SSRIs act as selective brain steroidogenic stimulants (SBSSs) at low doses that are inactive on 5-HT reuptake
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Neurosteroids and GABA(A) Receptor Interactions: A Focus on StressIn a mouse model relevant for post-traumatic stress disorder, selective brain steroidogenic stimulants (SBSS) improve behavioral deficits by normalizing allopregnanolone biosynthesis.The role of ovarian hormone-derived neurosteroids on the regulation of GABAA receptors in affective disordersAnxiety disorders and GABA neurotransmission: a disturbance of modulationGABA receptor subunit distribution and FMRP-mGluR5 signaling abnormalities in the cerebellum of subjects with schizophrenia, mood disorders, and autism.Midazolam ameliorates the behavior deficits of a rat posttraumatic stress disorder model through dual 18 kDa translocator protein and central benzodiazepine receptor and neurosteroidogenesisSex differences in anxiety and emotional behavior.Expression of GABAA α2-, β1- and ε-receptors are altered significantly in the lateral cerebellum of subjects with schizophrenia, major depression and bipolar disorderThe GABAergic deficit hypothesis of major depressive disorder.Neurosteroids reduce social isolation-induced behavioral deficits: a proposed link with neurosteroid-mediated upregulation of BDNF expression.Enhanced fear responses in mice treated with anabolic androgenic steroids.Behavioral and neurogenomic transcriptome changes in wild-derived zebrafish with fluoxetine treatmentChronic SSRI stimulation of astrocytic 5-HT2B receptors change multiple gene expressions/editings and metabolism of glutamate, glucose and glycogen: a potential paradigm shift.Neurosteroids and GABAergic signaling in health and diseaseHormone-specific psychiatric disorders: do they exist?Allopregnanolone elevations following pregnenolone administration are associated with enhanced activation of emotion regulation neurocircuits.Neurosteroid biosynthesis downregulation and changes in GABAA receptor subunit composition: A biomarker axis in stress-induced cognitive and emotional impairment.S-norfluoxetine microinfused into the basolateral amygdala increases allopregnanolone levels and reduces aggression in socially isolated mice.Up-regulation of neurosteroid biosynthesis as a pharmacological strategy to improve behavioural deficits in a putative mouse model of post-traumatic stress disorderProgesterone attenuates depressive behavior of younger and older adult C57/BL6, wildtype, and progesterone receptor knockout mice.Role of 5-HT(1A) receptors in fluoxetine-induced lordosis inhibition.GABAergic control of depression-related brain statesmRNA and protein expression for novel GABAA receptors θ and ρ2 are altered in schizophrenia and mood disorders; relevance to FMRP-mGluR5 signaling pathway.Sex differences in anxiety and depression clinical perspectives.Fluoxetine Ameliorates Atopic Dermatitis-Like Skin Lesions in BALB/c Mice through Reducing Psychological Stress and Inflammatory ResponseClinical efficacy and safety of fluoxetine in generalized anxiety disorder in Chinese patients.Estrous cycle and stress: influence of progesterone on the female brainTargeting neurosteroidogenesis as therapy for PTSD.The antidepressant-like activity of AC-5216, a ligand for 18KDa translocator protein (TSPO), in an animal model of diabetes mellitus.Translocator protein (18 kDa) as a target for novel anxiolytics with a favourable side-effect profile.A neurochemical basis for an epigenetic vision of psychiatric disorders (1994-2009).Allopregnanolone modulation of HPA axis function in the adult rat.Development of novel therapy of schizophrenia in children and adolescents.Short term, low dose fluoxetine blocks estrous cycle-linked changes in responsiveness to diazepam in female rats.The anxiolytic-like effects of puerarin are associated with the changes of monoaminergic neurotransmitters and biosynthesis of allopregnanolone in the brain.Correlation between allopregnanolone levels and depressive symptoms during late menopausal transition and early postmenopause.Social Isolation in Early versus Late Adolescent Mice Is Associated with Persistent Behavioral Deficits That Can Be Improved by Neurosteroid-Based Treatment.5α-reductase type I expression is downregulated in the prefrontal cortex/Brodmann's area 9 (BA9) of depressed patients.GABAA and GABAB receptor dysregulation in superior frontal cortex of subjects with schizophrenia and bipolar disorder.SSRIs and the female brain--potential for utilizing steroid-stimulating properties to treat menstrual cycle-linked dysphorias.
P2860
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P2860
SSRIs act as selective brain steroidogenic stimulants (SBSSs) at low doses that are inactive on 5-HT reuptake
description
2009 nî lūn-bûn
@nan
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@ast
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@en
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@nl
type
label
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@ast
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@en
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@nl
prefLabel
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@ast
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@en
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@nl
P2093
P2860
P3181
P1476
SSRIs act as selective brain s ...... are inactive on 5-HT reuptake
@en
P2093
Alessandro Guidotti
Erminio Costa
Graziano Pinna
P2860
P3181
P356
10.1016/J.COPH.2008.12.006
P407
P577
2009-02-01T00:00:00Z