Insulin resistance is a cellular antioxidant defense mechanism - Wikidata - wikimedia - TriplyDB

Insulin resistance is a cellular antioxidant defense mechanism

Insulin resistance is a cellular antioxidant defense mechanism

http://www.wikidata.org/entity/Q24650976

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Beyond Diabetes: Does Obesity-Induced Oxidative Stress Drive the Aging Process?The Elevated Susceptibility to Diabetes in India: An Evolutionary Perspective Roles of Reactive Oxygen Species on Insulin Resistance in Adipose Tissue Mitochondrial Regulation of the Muscle Microenvironment in Critical Limb Ischemia Redox implications in adipose tissue (dys)function--A new look at old acquaintances Insulin resistance protects the heart from fuel overload in dysregulated metabolic states Oxidative stress and diabetes: what can we learn about insulin resistance from antioxidant mutant mouse models?SIRT3 regulates progression and development of diseases of aging.Insulin resistance as a physiological defense against metabolic stress: implications for the management of subsets of type 2 diabetes Genetics of oxidative stress in obesity Cardiac dysfunction and oxidative stress in the metabolic syndrome: an update on antioxidant therapies Redox homeostasis in pancreatic β cells Targeting the redox balance in inflammatory skin conditions Curcumin prevents high fat diet induced insulin resistance and obesity via attenuating lipogenesis in liver and inflammatory pathway in adipocytes MsrA Overexpression Targeted to the Mitochondria, but Not Cytosol, Preserves Insulin Sensitivity in Diet-Induced Obese Mice Free fatty acids, lipopolysaccharide and IL-1α induce adipocyte manganese superoxide dismutase which is increased in visceral adipose tissues of obese rodents Chaperoning to the metabolic party: The emerging therapeutic role of heat-shock proteins in obesity and type 2 diabetes Inactivation of pyruvate dehydrogenase kinase 2 by mitochondrial reactive oxygen species Protective mechanisms of mitochondria and heart function in diabetes Site-specific covalent modifications of human insulin by catechol estrogens: Reactivity and induced structural and functional changes Signal transduction by reactive oxygen species Involvement of mTOR in Type 2 CRF Receptor Inhibition of Insulin Signaling in Muscle Cells.Stress-induced dissociations between intracellular calcium signaling and insulin secretion in pancreatic islets Activation of murine pre-proglucagon-producing neurons reduces food intake and body weight.Hepatitis C virus infection promotes hepatic gluconeogenesis through an NS5A-mediated, FoxO1-dependent pathway.Increased serum CXCL1 and CXCL5 are linked to obesity, hyperglycemia, and impaired islet function Identification of a novel mitochondrial uncoupler that does not depolarize the plasma membrane.Opening of the mitochondrial permeability transition pore links mitochondrial dysfunction to insulin resistance in skeletal muscle.Prolonged exposure to insulin induces mitochondrion-derived oxidative stress through increasing mitochondrial cholesterol content in hepatocytes.Nrf2 deficiency in myeloid cells is not sufficient to protect mice from high-fat diet-induced adipose tissue inflammation and insulin resistance.Insulin and insulin signaling play a critical role in fat induction of insulin resistance in mouse.Superoxide flashes: early mitochondrial signals for oxidative stress-induced apoptosis.Preserved endothelial function in human obesity in the absence of insulin resistance Biphasic response of mitochondrial biogenesis to oxidative stress in visceral fat of diet-induced obesity mice.Update on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?Electron transport chain-dependent and -independent mechanisms of mitochondrial H2O2 emission during long-chain fatty acid oxidation.Acute and chronic changes in rat soleus muscle after high-fat high-sucrose diet.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Mitochondrial dysfunction has divergent, cell type-dependent effects on insulin action Thresholds of oxidative stress in newly diagnosed diabetic patients on intensive glucose-control therapy.

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Insulin resistance is a cellular antioxidant defense mechanism

http://www.wikidata.org/entity/Q24650976

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2009 nî lūn-bûn

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2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Insulin resistance is a cellular antioxidant defense mechanism

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Insulin resistance is a cellular antioxidant defense mechanism

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Insulin resistance is a cellular antioxidant defense mechanism

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Adam B Salmon

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Arlan R Richardson

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Cordula Hohnen-Behrens

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Edward W Kraegen

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Ghassan J Maghzal

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Greg J Cooney

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Roland Stocker

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P2860

IRS1-independent defects define major nodes of insulin resistance

Biochemistry and molecular cell biology of diabetic complications

Life-long reduction in MnSOD activity results in increased DNA damage and higher incidence of cancer but does not accelerate aging

Inhibition of ceramide synthesis ameliorates glucocorticoid-, saturated-fat-, and obesity-induced insulin resistance

A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase

Effects of intensive glucose lowering in type 2 diabetes

Critical nodes in signalling pathways: insights into insulin action

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

The mitochondrial generation of hydrogen peroxide. General properties and effect of hyperbaric oxygen

Reactive oxygen species have a causal role in multiple forms of insulin resistance

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