Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis - Wikidata - wikimedia - TriplyDB

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

http://www.wikidata.org/entity/Q24675945

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Several distinct polycomb complexes regulate and co-localize on the INK4a tumor suppressor locus Molecular genetic analysis of Suppressor 2 of zeste identifies key functional domains BMI-1 promotes ewing sarcoma tumorigenicity independent of CDKN2A repression BMI1: A Biomarker of Hematologic Malignancies Polycomb Group (PcG) Proteins and Human Cancers: Multifaceted Functions and Therapeutic Implications Zfp423 Regulates Sonic Hedgehog Signaling via Primary Cilium Function Multiple recurrent genetic events converge on control of histone lysine methylation in medulloblastoma GFAP-Cre-mediated transgenic activation of Bmi1 results in pituitary tumors Polycomb group gene BMI1 controls invasion of medulloblastoma cells and inhibits BMP-regulated cell adhesion.Su(z)2 antagonizes auto-repression of Myc in Drosophila, increasing Myc levels and subsequent trans-activation Detection of lower levels of SNAP25 using multiple microarray systems and its functional significance in medulloblastoma BMI1 and Mel-18 oppositely regulate carcinogenesis and progression of gastric cancer.Polycomb and trithorax opposition in development and disease.Oncogenic role of the chromobox protein CBX7 in gastric cancer.The tandem affinity purification technology: an overview.Identification and characterization of Bmi-1-responding element within the human p16 promoter.RNF2 is the target for phosphorylation by the p38 MAPK and ERK signaling pathways.Expression and clinicopathological significance of Mel-18 and Bmi-1 mRNA in gastric carcinoma.Polycomb genes expression as a predictor of poor clinical outcome in children with medulloblastoma.Validation of Bmi1 as a therapeutic target of hepatocellular carcinoma in mice.Studying clonal dynamics in response to cancer therapy using high-complexity barcoding.The genomic and transcriptomic landscape of anaplastic thyroid cancer: implications for therapy.Na,K-ATPase β1-subunit is a target of sonic hedgehog signaling and enhances medulloblastoma tumorigenicity.Bmi-1: At the crossroads of physiological and pathological biology.Triple-layer dissection of the lung adenocarcinoma transcriptome: regulation at the gene, transcript, and exon levels Contextualizing the Genes Altered in Bladder Neoplasms in Pediatric andTeen Patients Allows Identifying Two Main Classes of Biological ProcessesInvolved and New Potential Therapeutic Targets Ewing tumors that do not overexpress BMI-1 are a distinct molecular subclass with variant biology: a report from the Children's Oncology Group.Medulloblastoma stem cells.An Ehrlichia chaffeensis tandem repeat protein interacts with multiple host targets involved in cell signaling, transcriptional regulation, and vesicle trafficking Systems analysis uncovers inflammatory Th/Tc17-driven modules during acute GVHD in monkey and human T cells.Bmi-1, stem cells and cancer.Polycomb group protein gene silencing, non-coding RNA, stem cells, and cancer.Commonly used tag combinations for tandem affinity purification.Genetics of medulloblastoma: clues for novel therapies.Epigenetic factors in cancer development: polycomb group proteins.The proteomics of cancer stem cells: potential clinical applications for innovative research in oncology.Cancer stem cell epigenetics and chemoresistance.Tumor suppressive pathways in the control of neurogenesis.Roles of repressive epigenetic machinery in lineage decision of T cells.Knockdown of Bmi1 inhibits bladder cancer cell growth both in vitro and in vivo by blocking cell cycle at G1 phase and inducing apoptosis.

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Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

http://www.wikidata.org/entity/Q24675945

description

2007 nî lūn-bûn

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2007 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2007 թվականի հուլիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@ast

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@en

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@nl

type

label

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

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Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@en

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@nl

prefLabel

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@ast

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@en

Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

@nl

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Molecular and Cellular Biology

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Contribution of polycomb homologues Bmi-1 and Mel-18 to medulloblastoma pathogenesis

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P2093

Brett Johnson

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Dmitri Wiederschain

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Dowdy Jackson

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James Deeds

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John D Benson

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John Taraszka

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Kimberly Bettano

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Lin Chen

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Michael D Jones

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Michael Morrissey

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P2860

The core of the polycomb repressive complex is compositionally and functionally conserved in flies and humans.

Role of histone H2A ubiquitination in Polycomb silencing

mel-18, a Polycomb group-related mammalian gene, encodes a transcriptional negative regulator with tumor suppressive activity

Microarray analysis identifies a death-from-cancer signature predicting therapy failure in patients with multiple types of cancer

Hedgehog-interacting protein is highly expressed in endothelial cells but down-regulated during angiogenesis and in several human tumors

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

Role of histone H3 lysine 27 methylation in Polycomb-group silencing

A novel function for tissue inhibitor of metalloproteinases-3 (TIMP3): inhibition of angiogenesis by blockage of VEGF binding to VEGF receptor-2

Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells

Control of developmental regulators by Polycomb in human embryonic stem cells

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4968-79

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1619634

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10.1128/MCB.02244-06

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13

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27

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Christoph Lengauer

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2007-07-01T00:00:00Z

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17452456

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medulloblastoma

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1951487

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