Gene therapy in rheumatoid arthritis: how to target joint destruction? - Wikidata - wikimedia - TriplyDB

Gene therapy in rheumatoid arthritis: how to target joint destruction?

Gene therapy in rheumatoid arthritis: how to target joint destruction?

http://www.wikidata.org/entity/Q24804867

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Fibroblast biology. Role of synovial fibroblasts in the pathogenesis of rheumatoid arthritis Ex vivo gene transfer in the years to come.p53 in rheumatoid arthritis: friend or foe?Gene transfer as a future therapy for rheumatoid arthritis.Gene therapy for human osteoarthritis: principles and clinical translation.Inhibition of cartilage destruction by double gene transfer of IL-1Ra and IL-10 involves the activin pathway.Ribozymes that inhibit the production of matrix metalloproteinase 1 reduce the invasiveness of rheumatoid arthritis synovial fibroblasts.

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P2860

Gene therapy in rheumatoid arthritis: how to target joint destruction?

http://www.wikidata.org/entity/Q24804867

description

1999 nî lūn-bûn

@nan

1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի հոտեմբերին հրատարակված գիտական հոդված

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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name

Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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Arthritis Research and Therapy

Arthritis Research

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Gene therapy in rheumatoid arthritis: how to target joint destruction?

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R Gay

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P2860

Activation of synovial fibroblasts in rheumatoid arthritis: lack of Expression of the tumour suppressor PTEN at sites of invasive growth and destruction

PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer

Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts

Integrins, oncogenes, and anchorage independence.

Somatic mutations in the p53 tumor suppressor gene in rheumatoid arthritis synovium.

NF-kappaB activation provides the potential link between inflammation and hyperplasia in the arthritic joint.

Constitutive expression of c-fos and c-jun, overexpression of ets-2, and reduced expression of metastasis suppressor gene nm23-H1 in rheumatoid arthritis.

Inhibition of the production and effects of interleukin-1 and tumor necrosis factor alpha in rheumatoid arthritis.

Oncogenes in rheumatoid arthritis.

NF-kappa B as a target for anti-inflammatory gene therapy: suppression of inflammatory responses in monocytic and stromal cells by stable gene transfer of I kappa B alpha cDNA.

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1

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Ulf Müller-Ladner

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rheumatoid arthritis

biomedical investigative technique

human musculoskeletal system

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