Characterization of methylthioadenosin phosphorylase (MTAP) expression in malignant melanoma - Wikidata - wikimedia - TriplyDB

Characterization of methylthioadenosin phosphorylase (MTAP) expression in malignant melanoma

Characterization of methylthioadenosin phosphorylase (MTAP) expression in malignant melanoma

http://www.wikidata.org/entity/Q24685552

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Functional identification of APIP as human mtnB, a key enzyme in the methionine salvage pathway Identification of a shared genetic susceptibility locus for coronary heart disease and periodontitis Next generation sequencing of prostate cancer from a patient identifies a deficiency of methylthioadenosine phosphorylase, an exploitable tumor target Associations of 9p21 variants with cutaneous malignant melanoma, nevi, and pigmentation phenotypes in melanoma-prone families with and without CDKN2A mutations Long Non-coding RNA ANRIL and Polycomb in Human Cancers and Cardiovascular Disease Promoter-hypermethylation is causing functional relevant downregulation of methylthioadenosine phosphorylase (MTAP) expression in hepatocellular carcinoma Strong expression of methylthioadenosine phosphorylase (MTAP) in human colon carcinoma cells is regulated by TCF1/[beta]-catenin Selective killing of tumors deficient in methylthioadenosine phosphorylase: a novel strategy.Reduced expression of beta-catenin inhibitor Chibby in colon carcinoma cell lines.Promoter hypermethylation of CDH1, FHIT, MTAP and PLAGL1 in gastric adenocarcinoma in individuals from Northern Brazil.Expression of Hugl-1 is strongly reduced in malignant melanoma.Functional modulation of IGF-binding protein-3 expression in melanoma Aberrant DNA methylation in malignant melanoma Variants at the 9p21 locus and melanoma risk Germline Mutations in Mtap Cooperate with Myc to Accelerate Tumorigenesis in Mice Genome-wide association study identifies variants at 9p21 and 22q13 associated with development of cutaneous nevi Expression of MTAP inhibits tumor-related phenotypes in HT1080 cells via a mechanism unrelated to its enzymatic function.Downregulated MTAP expression in myxofibrosarcoma: A characterization of inactivating mechanisms, tumor suppressive function, and therapeutic relevance.Prognostic relevance of P-cadherin expression in melanocytic skin tumours analysed by high-throughput tissue microarrays.Immunohistochemistry in melanocytic proliferative lesions.EFA (9-beta-D-erythrofuranosyladenine) is an effective salvage agent for methylthioadenosine phosphorylase-selective therapy of T-cell acute lymphoblastic leukemia with L-alanosine.Characterization of the Methylthioadenosine Phosphorylase Polymorphism rs7023954 - Incidence and Effects on Enzymatic Function in Malignant Melanoma Epigenetic events in malignant melanoma.A potential predictive marker for response to interferon in malignant melanoma.Comprehensive analysis of CDKN2A (p16INK4A/p14ARF) and CDKN2B genes in 53 melanoma index cases considered to be at heightened risk of melanoma Characterization and Prognostic Significance of Methylthioadenosine Phosphorylase Deficiency in Nasopharyngeal Carcinoma.Mice heterozygous for germ-line mutations in methylthioadenosine phosphorylase (MTAP) die prematurely of T-cell lymphoma.Targeting tumors that lack methylthioadenosine phosphorylase (MTAP) activity: current strategies Suppressive effects of tumor cell-derived 5'-deoxy-5'-methylthioadenosine on human T cells Metabolic Hallmarks of Tumor and Immune Cells in the Tumor Microenvironment Genomic Study of Cardiovascular Continuum Comorbidity.Are STATS arginine-methylated?Promoter methylation profiling of 30 genes in human malignant melanoma.Analysis of p16 expression and allelic imbalance / loss of heterozygosity of 9p21 in cutaneous squamous cell carcinomas.Regulation of human methylthioadenosine phosphorylase gene by the CBF (CCAAT binding factor)/NF-Y (nuclear factor-Y)Deletion at chromosome arm 9p in relation to BRAF/NRAS mutations and prognostic significance for primary melanoma.Observations of the genomic landscape beyond 1p19q deletions and EGFR amplification in glioma.Methylthioadenosine phosphorylase represents a predictive marker for response to adjuvant interferon therapy in patients with malignant melanoma.Direct and tumor microenvironment mediated influences of 5'-deoxy-5'-(methylthio)adenosine on tumor progression of malignant melanoma.A phase II multicenter study of L-alanosine, a potent inhibitor of adenine biosynthesis, in patients with MTAP-deficient cancer.

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Characterization of methylthioadenosin phosphorylase (MTAP) expression in malignant melanoma

http://www.wikidata.org/entity/Q24685552

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2003 nî lūn-bûn

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2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2003 թվականի օգոստոսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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Characterization of methylthio ...... pression in malignant melanoma

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The American Journal of Pathology

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Characterization of methylthio ...... pression in malignant melanoma

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Anja-Katrin Bosserhoff

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Iris Behrmann

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Marion Schuierer

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Peter C Heinrich

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Reinhard Buettner

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Susanne Wallner

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Waraporn Komyod

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P2860

Cloning of a novel malignant melanoma-derived growth-regulatory protein, MIA

Methylthioadenosine phosphorylase, a gene frequently codeleted with p16(cdkN2a/ARF), acts as a tumor suppressor in a breast cancer cell line

High- and low-dose interferon alfa-2b in high-risk melanoma: first analysis of intergroup trial E1690/S9111/C9190.

Arginine methylation of STAT1 modulates IFNalpha/beta-induced transcription.

Interferon alfa therapy for malignant melanoma: a systematic review of randomized controlled trials.

Adjuvant therapy for patients with high-risk malignant melanoma.

Loss of E-cadherin expression in melanoma cells involves up-regulation of the transcriptional repressor Snail.

Comparative histological, histochemical, immunohistochemical and biochemical studies on oestrogen receptors, lectin receptors, and Barr bodies in human breast cancer.

Detection of invasion-related chromosomal changes in highly and weakly invasive melanoma cell clones by a modified comparative genomic hybridization approach.

Gene deletion chemoselectivity: codeletion of the genes for p16(INK4), methylthioadenosine phosphorylase, and the alpha- and beta-interferons in human pancreatic cell carcinoma lines and its implications for chemotherapy.

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