Hodgkin lymphoma cells express TACI and BCMA receptors and generate survival and proliferation signals in response to BAFF and APRIL - Wikidata - wikimedia - TriplyDB

Hodgkin lymphoma cells express TACI and BCMA receptors and generate survival and proliferation signals in response to BAFF and APRIL

Hodgkin lymphoma cells express TACI and BCMA receptors and generate survival and proliferation signals in response to BAFF and APRIL

http://www.wikidata.org/entity/Q24681342

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The transmembrane activator TACI triggers immunoglobulin class switching by activating B cells through the adaptor MyD88 Lessons from mathematically modeling the NF-κB pathway.Regulatory roles of the tumor necrosis factor receptor BCMA.Expression of TNF-superfamily members BAFF and APRIL in breast cancer: immunohistochemical study in 52 invasive ductal breast carcinomas.APRIL is overexpressed in cancer: link with tumor progression Selective regulation of axonal growth from developing hippocampal neurons by tumor necrosis factor superfamily member APRIL Role of the translocation partner in protection against AID-dependent chromosomal translocations Novel anti-B-cell maturation antigen antibody-drug conjugate (GSK2857916) selectively induces killing of multiple myeloma.Pediatric pulmonary Hodgkin lymphoma: analysis of 10 years data from a single center.Pathogenetic importance and therapeutic implications of NF-κB in lymphoid malignancies.B-cell activating factor-receptor specific activation of tumor necrosis factor receptor associated factor 6 and the phosphatidyl inositol 3-kinase pathway in lymphoma B cells Hodgkin's Lymphomas: A Tumor Recognized by Its Microenvironment.Innate signaling networks in mucosal IgA class switching Viral double-stranded RNA triggers Ig class switching by activating upper respiratory mucosa B cells through an innate TLR3 pathway involving BAFF.The biology of intestinal immunoglobulin A responses Different Donor Cell Culture Methods Can Influence the Developmental Ability of Cloned Sheep Embryos.Stromal endothelial cells establish a bidirectional crosstalk with chronic lymphocytic leukemia cells through the TNF-related factors BAFF, APRIL, and CD40L.Follicular dendritic cell networks of primary follicles and germinal centers: phenotype and function Clinical targeting of the TNF and TNFR superfamilies High serum levels of BAFF, APRIL, and TACI in common variable immunodeficiency.B-cell maturation antigen is modified by a single N-glycan chain that modulates ligand binding and surface retention.Recent insights into the biology of Hodgkin lymphoma: unraveling the mysteries of the Reed-Sternberg cell.B lymphocyte stimulator regulates adaptive immune responses by directly promoting dendritic cell maturation.APRIL and BCMA promote human multiple myeloma growth and immunosuppression in the bone marrow microenvironment.Crosstalk via the NF-kappaB signaling system.A proliferation-inducing ligand mediates follicular lymphoma B-cell proliferation and cyclin D1 expression through phosphatidylinositol 3-kinase-regulated mammalian target of rapamycin activation Kinetic control of negative feedback regulators of NF-kappaB/RelA determines their pathogen- and cytokine-receptor signaling specificity.Biologic features of Hodgkin lymphoma and the development of biologic prognostic factors in Hodgkin lymphoma: tumor and microenvironment.The BLyS family: toward a molecular understanding of B cell homeostasis.Genetic variation in B-cell-activating factor is associated with an increased risk of developing B-cell non-Hodgkin lymphoma.Inflammation and tissue repair markers distinguish the nodular sclerosis and mixed cellularity subtypes of classical Hodgkin's lymphoma.Involvement of BAFF and APRIL in Resistance to Apoptosis of Acute Myeloid Leukemia.The Epstein-Barr virus and the pathogenesis of lymphoma.The role of T cells in the microenvironment of Hodgkin lymphoma.Targeting B-cell maturation antigen in multiple myeloma.Prognostic Biomarkers for Hodgkin Lymphoma.Targeting BAFF and APRIL in systemic lupus erythematosus and other antibody-associated diseases.WNT5A: a motility-promoting factor in Hodgkin lymphoma.Insights into the molecular roles of heparan sulfate proteoglycans (HSPGs-syndecans) in autocrine and paracrine growth factor signaling in the pathogenesis of Hodgkin's lymphoma.New monoclonal antibodies on the horizon in multiple myeloma

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Hodgkin lymphoma cells express TACI and BCMA receptors and generate survival and proliferation signals in response to BAFF and APRIL

http://www.wikidata.org/entity/Q24681342

description

2007 nî lūn-bûn

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2007 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2007 թվականի հունվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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Hodgkin lymphoma cells express ...... in response to BAFF and APRIL

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Hodgkin lymphoma cells express ...... in response to BAFF and APRIL

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Hodgkin lymphoma cells express ...... in response to BAFF and APRIL

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Hodgkin lymphoma cells express ...... in response to BAFF and APRIL

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Hodgkin lymphoma cells express ...... in response to BAFF and APRIL

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Amy Chadburn

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Andrea Cerutti

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April Chiu

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Bing He

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Daniel M Knowles

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Elizabeth Hyjek

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Eric Sievers

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Ethel Cesarman

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Jane A Gross

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Joong-won Lee

http://www.w3.org/2001/XMLSchema#string

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NF-AT activation induced by a CAML-interacting member of the tumor necrosis factor receptor superfamily

APRIL, a new ligand of the tumor necrosis factor family, stimulates tumor cell growth

Biologically active APRIL is secreted following intracellular processing in the Golgi apparatus by furin convertase

An endogenous hybrid mRNA encodes TWE-PRIL, a functional cell surface TWEAK-APRIL fusion protein

The level of TACI gene expression in myeloma cells is associated with a signature of microenvironment dependence versus a plasmablastic signature

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

MEK/ERK pathway is aberrantly active in Hodgkin disease: a signaling pathway shared by CD30, CD40, and RANK that regulates cell proliferation and survival

Expression of BCMA, TACI, and BAFF-R in multiple myeloma: a mechanism for growth and survival

DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL

The role of APRIL and BAFF in lymphocyte activation

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