Deletion, mutation, and loss of expression of KLF6 in human prostate cancer - Wikidata - wikimedia - TriplyDB

Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

http://www.wikidata.org/entity/Q24684163

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Identification and characterization of putative tumor suppressor NGB, a GTP-binding protein that interacts with the neurofibromatosis 2 protein Gene expression profiling predicts clinical outcome of prostate cancer Mammalian Krüppel-like factors in health and diseases Molecular pathology of prostate cancer Krüppel cripples prostate cancer: KLF6 progress and prospects Frequent somatic mutations of the transcription factor ATBF1 in human prostate cancer Ubiquitin E3 ligase WWP1 as an oncogenic factor in human prostate cancer Tumor suppressor and hepatocellular carcinoma KLF6 induces apoptosis in prostate cancer cells through up-regulation of ATF3 p21 in cancer: intricate networks and multiple activities Nuclear expression of KLF6 tumor suppressor factor is highly associated with overexpression of ERBB2 oncoprotein in ductal breast carcinomas Aberrant expression of krUppel-like factor 6 protein in colorectal cancers.TGF-beta regulates the expression of transcription factor KLF6 and its splice variants and promotes co-operative transactivation of common target genes through a Smad3-Sp1-KLF6 interaction Nucleo-cytoplasmic localization domains regulate Krüppel-like factor 6 (KLF6) protein stability and tumor suppressor function Involvement of Kruppel-like factor 6 (KLF6) mutation in the development of nonpolypoid colorectal carcinoma.Functional role of the KLF6 tumour suppressor gene in gastric cancer.KLF6 loss of function in human prostate cancer progression is implicated in resistance to androgen deprivation.Epigenetics of prostate cancer: beyond DNA methylation Energy restriction-mimetic agents induce apoptosis in prostate cancer cells in part through epigenetic activation of KLF6 tumor suppressor gene expression.Emerging roles of Kruppel-like factor 6 and Kruppel-like factor 6 splice variant 1 in ovarian cancer progression and treatment.KLF6-SV1 overexpression accelerates human and mouse prostate cancer progression and metastasis.Developmental regulation of yolk sac hematopoiesis by Kruppel-like factor 6 Zinc and cancer: implications for LIV-1 in breast cancer.Prevalent mutations in prostate cancer.Establishment of transgenic mice carrying gene encoding human zinc finger protein 191.Downregulation of KLF6 is an early event in hepatocarcinogenesis, and stimulates proliferation while reducing differentiation.Molecular profiling of prostatic acinar morphogenesis identifies PDCD4 and KLF6 as tissue architecture-specific prognostic markers in prostate cancer.Ras promotes growth by alternative splicing-mediated inactivation of the KLF6 tumor suppressor in hepatocellular carcinoma.Expression and Function of Kruppel Like-Factors (KLF) in Carcinogenesis.Interaction of KLF6 and Sp1 regulates basigin-2 expression mediated proliferation, invasion and metastasis in hepatocellular carcinoma.Induction of Kruppel-like factor 5 expression by androgens results in increased CXCR4-dependent migration of prostate cancer cells in vitro Essential role of KLF5 transcription factor in cell proliferation and differentiation and its implications for human diseases.Acid ceramidase upregulation in prostate cancer: role in tumor development and implications for therapy Biology of Krüppel-like factor 6 transcriptional regulator in cell life and death.The complexity of prostate cancer: genomic alterations and heterogeneity.Regulatory variation: an emerging vantage point for cancer biology.Molecular pathways in prostate cancer.Analysis of human prostate cancers and cell lines for mutations in the TP53 and KLF6 tumour suppressor genes.Krüppel-like factors in mammalian stem cells and development.Effects of Kruppel-like factor 6 on osteosarcoma cell biological behavior.

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P2860

Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

http://www.wikidata.org/entity/Q24684163

description

2003 nî lūn-bûn

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2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2003 թվականի ապրիլին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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type

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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prefLabel

Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

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S0002-9440(10)63930-2

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The American Journal of Pathology

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Deletion, mutation, and loss of expression of KLF6 in human prostate cancer

@en

P2093

Eija-Riitta Hyytinen

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Heikki J Helin

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Henry F Frierson

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Pasi A Koivisto

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Robert L Vessella

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P2860

A tale of three fingers: the family of mammalian Sp/XKLF transcription factors

A possible tumor suppressor role of the KLF5 transcription factor in human breast cancer

KLF6, a candidate tumor suppressor gene mutated in prostate cancer

Three distinct regions of allelic loss at 13q14, 13q21-22, and 13q33 in prostate cancer.

Deletion at 13q21 is associated with aggressive prostate cancers.

An 800-kb region of deletion at 13q14 in human prostate and other carcinomas.

Defining a common region of deletion at 13q21 in human cancers.

Localization of multiple melanoma tumor-suppressor genes on chromosome 11 by use of homozygosity mapping-of-deletions analysis.

Tissue, cell type, and breast cancer stage-specific expression of a TGF-beta inducible early transcription factor gene.

Inactivation of the tumor suppressor PTEN/MMAC1 in advanced human prostate cancer through loss of expression.

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