State of the art: why do the lungs of patients with cystic fibrosis become infected and why can't they clear the infection?
about
17beta-Estradiol inhibits Ca2+-dependent homeostasis of airway surface liquid volume in human cystic fibrosis airway epithelia.Percutaneous lines for delivering intravenous antibiotics in people with cystic fibrosisPercutaneous lines for delivering intravenous antibiotics in people with cystic fibrosisThe relevance of sweat testing for the diagnosis of cystic fibrosis in the genomic era.SPLUNC1 regulates airway surface liquid volume by protecting ENaC from proteolytic cleavageThe neutrophil serine protease inhibitor serpinb1 preserves lung defense functions in Pseudomonas aeruginosa infectionRelation of exaggerated cytokine responses of CF airway epithelial cells to PAO1 adherenceMucociliary and long-term particle clearance in airways of patients with immotile ciliaEpithelial Sodium and Chloride Channels and AsthmaMolecular pathways for intracellular cholesterol accumulation: common pathogenic mechanisms in Niemann-Pick disease Type C and cystic fibrosisMolecular basis for pH-dependent mucosal dehydration in cystic fibrosis airwaysOrigins of cystic fibrosis lung diseaseAirway hydration and COPDA non-classical LysR-type transcriptional regulator PA2206 is required for an effective oxidative stress response in Pseudomonas aeruginosaPulmonary matrix metalloproteinase-9 activity in mechanically ventilated children with respiratory syncytial virus.Antibiotic management of lung infections in cystic fibrosis. I. The microbiome, methicillin-resistant Staphylococcus aureus, gram-negative bacteria, and multiple infectionsAntibiotic management of lung infections in cystic fibrosis. II. Nontuberculous mycobacteria, anaerobic bacteria, and fungiLung Microbiota and Its Impact on the Mucosal Immune Phenotype.Role of excessive inflammatory response to Stenotrophomonas maltophilia lung infection in DBA/2 mice and implications for cystic fibrosisCystic fibrosis pigs develop lung disease and exhibit defective bacterial eradication at birth.The microbiome in pediatric cystic fibrosis patients: the role of shared environment suggests a window of intervention.Neutrophil extracellular trap (NET)-mediated killing of Pseudomonas aeruginosa: evidence of acquired resistance within the CF airway, independent of CFTR.SPLUNC1 expression reduces surface levels of the epithelial sodium channel (ENaC) in Xenopus laevis oocytes.Cystic fibrosis transmembrane conductance regulator is involved in airway epithelial wound repairLinking increased airway hydration, ciliary beating, and mucociliary clearance through ENaC inhibitionAbnormal trafficking and degradation of TLR4 underlie the elevated inflammatory response in cystic fibrosis.Revisiting the host as a growth mediumPharmacological modulation of the AKT/microRNA-199a-5p/CAV1 pathway ameliorates cystic fibrosis lung hyper-inflammationDefective adenosine-stimulated cAMP production in cystic fibrosis airway epithelia: a novel role for CFTR in cell signaling.Phenotypic profiling of Scedosporium aurantiacum, an opportunistic pathogen colonizing human lungs.Progression of pulmonary hyperinflation and trapped gas associated with genetic and environmental factors in children with cystic fibrosis.Effect of airway Pseudomonas aeruginosa isolation and infection on steady-state bronchiectasis in Guangzhou, China.Functional analysis of a promoter variant identified in the CFTR gene in cis of a frameshift mutation.Pediatric Cystic Fibrosis Sputum Can Be Chemically Dynamic, Anoxic, and Extremely Reduced Due to Hydrogen Sulfide Formation.The Dynamics of Disease Progression in Cystic Fibrosis.Hyper-osmolarity and calcium chelation: Effects on cystic fibrosis mucus.Assessment of hypoxia in children with cystic fibrosis.Soluble mediators, not cilia, determine airway surface liquid volume in normal and cystic fibrosis superficial airway epithelia.Glucocorticoid receptor gene polymorphisms associated with progression of lung disease in young patients with cystic fibrosisImplications of rewiring bacterial quorum sensing.
P2860
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P2860
State of the art: why do the lungs of patients with cystic fibrosis become infected and why can't they clear the infection?
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
State of the art: why do the l ...... an't they clear the infection?
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State of the art: why do the l ...... an't they clear the infection?
@en
State of the art: why do the l ...... an't they clear the infection?
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label
State of the art: why do the l ...... an't they clear the infection?
@ast
State of the art: why do the l ...... an't they clear the infection?
@en
State of the art: why do the l ...... an't they clear the infection?
@nl
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State of the art: why do the l ...... an't they clear the infection?
@ast
State of the art: why do the l ...... an't they clear the infection?
@en
State of the art: why do the l ...... an't they clear the infection?
@nl
P2860
P921
P3181
P356
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State of the art: why do the l ...... an't they clear the infection?
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P2093
James F Chmiel
Pamela B Davis
P2860
P2888
P3181
P356
10.1186/1465-9921-4-8
P407
P577
2003-08-27T00:00:00Z
P5875
P6179
1048510246