A delayed and chronic treatment regimen with the 5-HT1A receptor agonist 8-OH-DPAT after cortical impact injury facilitates motor recovery and acquisition of spatial learning - Wikidata - wikimedia - TriplyDB

A delayed and chronic treatment regimen with the 5-HT1A receptor agonist 8-OH-DPAT after cortical impact injury facilitates motor recovery and acquisition of spatial learning

A delayed and chronic treatment regimen with the 5-HT1A receptor agonist 8-OH-DPAT after cortical impact injury facilitates motor recovery and acquisition of spatial learning

http://www.wikidata.org/entity/Q24651403

about

Traumatic brain injury-induced cognitive and histological deficits are attenuated by delayed and chronic treatment with the 5-HT1A-receptor agonist buspirone Hippocampal 5-HT1A Receptor and Spatial Learning and Memory.Found in translation: Understanding the biology and behavior of experimental traumatic brain injury Combination therapies for neurobehavioral and cognitive recovery after experimental traumatic brain injury: Is more better?Environmental enrichment promotes robust functional and histological benefits in female rats after controlled cortical impact injury A combined therapeutic regimen of buspirone and environmental enrichment is more efficacious than either alone in enhancing spatial learning in brain-injured pediatric rats Environmental enrichment as a viable neurorehabilitation strategy for experimental traumatic brain injury.Old dog, new tricks: the attentional set-shifting test as a novel cognitive behavioral task after controlled cortical impact injury.Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis Empirical comparison of typical and atypical environmental enrichment paradigms on functional and histological outcome after experimental traumatic brain injury.Evaluation of a combined therapeutic regimen of 8-OH-DPAT and environmental enrichment after experimental traumatic brain injury.Abbreviated environmental enrichment enhances neurobehavioral recovery comparably to continuous exposure after traumatic brain injury.Biologic and plastic effects of experimental traumatic brain injury treatment paradigms and their relevance to clinical rehabilitation.S-nitrosoglutathione reduces oxidative injury and promotes mechanisms of neurorepair following traumatic brain injury in rats.Divergent long-term consequences of chronic treatment with haloperidol, risperidone, and bromocriptine on traumatic brain injury-induced cognitive deficits Elucidating the role of 5-HT(1A) and 5-HT(7) receptors on 8-OH-DPAT-induced behavioral recovery after experimental traumatic brain injury.Evaluation of a combined treatment paradigm consisting of environmental enrichment and the 5-HT1A receptor agonist buspirone after experimental traumatic brain injury.A relatively brief exposure to environmental enrichment after experimental traumatic brain injury confers long-term cognitive benefits Donepezil is ineffective in promoting motor and cognitive benefits after controlled cortical impact injury in male rats Administration of S-nitrosoglutathione after traumatic brain injury protects the neurovascular unit and reduces secondary injury in a rat model of controlled cortical impact.Fluoxetine increases hippocampal neurogenesis and induces epigenetic factors but does not improve functional recovery after traumatic brain injury.5-hydroxytryptamine1A (5-HT1A) receptor agonists: A decade of empirical evidence supports their use as an efficacious therapeutic strategy for brain trauma.Comparable impediment of cognitive function in female and male rats subsequent to daily administration of haloperidol after traumatic brain injury.GSNO promotes functional recovery in experimental TBI by stabilizing HIF-1α.Abbreviated environmental enrichment confers neurobehavioral, cognitive, and histological benefits in brain-injured female rats.The Therapeutic Efficacy of Environmental Enrichment and Methylphenidate Alone and in Combination after Controlled Cortical Impact Injury.Spontaneous recovery of traumatic brain injury-induced functional deficits is not hindered by daily administration of lorazepam.Refining environmental enrichment to advance rehabilitation based research after experimental traumatic brain injury.Relative to Typical Antipsychotic Drugs, Aripiprazole Is a Safer Alternative for Alleviating Behavioral Disturbances After Experimental Brain Trauma.Validated RP-HPLC Method for the Determination of Buspirone in Pharmaceutical Formulations Executive (dys)function after traumatic brain injury: special considerations for behavioral pharmacology

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A delayed and chronic treatment regimen with the 5-HT1A receptor agonist 8-OH-DPAT after cortical impact injury facilitates motor recovery and acquisition of spatial learning

http://www.wikidata.org/entity/Q24651403

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2008 nî lūn-bûn

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2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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A delayed and chronic treatmen ...... cquisition of spatial learning

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A delayed and chronic treatmen ...... cquisition of spatial learning

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A delayed and chronic treatmen ...... cquisition of spatial learning

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J.BBR.2008.06.025

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Behavioural Brain Research

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A delayed and chronic treatmen ...... cquisition of spatial learning

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Ann N Hoffman

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Anthony E Kline

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Ross D Zafonte

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P2860

A review of central 5-HT receptors and their function

Postsynaptic 5-hydroxytryptamine(1A) receptor activation increases in vivo dopamine release in rat prefrontal cortex

5-HT system and cognition.

The neuroprotective effect of a new serotonin receptor agonist, BAY X3702, upon focal ischemic brain damage caused by acute subdural hematoma in the rat.

The selective 5-HT(1A) receptor agonist repinotan HCl attenuates histopathology and spatial learning deficits following traumatic brain injury in rats.

Repinotan (BAY x 3702): a 5HT1A agonist in traumatically brain injured patients.

Characterization of the aminomethylchroman derivative BAY x 3702 as a highly potent 5-hydroxytryptamine1A receptor agonist.

Differential effects of single versus multiple administrations of haloperidol and risperidone on functional outcome after experimental brain trauma

Acute treatment with the 5-HT(1A) receptor agonist 8-OH-DPAT and chronic environmental enrichment confer neurobehavioral benefit after experimental brain trauma.

The neurobehavioral benefit conferred by a single systemic administration of 8-OH-DPAT after brain trauma is confined to a narrow therapeutic window.

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79-85

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10.1016/J.BBR.2008.06.025

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