Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells - Wikidata - wikimedia - TriplyDB

Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells

Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells

http://www.wikidata.org/entity/Q24683705

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The 15-lipoxygenase-1 product 13-S-hydroxyoctadecadienoic acid down-regulates PPAR-delta to induce apoptosis in colorectal cancer cells Non-canonical Notch signaling: emerging role and mechanism Aspirin induces apoptosis through mitochondrial cytochrome c release Aspirin induces apoptosis through release of cytochrome c from mitochondria Role of mitochondria in aspirin-induced apoptosis in human gastric epithelial cells Inhibition of store-operated calcium entry contributes to the anti-proliferative effect of non-steroidal anti-inflammatory drugs in human colon cancer cells Dysregulation of integrin-linked kinase (ILK) signaling in colonic polyposis Effect of non-steroidal anti-inflammatory drugs on colon carcinoma Caco-2 cell responsiveness to topoisomerase inhibitor drugs Differential direct effects of cyclo-oxygenase-1/2 inhibition on proteoglycan turnover of human osteoarthritic cartilage: an in vitro study Aspirin and indomethacin exhibit antiproliferative effects and induce apoptosis in T98G human glioblastoma cells Aspirin induces apoptosis through the inhibition of proteasome function Aspirin blocks proliferation in colon cells by inducing a G1 arrest and apoptosis through activation of the checkpoint kinase ATM Acetyl salicylic acid augments functional recovery following sciatic nerve crush in mice PPARdelta is an APC-regulated target of nonsteroidal anti-inflammatory drugs Indomethacin-induced apoptosis in esophageal adenocarcinoma cells involves upregulation of Bax and translocation of mitochondrial cytochrome C independent of COX-2 expression A cyclooxygenase-2 inhibitor (SC-58125) blocks growth of established human colon cancer xenografts Inhibition of cell transformation by sulindac sulfide is confined to specific oncogenic pathways Chemoprevention of colorectal cancer Inhibition of rat colon tumors by sulindac and sulindac sulfone is independent of K-ras (codon 12) mutation Non-steroidol anti-inflammatory drug effect on crypt cell proliferation and apoptosis during initiation of rat colon carcinogenesis Aspirin delimits platelet life span by proteasomal inhibition Growth-suppressive effect of non-steroidal anti-inflammatory drugs on 11 colon-cancer cell lines and fluorescence differential display of genes whose expression is influenced by sulindac.Effects of sulindac on sporadic colorectal adenomatous polyps The role of prostaglandin synthesis in prostate cancer.Colorectal cancer: update on recent advances and their impact on screening protocols.Sulindac treatment alters collagen and matrilysin expression in adenomas of ApcMin/+ mice.Effects of nonsteroidal anti-inflammatory drugs on Helicobacter pylori-infected gastric mucosae of mice: apoptosis, cell proliferation, and inflammatory activity.Potential utility of COX-2 inhibitors in chemoprevention and chemotherapy.Peroxisome Proliferator-Activated Receptors Protect against Apoptosis via 14-3-3.Phospho-sulindac (OXT-922) inhibits the growth of human colon cancer cell lines: a redox/polyamine-dependent effect Molecular causes of colon cancer.The role of NSAIDs in the prevention of colon cancer.Inhibition of apoptosis in normal and transformed intestinal epithelial cells by cAMP through induction of inhibitor of apoptosis protein (IAP)-2 Rectal epithelial apoptosis in familial adenomatous polyposis patients treated with sulindac.Mechanisms underlying nonsteroidal antiinflammatory drug-mediated apoptosis Sulindac inhibits neointimal formation after arterial injury in wild-type and apolipoprotein E-deficient mice.Investigation of nuclear nano-morphology marker as a biomarker for cancer risk assessment using a mouse model.Sodium salicylate induces apoptosis via p38 mitogen-activated protein kinase but inhibits tumor necrosis factor-induced c-Jun N-terminal kinase/stress-activated protein kinase activation.The metabolism and pharmacokinetics of phospho-sulindac (OXT-328) and the effect of difluoromethylornithine.Nimesulide inhibits proliferation via induction of apoptosis and cell cycle arrest in human gastric adenocarcinoma cell line.

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P2860

Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells

http://www.wikidata.org/entity/Q24683705

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Sulindac sulfide, an aspirin-l ...... -29 colon adenocarcinoma cells

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Sulindac sulfide, an aspirin-l ...... -29 colon adenocarcinoma cells

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Sulindac sulfide, an aspirin-l ...... -29 colon adenocarcinoma cells

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B Rigas

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L L Tsai

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P2860

Isolation of the human cdk2 gene that encodes the cyclin A- and adenovirus E1A-associated p33 kinase

Identification of G1 kinase activity for cdk6, a novel cyclin D partner

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Measurement of protein using bicinchoninic acid

Formation and activation of a cyclin E-cdk2 complex during the G1 phase of the human cell cycle

Identification and properties of an atypical catalytic subunit (p34PSK-J3/cdk4) for mammalian D type G1 cyclins

Isolation of a human cyclin cDNA: evidence for cyclin mRNA and protein regulation in the cell cycle and for interaction with p34cdc2

Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

Cyclin D1 is a nuclear protein required for cell cycle progression in G1

D-type cyclin-dependent kinase activity in mammalian cells

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apoptotic process

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185223

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