An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema - Wikidata - wikimedia - TriplyDB

An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema

An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema

http://www.wikidata.org/entity/Q24802958

about

A distinctive alveolar macrophage activation state induced by cigarette smoking Characterization of T lymphocytes in chronic obstructive pulmonary disease Cigarette smoke-induced pulmonary emphysema in scid-mice. Is the acquired immune system required?Dust events, pulmonary diseases and immune system Heterogeneity of lung mononuclear phagocytes in chronic obstructive pulmonary disease Interrelated role of cigarette smoking, oxidative stress, and immune response in COPD and corresponding treatments Role of the inflammasome-caspase1/11-IL-1/18 axis in cigarette smoke driven airway inflammation: an insight into the pathogenesis of COPD P2X7 receptor and caspase 1 activation are central to airway inflammation observed after exposure to tobacco smoke Clinical aspects and cytokine response in severe H1N1 influenza A virus infection Role of LTB₄ in the pathogenesis of elastase-induced murine pulmonary emphysema Polarized localization of epithelial CXCL11 in chronic obstructive pulmonary disease and mechanisms of T cell egression Focused Analysis of Exome Sequencing Data for Rare Germline Mutations in Familial and Sporadic Lung Cancer Tobacco use induces anti-apoptotic, proliferative patterns of gene expression in circulating leukocytes of Caucasian males.Chemokine receptor CXCR3 is important for lung tissue damage and airway remodeling induced by short-term exposure to cigarette smoking in mice Attenuating immune pathology using a microbial-based intervention in a mouse model of cigarette smoke-induced lung inflammation.Serum IP-10 as a biomarker of human rhinovirus infection at exacerbation of COPD Essential role for autophagy in the maintenance of immunological memory against influenza infection.Smoking-dependent reprogramming of alveolar macrophage polarization: implication for pathogenesis of chronic obstructive pulmonary disease.Alveolar macrophage recruitment and activation by chronic second hand smoke exposure in mice.DAP12 is required for macrophage recruitment to the lung in response to cigarette smoke and chemotaxis toward CCL2 Human rhinovirus proteinase 2A induces TH1 and TH2 immunity in patients with chronic obstructive pulmonary disease CD46 protects against chronic obstructive pulmonary disease.Cytotoxic potential of lung CD8(+) T cells increases with chronic obstructive pulmonary disease severity and with in vitro stimulation by IL-18 or IL-15.IL-17RA is required for CCL2 expression, macrophage recruitment, and emphysema in response to cigarette smoke.Pneumocystis jirovecii colonization is associated with enhanced Th1 inflammatory gene expression in lungs of humans with chronic obstructive pulmonary disease.Chronic cigarette smoke exposure generates pathogenic T cells capable of driving COPD-like disease in Rag2-/- mice.Human CD56+ cytotoxic lung lymphocytes kill autologous lung cells in chronic obstructive pulmonary disease TNFα and IFNγ synergistically enhance transcriptional activation of CXCL10 in human airway smooth muscle cells via STAT-1, NF-κB, and the transcriptional coactivator CREB-binding protein.Role of CCR5 in IFN-gamma-induced and cigarette smoke-induced emphysema.Role of th17 cell and autoimmunity in chronic obstructive pulmonary disease.Antiviral immune responses in H5N1-infected human lung tissue and possible mechanisms underlying the hyperproduction of interferon-inducible protein IP-10.Role for cathepsin K in emphysema in smoke-exposed guinea pigs Biodegradable nanoparticle-entrapped vaccine induces cross-protective immune response against a virulent heterologous respiratory viral infection in pigs Autoantibodies in patients with chronic obstructive pulmonary disease Nontypeable Haemophilus influenzae in chronic obstructive pulmonary disease and lung cancer.Effect of an MMP-9/MMP-12 inhibitor on smoke-induced emphysema and airway remodelling in guinea pigs.Novel insights into the aetiology and pathophysiology of increased airway inflammation during COPD exacerbations.New immune pathways from chronic post-viral lung disease Imaging of pulmonary emphysema: a pictorial review.Current and former smoking and risk for venous thromboembolism: a systematic review and meta-analysis

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An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema

http://www.wikidata.org/entity/Q24802958

description

2004 nî lūn-bûn

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2004 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2004 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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An immune basis for lung paren ...... ulmonary disease and emphysema

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David B Corry

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Dorothy E Lewis

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Farrah Kheradmand

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Joan Hacken

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Joseph Huh

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Li-Zhen Song

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Rafael Espada

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Sandra Grumelli

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P2860

Interferon-inducible T cell alpha chemoattractant (I-TAC): a novel non-ELR CXC chemokine with potent activity on activated T cells through selective high affinity binding to CXCR3

The ligands of CXC chemokine receptor 3, I-TAC, Mig, and IP10, are natural antagonists for CCR3

Polymorphisms in the IL13, IL13RA1, and IL4RA genes and rate of decline in lung function in smokers

Hydrolysis of a broad spectrum of extracellular matrix proteins by human macrophage elastase

Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease. NHLBI/WHO Global Initiative for Chronic Obstructive Lung Disease (GOLD) Workshop summary

Chemokines: a new classification system and their role in immunity

How matrix metalloproteinases regulate cell behavior

Decreased allergic lung inflammatory cell egression and increased susceptibility to asphyxiation in MMP2-deficiency

Chronic obstructive pulmonary disease. Inflammation of small airways and lung parenchyma.

Airway inflammation and remodeling in asthma.

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chronic obstructive pulmonary disease

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