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FXYD5: Na(+)/K(+)-ATPase Regulator in Health and DiseaseBeneficial Renal and Pancreatic Phenotypes in a Mouse Deficient in FXYD2 Regulatory Subunit of Na,K-ATPaseStructure of the Na,K-ATPase regulatory protein FXYD2b in micelles: Implications for membrane–water interfacial argininesDiscovering collectively informative descriptors from high-throughput experimentsDysadherin specific drug conjugates for the treatment of thyroid cancers with aggressive phenotypes.The Structure and Function of the Na,K-ATPase Isoforms in Health and Disease.FXYD5 (dysadherin) regulates the paracellular permeability in cultured kidney collecting duct cells.Phospholemman: a novel cardiac stress protein.Alterations in dysadherin expression and F-actin reorganization: a possible mechanism of hypericin-mediated photodynamic therapy in colon adenocarcinoma cells.Transcriptome sequencing reveals potential mechanism of cryptic 3' splice site selection in SF3B1-mutated cancersThe Na-K-ATPase α₁β₁ heterodimer as a cell adhesion molecule in epithelia.Intracellular trafficking of FXYD1 (phospholemman) and FXYD7 proteins in Xenopus oocytes and mammalian cells.Extracellular Antibody Drug Conjugates Exploiting the Proximity of Two ProteinsAberrantly regulated dysadherin and B-cell lymphoma 2/B-cell lymphoma 2-associated X enhances tumorigenesis and DNA targeting drug resistance of liver cancer stem cells.Constitutive expression of Wnt/β‑catenin target genes promotes proliferation and invasion of liver cancer stem cells.FXYD5 Protein Has a Pro-inflammatory Role in Epithelial Cells.The O-glycosylated ectodomain of FXYD5 impairs adhesion by disrupting cell-cell trans-dimerization of Na,K-ATPase β1 subunitsRole of the recently identified dysadherin in E-cadherin adhesion molecule downregulation in head and neck cancer.Osteoblast-secreted factors enhance the expression of dysadherin and CCL2-dependent migration of renal carcinoma cells.FXYD5 (dysadherin) may mediate metastatic progression through regulation of the β-Na+-K+-ATPase subunit in the 4T1 mouse breast cancer model.Functional genomics identifies novel genes essential for clear cell renal cell carcinoma tumor cell proliferation and migration.Modulation of cell polarization by the Na+-K+-ATPase-associated protein FXYD5 (dysadherin).Dysadherin expression promotes the motility and survival of human breast cancer cells by AKT activation.eIF3m expression influences the regulation of tumorigenesis-related genes in human colon cancer.Membrane orientation of the Na,K-ATPase regulatory membrane protein CHIF determined by solid-state NMR.
P2860
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P2860
description
2007 nî lūn-bûn
@nan
2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
name
Dysadherin: a new player in cancer progression
@ast
Dysadherin: a new player in cancer progression
@en
Dysadherin: a new player in cancer progression
@nl
type
label
Dysadherin: a new player in cancer progression
@ast
Dysadherin: a new player in cancer progression
@en
Dysadherin: a new player in cancer progression
@nl
prefLabel
Dysadherin: a new player in cancer progression
@ast
Dysadherin: a new player in cancer progression
@en
Dysadherin: a new player in cancer progression
@nl
P2093
P2860
P3181
P1433
P1476
Dysadherin: a new player in cancer progression
@en
P2093
Jeong-Seok Nam
Lalage M Wakefield
Setsuo Hirohashi
P2860
P3181
P356
10.1016/J.CANLET.2007.02.018
P407
P577
2007-10-08T00:00:00Z