Role of outer surface protein D in the Borrelia burgdorferi life cycle - Wikidata - wikimedia - TriplyDB

Role of outer surface protein D in the Borrelia burgdorferi life cycle

Role of outer surface protein D in the Borrelia burgdorferi life cycle

http://www.wikidata.org/entity/Q24676358

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A chromosomally encoded virulence factor protects the Lyme disease pathogen against host-adaptive immunity Reviewing molecular adaptations of Lyme borreliosis spirochetes in the context of reproductive fitness in natural transmission cycles Borrelia burgdorferi complement regulator-acquiring surface protein 2 does not contribute to complement resistance or host infectivity.Passage through Ixodes scapularis ticks enhances the virulence of a weakly pathogenic isolate of Borrelia burgdorferi.Characterization of the highly regulated antigen BBA05 in the enzootic cycle of Borrelia burgdorferi.Modal codon usage: assessing the typical codon usage of a genome The bba64 gene of Borrelia burgdorferi, the Lyme disease agent, is critical for mammalian infection via tick bite transmission.BB0323 function is essential for Borrelia burgdorferi virulence and persistence through tick-rodent transmission cycle.Genome stability of Lyme disease spirochetes: comparative genomics of Borrelia burgdorferi plasmids.Comparative molecular analyses of Borrelia burgdorferi sensu stricto strains B31 and N40D10/E9 and determination of their pathogenicity.Molecular interactions that enable movement of the Lyme disease agent from the tick gut into the hemolymph.Borrelia burgdorferi linear plasmid 38 is dispensable for completion of the mouse-tick infectious cycle.Characterization of multiprotein complexes of the Borrelia burgdorferi outer membrane vesicles.The role of Borrelia burgdorferi outer surface proteins Borrelia burgdorferi malQ mutants utilize disaccharides and traverse the enzootic cycle Borrelia burgdorferi and tick proteins supporting pathogen persistence in the vector.A tightly regulated surface protein of Borrelia burgdorferi is not essential to the mouse-tick infectious cycle Deletion of BBA64, BBA65, and BBA66 loci does not alter the infectivity of Borrelia burgdorferi in the murine model of Lyme disease.Essential protective role attributed to the surface lipoproteins of Borrelia burgdorferi against innate defences Tick-specific borrelial antigens appear to be upregulated in American but not European patients with Lyme arthritis, a late manifestation of Lyme borreliosis.Physiologic and Genetic Factors Influencing the Zoonotic Cycle of Borrelia burgdorferi.

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

http://www.wikidata.org/entity/Q24676358

description

2007 nî lūn-bûn

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2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Infection and Immunity

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Role of outer surface protein D in the Borrelia burgdorferi life cycle

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Deborah S Beck

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Fred S Kantor

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Girish Neelakanta

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John F Anderson

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Xianzhong Liu

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Xin Li

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P2860

Outer surface protein B is critical for Borrelia burgdorferi adherence and survival within Ixodes ticks

Genomic sequence of a Lyme disease spirochaete, Borrelia burgdorferi

Lyme disease-a tick-borne spirochetosis?

Temporal changes in outer surface proteins A and C of the lyme disease-associated spirochete, Borrelia burgdorferi, during the chain of infection in ticks and mice

OspC facilitates Borrelia burgdorferi invasion of Ixodes scapularis salivary glands

Outer-surface protein C of the Lyme disease spirochete: a protein induced in ticks for infection of mammals

A bacterial genome in flux: the twelve linear and nine circular extrachromosomal DNAs in an infectious isolate of the Lyme disease spirochete Borrelia burgdorferi

The Lyme disease agent exploits a tick protein to infect the mammalian host

TROSPA, an Ixodes scapularis receptor for Borrelia burgdorferi

Borrelia burgdorferi transcriptome in the central nervous system of non-human primates.

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infectious disease

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