Kuru prions and sporadic Creutzfeldt-Jakob disease prions have equivalent transmission properties in transgenic and wild-type mice - Wikidata - wikimedia - TriplyDB

Kuru prions and sporadic Creutzfeldt-Jakob disease prions have equivalent transmission properties in transgenic and wild-type mice

Kuru prions and sporadic Creutzfeldt-Jakob disease prions have equivalent transmission properties in transgenic and wild-type mice

http://www.wikidata.org/entity/Q24651920

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Nonruminant Nutrition Symposium: Involvement of gut neural and endocrine systems in pathological disorders of the digestive tract An overview of human prion diseases The kuru infectious agent is a unique geographic isolate distinct from Creutzfeldt-Jakob disease and scrapie agents.HECTD2 is associated with susceptibility to mouse and human prion disease Kuru: genes, cannibals and neuropathology.Agent strain variation in human prion disease: insights from a molecular and pathological review of the National Institutes of Health series of experimentally transmitted disease.Kuru: a journey back in time from papua new Guinea to the neanderthals' extinction Molecular pathology of human prion disease.Review: contribution of transgenic models to understanding human prion disease.A naturally occurring variant of the human prion protein completely prevents prion disease.Transmission characteristics of variably protease-sensitive prionopathy.Chronic wasting disease prions are not transmissible to transgenic mice overexpressing human prion protein.Inherited prion disease A117V is not simply a proteinopathy but produces prions transmissible to transgenic mice expressing homologous prion protein.Replication and spread of CJD, kuru and scrapie agents in vivo and in cell culture Structural determinants of phenotypic diversity and replication rate of human prions.Increased infectivity of anchorless mouse scrapie prions in transgenic mice overexpressing human prion protein Transmission Properties of Human PrP 102L Prions Challenge the Relevance of Mouse Models of GSS.Iatrogenic CJD due to pituitary-derived growth hormone with genetically determined incubation times of up to 40 years.Persistent propagation of variant Creutzfeldt-Jakob disease agent in murine spleen stromal cell culture with features of mesenchymal stem cells.Review. Lessons of kuru research: background to recent studies with some personal reflections.Review. The origin of the prion agent of kuru: molecular and biological strain typing.Central and peripheral pathology of kuru: pathological analysis of a recent case and comparison with other forms of human prion disease.Creutzfeldt-Jakob disease: case discussion and imaging review.Atypical scrapie prions from sheep and lack of disease in transgenic mice overexpressing human prion protein.A novel prion disease associated with diarrhea and autonomic neuropathy.Prion protein misfolding, strains, and neurotoxicity: an update from studies on Mammalian prions.Prion disease: experimental models and reality.Cultural factors that affected the spatial and temporal epidemiology of kuru.The contribution of different prion protein types and host polymorphisms to clinicopathological variations in Creutzfeldt-Jakob disease.Review: Creutzfeldt-Jakob disease: prion protein type, disease phenotype and agent strain.Human prion diseases: molecular, cellular and population biology.Prion disease: a tale of folds and strains.The influence of PRNP polymorphisms on human prion disease susceptibility: an update.Neuropathological and biochemical criteria to identify acquired Creutzfeldt-Jakob disease among presumed sporadic cases.Sporadic and Infectious Human Prion Diseases.Methods for Molecular Diagnosis of Human Prion Disease.Familial human prion diseases associated with prion protein mutations Y226X and G131V are transmissible to transgenic mice expressing human prion protein.Experimental sheep BSE prions generate the vCJD phenotype when serially passaged in transgenic mice expressing human prion protein.

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P2860

Kuru prions and sporadic Creutzfeldt-Jakob disease prions have equivalent transmission properties in transgenic and wild-type mice

http://www.wikidata.org/entity/Q24651920

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2008 nî lūn-bûn

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Kuru prions and sporadic Creut ...... transgenic and wild-type mice

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Kuru prions and sporadic Creut ...... transgenic and wild-type mice

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Kuru prions and sporadic Creut ...... transgenic and wild-type mice

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Kuru prions and sporadic Creut ...... transgenic and wild-type mice

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Proceedings of the National Academy of Sciences of the United States of America

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Kuru prions and sporadic Creut ...... transgenic and wild-type mice

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Jacqueline M Linehan

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John Collinge

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Jonathan D F Wadsworth

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Katie Fox

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Sabrina Cronier

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P2860

Prion diseases of humans and animals: their causes and molecular basis

The state of the prion

Eight prion strains have PrP(Sc) molecules with different conformations

Distinct PrP properties suggest the molecular basis of strain variation in transmissible mink encephalopathy.

Neuropathology and molecular biology of variant Creutzfeldt-Jakob disease.

Increased susceptibility to Kuru of carriers of the PRNP 129 methionine/methionine genotype.

Variant Creutzfeldt-Jakob disease.

Iatrogenic Creutzfeldt-Jakob disease at the millennium.

The epidemiology of kuru in the period 1987 to 1995.

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Melanie Desbruslais

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5534969

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Prion protein family

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