Hemolytic uremic syndrome-associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers - Wikidata - wikimedia - TriplyDB

Hemolytic uremic syndrome-associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

Hemolytic uremic syndrome-associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

http://www.wikidata.org/entity/Q24669689

about

Hemolytic uremic syndrome: new developments in pathogenesis and treatment Pathophysiology of thrombotic thrombocytopenic purpura The molecular biology of thrombotic microangiopathy Pathogenesis of thrombotic microangiopathies Proteolytic processing of von Willebrand factor by adamts13 and leukocyte proteases Nxf1 natural variant E610G is a semi-dominant suppressor of IAP-induced RNA processing defects Down-regulation of platelet surface CD47 expression in Escherichia coli O157:H7 infection-induced thrombocytopenia N-acetylcysteine reduces the size and activity of von Willebrand factor in human plasma and mice Flow-driven assembly of VWF fibres and webs in in vitro microvessels Degradation of circulating von Willebrand factor and its regulator ADAMTS13 implicates secreted Bacillus anthracis metalloproteases in anthrax consumptive coagulopathy.Thrombotic microangiopathies: multimers, metalloprotease, and beyond.Shiga toxin B subunits induce VWF secretion by human endothelial cells and thrombotic microangiopathy in ADAMTS13-deficient mice.Shiga toxin (Stx)1B and Stx2B induce von Willebrand factor secretion from human umbilical vein endothelial cells through different signaling pathways Shiga toxin pathogenesis: kidney complications and renal failure.In vitro modeling of the microvascular occlusion and thrombosis that occur in hematologic diseases using microfluidic technology The CXCR4/CXCR7/SDF-1 pathway contributes to the pathogenesis of Shiga toxin-associated hemolytic uremic syndrome in humans and mice.Endothelial cells and thrombotic microangiopathy.Thrombotic microangiopathy and associated renal disorders.Shigatoxin-associated hemolytic uremic syndrome: current molecular mechanisms and future therapies.The challenge of managing hemophilia A and STEC-induced hemolytic uremic syndrome.Shiga toxins and the pathophysiology of hemolytic uremic syndrome in humans and animals.Complement activation associated with ADAMTS13 deficiency in human and murine thrombotic microangiopathy New insights into Shiga toxin-mediated endothelial dysfunction in hemolytic uremic syndrome Interaction of Shiga toxin with the A-domains and multimers of von Willebrand Factor.Circulating endothelial cells and progenitors as prognostic factors during autoimmune thrombotic thrombocytopenic purpura: results of a prospective multicenter French study.ADAMTS-13 in the Diagnosis and Management of Thrombotic Microangiopathies.Thrombocytopenia-Associated Multiple Organ Failure and Acute Kidney Injury.The prognostic value of ADAMTS13 (a disintegrin and metalloprotease with thrombospondin type 1 repeats, member 13) deficiency in septic shock patients involves interleukin-6 and is not dependent on disseminated intravascular coagulation Protein phosphatase 2B inhibition promotes the secretion of von Willebrand factor from endothelial cells.Current status in diagnosis and treatment of hereditary thrombotic thrombocytopenic purpura.Assembly and activation of alternative complement components on endothelial cell-anchored ultra-large von Willebrand factor links complement and hemostasis-thrombosis Porcine and canine von Willebrand factor and von Willebrand disease: hemostasis, thrombosis, and atherosclerosis studies.Factor VIII Is Synthesized in Human Endothelial Cells, Packaged in Weibel-Palade Bodies and Secreted Bound to ULVWF Strings.THE ENDOTHELIUM IN SEPSIS.The role of plasmapheresis in critical illness.The B subunits of Shiga-like toxins induce regulated VWF secretion in a phospholipase D1-dependent manner.Tissue factor: A potent stimulator of Von Willebrand factor synthesis by human umbilical vein endothelial cells.Endothelial cell ADAMTS-13 and VWF: production, release, and VWF string cleavage.Biomarkers of endothelial activation/dysfunction in infectious diseases.Facing glycosphingolipid-Shiga toxin interaction: dire straits for endothelial cells of the human vasculature.

P2860

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P2860

Hemolytic uremic syndrome-associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

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Aubrey Bernardo

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Jing-Fei Dong

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Joel L Moake

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Leticia H Nolasco

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Nancy A Turner

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Thomas G Cleary

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Zhenyin Tao

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P2860

Purification of human von Willebrand factor-cleaving protease and its identification as a new member of the metalloproteinase family

ADAMTS-13 metalloprotease interacts with the endothelial cell-derived ultra-large von Willebrand factor

Antibodies to von Willebrand factor-cleaving protease in acute thrombotic thrombocytopenic purpura

A mutant cholera toxin B subunit that binds GM1- ganglioside but lacks immunomodulatory or toxic activity

Cholera toxin: a paradigm for multi-functional engagement of cellular mechanisms (Review)

Shiga toxin binding to globotriaosyl ceramide induces intracellular signals that mediate cytoskeleton remodeling in human renal carcinoma-derived cells.

Coagulation changes associated with the hemolytic uremic syndrome.

Unrecognized pattern of von Willebrand factor abnormalities in hemolytic uremic syndrome and thrombotic thrombocytopenic purpura.

Shiga toxins stimulate secretion of interleukin-8 from intestinal epithelial cells.

Assay of von Willebrand factor (vWF)-cleaving protease based on decreased collagen binding affinity of degraded vWF: a tool for the diagnosis of thrombotic thrombocytopenic purpura (TTP)

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