Deadly liaisons: fatal attraction between CCN matricellular proteins and the tumor necrosis factor family of cytokines - Wikidata - wikimedia - TriplyDB

Deadly liaisons: fatal attraction between CCN matricellular proteins and the tumor necrosis factor family of cytokines

Deadly liaisons: fatal attraction between CCN matricellular proteins and the tumor necrosis factor family of cytokines

http://www.wikidata.org/entity/Q24648222

about

CCN: core regulatory proteins in the microenvironment that affect the metastasis of hepatocellular carcinoma?Taking aim at the extracellular matrix: CCN proteins as emerging therapeutic targets NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9 Apoptosis and angiogenesis: an evolving mechanism for fibrosis CCN1/CYR61: the very model of a modern matricellular protein.TNFα-induced apoptosis enabled by CCN1/CYR61: pathways of reactive oxygen species generation and cytochrome c release.Connective tissue growth factor regulates retinal neovascularization through p53 protein-dependent transactivation of the matrix metalloproteinase (MMP)-2 gene Antifibrotic effect of pirfenidone in a mouse model of human nonalcoholic steatohepatitis.The role of the CCN family of proteins in female reproduction.Effects of CCN3 on rat cartilage endplate chondrocytes cultured under serum deprivation in vitro The matricellular protein CCN1 suppresses lung cancer cell growth by inducing senescence via the p53/p21 pathway.CCN2/decorin interactions: a novel approach to combating fibrosis?The matricellular protein CCN1 regulates TNF-α induced vascular endothelial cell apoptosis.CCN3: the-pain-killer inside me.CCN2: a bona fide target for anti-fibrotic drug intervention Bone marrow and adipose mesenchymal stem cells attenuate cardiac fibrosis induced by methotrexate in rats.Effect of ALDH2 on High Glucose-Induced Cardiac Fibroblast Oxidative Stress, Apoptosis, and Fibrosis.The fibrosis of ketamine, a noncompetitive N-methyl-d-aspartic acid receptor antagonist dose-dependent change in a ketamine-induced cystitis rat model.Regulation of CCN1 (Cyr61) in a porcine model of intestinal ischemia/reperfusion.Extracellular matrix protein CCN1 regulates cardiomyocyte apoptosis in mice with stress-induced cardiac injury.

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Deadly liaisons: fatal attraction between CCN matricellular proteins and the tumor necrosis factor family of cytokines

http://www.wikidata.org/entity/Q24648222

description

2010 nî lūn-bûn

@nan

2010 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մարտին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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S12079-009-0080-4

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Journal of Cell Communication and Signaling

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Deadly liaisons: fatal attract ...... sis factor family of cytokines

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Chih-Chiun Chen

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P2860

Fas-mediated apoptosis is regulated by the extracellular matrix protein CCN1 (CYR61) in vitro and in vivo

Riboflavin kinase couples TNF receptor 1 to NADPH oxidase

Functions and mechanisms of action of CCN matricellular proteins

Matrix protein CCN1 is critical for prostate carcinoma cell proliferation and TRAIL-induced apoptosis

The CCN family of proteins: structure-function relationships

Diversity of function is inherent in matricellular proteins: an appraisal of thrombospondin 1

The matrix protein CCN1 (CYR61) induces apoptosis in fibroblasts

Cytotoxicity of TNFalpha is regulated by integrin-mediated matrix signaling

The TNF and TNF receptor superfamilies: integrating mammalian biology

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

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