Increased activity of coagulation factor XII (Hageman factor) causes hereditary angioedema type III - Wikidata - wikimedia - TriplyDB

Increased activity of coagulation factor XII (Hageman factor) causes hereditary angioedema type III

Increased activity of coagulation factor XII (Hageman factor) causes hereditary angioedema type III

http://www.wikidata.org/entity/Q24671677

about

Diagnosis and treatment of hereditary angioedema with normal C1 inhibitor Examination of genetic variants involved in generation and biodisposition of kinins in patients with angioedema Misfolded proteins activate factor XII in humans, leading to kallikrein formation without initiating coagulation Factor XII: what does it contribute to our understanding of the physiology and pathophysiology of hemostasis & thrombosis The factor XIIa blocking antibody 3F7: a safe anticoagulant with anti-inflammatory activities Classification, diagnosis, and approach to treatment for angioedema: consensus report from the Hereditary Angioedema International Working Group Bradykinin: Inflammatory Product of the Coagulation System Angioedema: Classification, management and emerging therapies for the perioperative physician Hereditary angioedema with normal C1 inhibitor in a French cohort: Clinical characteristics and response to treatment with icatibant A predictive clinical-genetic model of tissue plasminogen activator response in acute ischemic stroke.The establishment and utility of Sweha-Reg: a Swedish population-based registry to understand hereditary angioedema Transforming growth factor-β1 induces expression of human coagulation factor XII via Smad3 and JNK signaling pathways in human lung fibroblasts.Obstetrical Complications and Outcome in Two Families with Hereditary Angioedema due to Mutation in the F12 Gene.Activated factor V is a cofactor for the activation of factor XI by thrombin in plasma.Hereditary angioedema in women Factor XII mutations, estrogen-dependent inherited angioedema, and related conditions.Diagnosis and management of angioedema with abdominal involvement: a gastroenterology perspective.In vivo roles of factor XII.Role of Factor XII in hemostasis and thrombosis: clinical implications.Enzymatic assays for the diagnosis of bradykinin-dependent angioedema.Complement system in dermatological diseases - fire under the skin Thrombosis Prevention without Anticoagulation.Brazilian guidelines for the diagnosis and treatment of hereditary angioedema.Defective glycosylation of coagulation factor XII underlies hereditary angioedema type III Kininogen Cleavage Assay: Diagnostic Assistance for Kinin-Mediated Angioedema Conditions.Regulation of Complement and Contact System Activation via C1 Inhibitor Potentiation and Factor XIIa Activity Modulation by Sulfated Glycans - Structure-Activity Relationships Disease-modifying factors in hereditary angioedema: an RNA expression-based screening.Oestrogen-dependent hereditary angio-oedema with normal C1 inhibitor: description of six new cases and review of pathogenic mechanisms and treatment.Deficiency of plasminogen activator inhibitor 2 in plasma of patients with hereditary angioedema with normal C1 inhibitor levels.Hereditary angioedema type III (estrogen-dependent) report of three cases and literature review.Clinical Immunology Review Series: An approach to the patient with angio-oedema.Physiological responses to protein aggregates: fibrinolysis, coagulation and inflammation (new roles for old factors).Managing the female patient with hereditary angioedema.The procoagulant and proinflammatory plasma contact system.Target levels of functional C1-inhibitor in hereditary angioedema.Icatibant , the bradykinin B2 receptor antagonist with target to the interconnected kinin systems.Therapeutic options for patients with angioedema due to C1-inhibitor deficiencies: from pathophysiology to the clinic.Current pharmacotherapy of bradykinin-mediated angioedema.Chronic urticaria and coagulation: pathophysiological and clinical aspects.Tracking down contact activation - from coagulation in vitro to inflammation in vivo.

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P2860

Increased activity of coagulation factor XII (Hageman factor) causes hereditary angioedema type III

http://www.wikidata.org/entity/Q24671677

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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name

Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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type

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Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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prefLabel

Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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Increased activity of coagulat ...... hereditary angioedema type III

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American Journal of Human Genetics

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Increased activity of coagulat ...... hereditary angioedema type III

@en

P2093

Anna Karpushova

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Felicitas Müller

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Karen Van Driessche

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Konrad Bork

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Ludovic Martin

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Roberto Colombo

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Thomas Renné

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Wim Stevens

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P2860

Mutations of Bacteria from Virus Sensitivity to Virus Resistance

Defective thrombus formation in mice lacking coagulation factor XII

Linkage disequilibrium analysis in young populations: pseudo-vitamin D-deficiency rickets and the founder effect in French Canadians

Clinical, biochemical, and genetic characterization of a novel estrogen-dependent inherited form of angioedema

Targeting coagulation factor XII provides protection from pathological thrombosis in cerebral ischemia without interfering with hemostasis

A high-resolution recombination map of the human genome

Hereditary angioedema with normal C1-inhibitor activity in women.

High-resolution multipoint linkage-disequilibrium mapping in the context of a human genome sequence.

A review of the reported defects in the human C1 esterase inhibitor gene producing hereditary angioedema including four new mutations.

Measuring the strength of associations between HLA antigens and diseases.

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1098-104

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scholarly article

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2793986

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10.1086/509899

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6

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79

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Markus M Nöthen

Hans Christian Hennies

Christian Drouet

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2006-12-01T00:00:00Z

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6614929

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17186468

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1698720

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