Growth factors in idiopathic pulmonary fibrosis: relative roles - Wikidata - wikimedia - TriplyDB

Growth factors in idiopathic pulmonary fibrosis: relative roles

Growth factors in idiopathic pulmonary fibrosis: relative roles

http://www.wikidata.org/entity/Q24798972

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Bone morphogenetic protein-4 inhibitor gremlin is overexpressed in idiopathic pulmonary fibrosis Deficiency in type 1 insulin-like growth factor receptor in mice protects against oxygen-induced lung injury The "Goldilocks effect" in cystic fibrosis: identification of a lung phenotype in the cftr knockout and heterozygous mouse Focusing on diffuse (interstitial) lung disease: a rapidly evolving field TGF-beta1 induces human alveolar epithelial to mesenchymal cell transition (EMT)Soluble TNF mediates the transition from pulmonary inflammation to fibrosis Small molecule receptor tyrosine kinase inhibitor of platelet-derived growth factor signaling (SU9518) modifies radiation response in fibroblasts and endothelial cells.Cell-specific gene expression in patients with usual interstitial pneumonia.Laser capture microdissection reveals dose-response of gene expression in situ consequent to asbestos exposure.Regulation of transforming growth factor-beta1 (TGF-β1)-induced pro-fibrotic activities by circadian clock gene BMAL1.Lymphatics in idiopathic pulmonary fibrosis: new insights into an old disease.Potential risk of TNF inhibitors on the progression of interstitial lung disease in patients with rheumatoid arthritis.New therapeutic targets in idiopathic pulmonary fibrosis. Aiming to rein in runaway wound-healing responses.TGF-β and TGF-β/Smad signaling in the interactions between Echinococcus multilocularis and its hosts Small interfering RNAs (siRNAs) targeting TGF-beta1 mRNA suppress asbestos-induced expression of TGF-beta1 and CTGF in fibroblasts.Human pulmonary fibroblasts exhibit altered interleukin-4 and interleukin-13 receptor subunit expression in idiopathic interstitial pneumonia Severity of lung injury in cyclooxygenase-2-deficient mice is dependent on reduced prostaglandin E(2) production.Treatment with etanercept in a patient with rheumatoid arthritis-associated interstitial lung disease Caveolin-1: a critical regulator of lung fibrosis in idiopathic pulmonary fibrosis Impact of interleukin-13 responsiveness on the synthetic and proliferative properties of Th1- and Th2-type pulmonary granuloma fibroblasts Reduced FoxO3a expression causes low autophagy in idiopathic pulmonary fibrosis fibroblasts on collagen matrices.Current and novel drug therapies for idiopathic pulmonary fibrosis.Inhibition of platelet-derived growth factor signaling attenuates pulmonary fibrosis.A review of current and novel therapies for idiopathic pulmonary fibrosis Idiopathic pulmonary fibrosis: a decade of progress.Imatinib mesylate inhibits fibrogenesis in asbestos-induced interstitial pneumonia.Pulmonary fibrosis induced by H5N1 viral infection in mice Fibrotic extracellular matrix activates a profibrotic positive feedback loop.Host-directed therapeutics for tuberculosis: can we harness the host?Emerging therapeutic interventions for idiopathic pulmonary fibrosis.Recent evidence for pharmacological treatment of idiopathic pulmonary fibrosis.Inhibition of connective tissue growth factor attenuates paraquat-induced lung fibrosis in a human MRC-5 cell line.MicroRNAs in non-small cell lung cancer and idiopathic pulmonary fibrosis.Decreased expression of autophagic beclin 1 protein in idiopathic pulmonary fibrosis fibroblasts.Anti-TNFα therapy in inflammatory lung diseases.Insulin-like growth factor binding protein 5 enhances survival of LX2 human hepatic stellate cells Deficient repair response of IPF fibroblasts in a co-culture model of epithelial injury and repair.Tumor necrosis factor-alpha enhances both epithelial-mesenchymal transition and cell contraction induced in A549 human alveolar epithelial cells by transforming growth factor-beta1.Collagen I promotes epithelial-to-mesenchymal transition in lung cancer cells via transforming growth factor-beta signaling.Tumor necrosis factor-alpha induces transforming growth factor-beta1 expression in lung fibroblasts through the extracellular signal-regulated kinase pathway.

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Growth factors in idiopathic pulmonary fibrosis: relative roles

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Jeremy T Allen

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Monica A Spiteri

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P2860

Tumor necrosis factor alpha suppresses the induction of connective tissue growth factor by transforming growth factor-beta in normal and scleroderma fibroblasts

Role of endothelin-1 in lung disease

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

Interstitial fibrosis and growth factors

Attenuation of bleomycin-induced pneumopathy in mice by a caspase inhibitor.

The connective tissue growth factor/cysteine-rich 61/nephroblastoma overexpressed (CCN) family.

A histologically distinctive interstitial pneumonia induced by overexpression of the interleukin 6, transforming growth factor beta 1, or platelet-derived growth factor B gene

Idiopathic pulmonary fibrosis: clinical relevance of pathologic classification.

Cultured lung fibroblasts isolated from patients with idiopathic pulmonary fibrosis have a diminished capacity to synthesize prostaglandin E2 and to express cyclooxygenase-2.

Expression and localization of tumor necrosis factor-alpha and its mRNA in idiopathic pulmonary fibrosis

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pulmonary fibrosis

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