet fragments in venison from rifle-killed deer: potential for human dietary exposure Electrochemical sensors for the detection of lead and other toxic heavy metals: the next generation of personal exposure biomonitors Environmental determinants of chronic disease and medical approaches: recognition, avoidance, supportive therapy, and detoxification A review on the importance of metals and metalloids in atmospheric dust and aerosol from mining operations Perinatal lead (Pb) exposure results in sex-specific effects on food intake, fat, weight, and insulin response across the murine life-course Developmental neurotoxicity: some old and new issues Lead, manganese, and methylmercury as risk factors for neurobehavioral impairment in advanced age Lead exposure in Nunavik: from research to action Mechanisms of lead and manganese neurotoxicity Chronic low-level lead exposure affects the monoaminergic system in the mouse superior olivary complex Metal concentrations in cosmetics commonly used in Nigeria Early-life lead exposure results in dose- and sex-specific effects on weight and epigenetic gene regulation in weanling mice Blood lead levels and associated sociodemographic factors among preschool children in the South Eastern region of China Perinatal Lead Exposure Alters Gut Microbiota Composition and Results in Sex-specific Bodyweight Increases in Adult Mice Lead and children: clinical management for family physicians Blood levels of the heavy metal, lead, and caries in children aged 24-72 months: NHANES III Neuropsychological function in children with blood lead levels <10 microg/dL High prevalence of elevated blood lead levels in both rural and urban Iowa newborns: Spatial patterns and area-level covariates.Blood lead concentrations and children's behavioral and emotional problems: a cohort study Chronic low-level Pb exposure during development decreases the expression of the voltage-dependent anion channel in auditory neurons of the brainstem.Lead (Pb) in biota and perceptions of Pb exposure at a recently designated Superfund beach site in New Jersey.Community-based participatory research (CBPR) approach to study children's health in China: experiences and reflections Same sex, no sex, and unaware sex in neurotoxicology Cytotoxic, genotoxic, and neurotoxic effects of Mg, Pb, and Fe on pheochromocytoma (PC-12) cells.Impact of low blood lead concentrations on IQ and school performance in Chinese children.Lead Exposure Induces Telomere Instability in Human Cells Higher infant blood lead levels with longer duration of breastfeeding Placental transfer and concentrations of cadmium, mercury, lead, and selenium in mothers, newborns, and young children Early chronic lead exposure reduces exploratory activity in young C57BL/6J mice Emerging aspects of assessing lead poisoning in childhood.δ-Aminolevulinic acid dehydratase single nucleotide polymorphism 2 and peptide transporter 2*2 haplotype may differentially mediate lead exposure in male children.A hypothesis about how early developmental methylmercury exposure disrupts behavior in adulthood.Olfactory recognition memory is disrupted in young mice with chronic low-level lead exposure.Blood lead in the 21st Century: The sub-microgram challenge Current needs and future directions of occupational safety and heath in a globalized world Lead Toxicity Risks in Gunshot Victims The relationship between early childhood blood lead levels and performance on end-of-grade tests.Inverse association of intellectual function with very low blood lead but not with manganese exposure in Italian adolescents.Blood lead concentrations < 10 microg/dL and child intelligence at 6 years of age.Low-level human equivalent gestational lead exposure produces sex-specific motor and coordination abnormalities and late-onset obesity in year-old mice

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

http://www.wikidata.org/entity/Q24647248

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2006 nî lūn-bûn

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2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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type

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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prefLabel

A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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J.NEURO.2006.06.008

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NeuroToxicology

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A rationale for lowering the blood lead action level from 10 to 2 microg/dL

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Bernard Weiss

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Steven G Gilbert

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P2860

A critical review of biomarkers used for monitoring human exposure to lead: advantages, limitations, and future needs

Low-level environmental lead exposure and children's intellectual function: an international pooled analysis.

Environmental Pollutants and Disease in American Children: Estimates of Morbidity, Mortality, and Costs for Lead Poisoning, Asthma, Cancer, and Developmental Disabilities

Maternal stress modulates the effects of developmental lead exposure

Ethics assessment as an adjunct to risk assessment in the evaluation of developmental neurotoxicants.

Intellectual impairment in children with blood lead concentrations below 10 microg per deciliter

Cognitive deficits associated with blood lead concentrations <10 microg/dL in US children and adolescents

Issues in developmental neurotoxicology: interpretation and implications of the data.

Blood lead levels in residents of homes with elevated lead in tap water--District of Columbia, 2004.

Measurement by ICP-MS of lead in plasma and whole blood of lead workers and controls.

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