Epstein-Barr virus-induced miR-155 attenuates NF-kappaB signaling and stabilizes latent virus persistence - Wikidata - wikimedia - TriplyDB

Epstein-Barr virus-induced miR-155 attenuates NF-kappaB signaling and stabilizes latent virus persistence

Epstein-Barr virus-induced miR-155 attenuates NF-kappaB signaling and stabilizes latent virus persistence

http://www.wikidata.org/entity/Q24648180

about

The viral and cellular microRNA targetome in lymphoblastoid cell lines MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine response In vivo microRNA-155 expression influences antigen-specific T cell-mediated immune responses generated by DNA vaccination Helicobacter pylori induces miR-155 in T cells in a cAMP-Foxp3-dependent manner The A20 deubiquitinase activity negatively regulates LMP1 activation of IRF7 Termination of NF-kappaB activity through a gammaherpesvirus protein that assembles an EC5S ubiquitin-ligase Virally induced cellular microRNA miR-155 plays a key role in B-cell immortalization by Epstein-Barr virus MicroRNA miR-155 inhibits bone morphogenetic protein (BMP) signaling and BMP-mediated Epstein-Barr virus reactivation Induction of microRNA-155 is TLR- and type IV secretion system-dependent in macrophages and inhibits DNA-damage induced apoptosis Oncogenic IRFs provide a survival advantage for Epstein-Barr virus- or human T-cell leukemia virus type 1-transformed cells through induction of BIC expression Reticuloendotheliosis virus strain T induces miR-155, which targets JARID2 and promotes cell survival Virus meets host microRNA: the destroyer, the booster, the hijacker Mammalian microRNA: an important modulator of host-pathogen interactions in human viral infections Human metapneumovirus infection induces significant changes in small noncoding RNA expression in airway epithelial cells.Chromatin organization of gammaherpesvirus latent genomes.MiR-155 induction in microglial cells suppresses Japanese encephalitis virus replication and negatively modulates innate immune responses.Critical role of the virus-encoded microRNA-155 ortholog in the induction of Marek's disease lymphomas.MicroRNAs of the immune system: roles in inflammation and cancer.Upregulation of MiR-155 in nasopharyngeal carcinoma is partly driven by LMP1 and LMP2A and downregulates a negative prognostic marker JMJD1A.Epstein-Barr virus BART9 miRNA modulates LMP1 levels and affects growth rate of nasal NK T cell lymphomas A systems biology approach identified different regulatory networks targeted by KSHV miR-K12-11 in B cells and endothelial cells Alteration of microRNA profiles in squamous cell carcinoma of the head and neck cell lines by human papillomavirus.The Epstein-Barr virus (EBV)-induced tumor suppressor microRNA MiR-34a is growth promoting in EBV-infected B cells.Kaposi's Sarcoma-Associated Herpesvirus microRNAs.Differential activation and functional specialization of miR-146 and miR-155 in innate immune sensing.Viral oncogenes, noncoding RNAs, and RNA splicing in human tumor viruses A microRNA encoded by Kaposi sarcoma-associated herpesvirus promotes B-cell expansion in vivo miRNAs in the pathogenesis of oncogenic human viruses.The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.Sulfated small molecules targeting eBV in Burkitt lymphoma: from in silico screening to the evidence of in vitro effect on viral episomal DNA.miR-155: on the crosstalk between inflammation and cancer.Wolbachia uses host microRNAs to manipulate host gene expression and facilitate colonization of the dengue vector Aedes aegypti.Epstein-Barr Virus_Encoded LMP1 upregulates microRNA-21 to promote the resistance of nasopharyngeal carcinoma cells to cisplatin-induced Apoptosis by suppressing PDCD4 and Fas-L.Ectopic expression of microRNA-155 enhances innate antiviral immunity against HBV infection in human hepatoma cells A Kaposi's sarcoma-associated herpesvirus-encoded ortholog of microRNA miR-155 induces human splenic B-cell expansion in NOD/LtSz-scid IL2Rγnull mice.Both MicroRNA-155 and Virus-Encoded MiR-155 Ortholog Regulate TLR3 Expression.MicroRNA-155 Reinforces HIV Latency.miR-155 regulates HGAL expression and increases lymphoma cell motility.Looking at Kaposi's Sarcoma-Associated Herpesvirus-Host Interactions from a microRNA Viewpoint.NF-κB/STAT5/miR-155 network targets PU.1 in FLT3-ITD-driven acute myeloid leukemia.

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Epstein-Barr virus-induced miR-155 attenuates NF-kappaB signaling and stabilizes latent virus persistence

http://www.wikidata.org/entity/Q24648180

description

2008 nî lūn-bûn

@nan

2008 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@ast

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@en

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@nl

type

label

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@ast

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@en

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@nl

prefLabel

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@ast

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@en

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@nl

P1433

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P1476

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P2093

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P2860

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P932

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P1433

Journal of Virology

P1476

Epstein-Barr virus-induced miR ...... lizes latent virus persistence

@en

P2093

Andreas Weidmer

http://www.w3.org/2001/XMLSchema#string

Chang-Gong Liu

http://www.w3.org/2001/XMLSchema#string

Fang Lu

http://www.w3.org/2001/XMLSchema#string

Paul M Lieberman

http://www.w3.org/2001/XMLSchema#string

P2860

bic, a novel gene activated by proviral insertions in avian leukosis virus-induced lymphomas, is likely to function through its noncoding RNA

MicroRNA-155 is an Epstein-Barr virus-induced gene that modulates Epstein-Barr virus-regulated gene expression pathways

A viral microRNA functions as an orthologue of cellular miR-155

Requirement of bic/microRNA-155 for normal immune function

B-cell receptor activation induces BIC/miR-155 expression through a conserved AP-1 element

Kaposi's sarcoma-associated herpesvirus encodes an ortholog of miR-155

MicroRNA-155 is induced during the macrophage inflammatory response

Oncomirs - microRNAs with a role in cancer

Silencing of microRNAs in vivo with 'antagomirs'

Triggering the interferon antiviral response through an IKK-related pathway

P304

10436-43

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P3181

1456376

http://www.w3.org/2001/XMLSchema#string

P356

10.1128/JVI.00752-08

http://www.w3.org/2001/XMLSchema#string

P407

P433

21

http://www.w3.org/2001/XMLSchema#string

P478

82

http://www.w3.org/2001/XMLSchema#string

P50

Stefano Volinia

Carlo Maria Croce

P577

2008-11-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

18753206

http://www.w3.org/2001/XMLSchema#string

P921

viral persistence

Epstein–Barr virus

P932

2573162

http://www.w3.org/2001/XMLSchema#string