A new direction in the pathogenesis of idiopathic pulmonary fibrosis? - Wikidata - wikimedia - TriplyDB

A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

http://www.wikidata.org/entity/Q24798888

about

WNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis Focusing on diffuse (interstitial) lung disease: a rapidly evolving field TGF-beta1 induces human alveolar epithelial to mesenchymal cell transition (EMT)Macrophage metalloelastase (MMP-12) deficiency does not alter bleomycin-induced pulmonary fibrosis in mice.Involvement of matrix metalloproteinases (MMPs) and inflammasome pathway in molecular mechanisms of fibrosis Soluble TNF mediates the transition from pulmonary inflammation to fibrosis Pulmonary fibrosis in response to environmental cues and molecular targets involved in its pathogenesis Transforming growth factor-beta antagonizes alveolar type II cell proliferation induced by keratinocyte growth factor Airway reopening through catastrophic events in a hierarchical network.Cigarette smoke drives small airway remodeling by induction of growth factors in the airway wall.The effect of TGF-β1 and Smad7 gene transfer on the phenotypic changes of rat alveolar epithelial cells.Proinflammatory cytokines are involved in the initiation of the abnormal matrix process in pseudoexfoliation syndrome/glaucoma.Idiopathic interstitial pneumonias: progress in classification, diagnosis, pathogenesis and management Grape seed pro-anthocyanidins ameliorates radiation-induced lung injury.Cigarette smoke induces cyclooxygenase-2 and microsomal prostaglandin E2 synthase in human lung fibroblasts: implications for lung inflammation and cancer.Proapoptotic Bid is required for pulmonary fibrosis The effects of cigarette smoke condensate on vocal fold transepithelial resistance and inflammatory signaling in vocal fold fibroblasts.Severity of lung injury in cyclooxygenase-2-deficient mice is dependent on reduced prostaglandin E(2) production.Aberrant Wnt/beta-catenin pathway activation in idiopathic pulmonary fibrosis.Transfer of the active form of transforming growth factor-beta 1 gene to newborn rat lung induces changes consistent with bronchopulmonary dysplasia The Role of PPARs in Lung Fibrosis.Pulmonary fibrosis: searching for model answers.Role of matrix metalloproteinases in the development of airway inflammation and remodeling.Smad3 signaling involved in pulmonary fibrosis and emphysema.Chronic obstructive pulmonary disease: role of matrix metalloproteases and future challenges of drug therapy.Role of hypoxia-inducible factor 1, α subunit and cAMP-response element binding protein 1 in synergistic release of interleukin 8 by prostaglandin E2 and nickel in lung fibroblasts Pleural mesothelial cell transformation into myofibroblasts and haptotactic migration in response to TGF-beta1 in vitro.Overexpression of fibroblast growth factor-10 during both inflammatory and fibrotic phases attenuates bleomycin-induced pulmonary fibrosis in mice.Haploinsufficiency of the folliculin gene leads to impaired functions of lung fibroblasts in patients with Birt-Hogg-Dubé syndrome.Peroxisome proliferator-activated receptor-gamma ligands induce heme oxygenase-1 in lung fibroblasts by a PPARgamma-independent, glutathione-dependent mechanism.The Witschi Hypothesis revisited after 35 years: genetic proof from SP-C BRICHOS domain mutations Cellular therapies for lung disease: a distant horizon.Myofibroblast repair mechanisms post-inflammatory response: a fibrotic perspective.Inhibition of TRPM7 channels prevents proliferation and differentiation of human lung fibroblasts.The Cellular and Molecular Mechanism of Radiation-Induced Lung Injury.Biomarkers of fibroproliferative healing in fibrosing idiopathic interstitial pneumonias.Age-Dependent Susceptibility to Pulmonary Fibrosis Is Associated with NLRP3 Inflammasome Activation Essential roles for angiotensin receptor AT1a in bleomycin-induced apoptosis and lung fibrosis in mice.Splicosomal and serine and arginine-rich splicing factors as targets for TGF-β.Role of the chemokine receptor CXCR2 in bleomycin-induced pulmonary inflammation and fibrosis.

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

http://www.wikidata.org/entity/Q24798888

description

2002 nî lūn-bûn

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2002年の論文

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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A new direction in the pathogenesis of idiopathic pulmonary fibrosis?

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Jack Gauldie

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Martin Kolb

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Patricia J Sime

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P2860

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

Localisation of transforming growth factor beta1 and beta3 mRNA transcripts in normal and fibrotic human lung

Costimulation of fibroblast collagen and transforming growth factor beta1 gene expression by monocyte chemoattractant protein-1 via specific receptors

Epithelial-mesenchymal interactions in the pathogenesis of asthma.

The molecular basis of lung morphogenesis.

Fibrosis of the lung and other tissues: new concepts in pathogenesis and treatment.

Transient expression of IL-1beta induces acute lung injury and chronic repair leading to pulmonary fibrosis.

Atherosclerosis: the emerging role of inflammation and the CD40-CD40 ligand system

Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts

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