Traumatic brain injury: can the consequences be stopped?
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Citicoline (CDP-choline) for traumatic brain injuryExcitatory amino acid inhibitors for traumatic brain injuryThe cysteine protease cathepsin B is a key drug target and cysteine protease inhibitors are potential therapeutics for traumatic brain injuryTraumatic Brain Injury and Neuronal Functionality Changes in Sensory CortexHippocampal Neurophysiologic Changes after Mild Traumatic Brain Injury and Potential Neuromodulation Treatment ApproachesCathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug CandidateEfficacy of progesterone for moderate to severe traumatic brain injury: a meta-analysis of randomized clinical trialsEnvironmental enrichment and the sensory brain: the role of enrichment in remediating brain injury.Cells, biomarkers, and post-traumatic stress disorder: evidence for peripheral involvement in a central diseaseSmartphone-enabled optofluidic exosome diagnostic for concussion recoveryN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationNeurochemical profile of dementia pugilisticaProphylactic and therapeutic functions of drug combinations against noise-induced hearing lossCognitive Impairments Induced by Concussive Mild Traumatic Brain Injury in Mouse Are Ameliorated by Treatment with Phenserine via Multiple Non-Cholinergic and Cholinergic MechanismsThe Effects of Shilajit on Brain Edema, Intracranial Pressure and Neurologic Outcomes following the Traumatic Brain Injury in RatLong-Term Kinetics of Immunologic Components and Neurological Deficits in Rats Following Repetitive Mild Traumatic Brain Injury.Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesisMetabolic acetate therapy for the treatment of traumatic brain injury.Traumatic brain injury: an overview of pathobiology with emphasis on military populations.Diminished brain resilience syndrome: A modern day neurological pathology of increased susceptibility to mild brain trauma, concussion, and downstream neurodegeneration.Incretin mimetics as pharmacologic tools to elucidate and as a new drug strategy to treat traumatic brain injuryReduction of cerebral edema after traumatic brain injury using an osmotic transport device.Towards systemic sustainable performance of TBI care systems: emergency leadership frontiers.Early Health Risk Factors for Violence: Conceptualization, Review of the Evidence, and Implications.The occurrence of diffuse axonal injury in the brain: associated with the accumulation and clearance of myelin debrisExcitatory synaptic transmission and network activity are depressed following mechanical injury in cortical neuronsNormobaric hyperoxia in traumatic brain injury: does brain metabolic state influence the response to hyperoxic challenge?Comparative study of outcome measures and analysis methods for traumatic brain injury trials.Calcium signaling is implicated in the diffuse axonal injury of brain stem.A Pilot Study of Cerebral and Hemodynamic Changes During Sedation with Low Dose of Thiopental Sodium or Propofol in Patients with Acute Brain Injury.Targeting mTOR as a novel therapeutic strategy for traumatic CNS injuries.An Epidemiologic Study of Traumatic Brain Injuries in Emergency Department.Fractal Analysis of Brain Blood Oxygenation Level Dependent (BOLD) Signals from Children with Mild Traumatic Brain Injury (mTBI)Subventricular zone-derived neural stem cell grafts protect against hippocampal degeneration and restore cognitive function in the mouse following intrahippocampal kainic acid administrationBattlefield brain: unexplained symptoms and blast-related mild traumatic brain injury.Stretch injury selectively enhances extrasynaptic, GluN2B-containing NMDA receptor function in cortical neuronsThe potential utility of blood-derived biochemical markers as indicators of early clinical trends following severe traumatic brain injury.Differential influence of arterial blood glucose on cerebral metabolism following severe traumatic brain injuryIntracranial pressure monitoring in severe traumatic brain injury: results from the American College of Surgeons Trauma Quality Improvement Program.Prehospital hypertonic saline resuscitation attenuates the activation and promotes apoptosis of neutrophils in patients with severe traumatic brain injury.
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P2860
Traumatic brain injury: can the consequences be stopped?
description
2008 nî lūn-bûn
@nan
2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Traumatic brain injury: can the consequences be stopped?
@ast
Traumatic brain injury: can the consequences be stopped?
@en
Traumatic brain injury: can the consequences be stopped?
@nl
type
label
Traumatic brain injury: can the consequences be stopped?
@ast
Traumatic brain injury: can the consequences be stopped?
@en
Traumatic brain injury: can the consequences be stopped?
@nl
prefLabel
Traumatic brain injury: can the consequences be stopped?
@ast
Traumatic brain injury: can the consequences be stopped?
@en
Traumatic brain injury: can the consequences be stopped?
@nl
P2093
P2860
P3181
P356
P1476
Traumatic brain injury: can the consequences be stopped?
@en
P2093
Andrew J Baker
Eugene Park
Joshua D Bell
P2860
P304
P3181
P356
10.1503/CMAJ.080282
P407
P577
2008-04-22T00:00:00Z