Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK - Wikidata - wikimedia - TriplyDB

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

http://www.wikidata.org/entity/Q24808691

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Lysine methylation of FOXO3 regulates oxidative stress-induced neuronal cell death A long nuclear-retained non-coding RNA regulates synaptogenesis by modulating gene expression MicroRNA-155 regulates cell survival, growth, and chemosensitivity by targeting FOXO3a in breast cancer Regenerative and fibrotic pathways in canine hepatic portosystemic shunt and portal vein hypoplasia, new models for clinical hepatocyte growth factor treatment.Protective effects and mechanisms of sirtuins in the nervous system Histone deacetylases and their role in motor neuron degeneration FOXO3a/p27kip1 expression and essential role after acute spinal cord injury in adult rat.Resveratrol induces growth arrest and apoptosis through activation of FOXO transcription factors in prostate cancer cells FoxO3a is activated and executes neuron death via Bim in response to β-amyloid Splice variants of the forkhead box protein AFX exhibit dominant negative activity and inhibit AFXalpha-mediated tumor cell apoptosis.FoxO3a governs early microglial proliferation and employs mitochondrial depolarization with caspase 3, 8, and 9 cleavage during oxidant induced apoptosis.Oxidative stress: Biomarkers and novel therapeutic pathways.New strategies for Alzheimer's disease and cognitive impairment.FOXO3a governs early and late apoptotic endothelial programs during elevated glucose through mitochondrial and caspase signaling.The "O" class: crafting clinical care with FoxO transcription factors.Involvement of FOXO transcription factors, TRAIL-FasL/Fas, and sirtuin proteins family in canine coronavirus type II-induced apoptosis.The adaptor protein SH2B1β reduces hydrogen peroxide-induced cell death in PC12 cells and hippocampal neurons.Cu,Zn-superoxide dismutase increases toxicity of mutant and zinc-deficient superoxide dismutase by enhancing protein stability.Forkhead transcription factor FOXO3a levels are increased in Huntington disease because of overactivated positive autofeedback loop.FoxO4 is the main forkhead transcriptional factor localized in the gastrointestinal tracts of pigs FoxO limits microtubule stability and is itself negatively regulated by microtubule disruption Expression of PUMA in Follicular Granulosa Cells Regulated by FoxO1 Activation During Oxidative Stress.Highly phosphorylated FOXO3A is an adverse prognostic factor in acute myeloid leukemia A Drosophila gain-of-function screen for candidate genes involved in steroid-dependent neuroendocrine cell remodeling Nitration of Hsp90 induces cell death "Sly as a FOXO": new paths with Forkhead signaling in the brain OutFOXOing disease and disability: the therapeutic potential of targeting FoxO proteins Clever cancer strategies with FoxO transcription factors.Tat-induced FOXO3a is a key mediator of apoptosis in HIV-1-infected human CD4+ T lymphocytes.A "FOXO" in sight: targeting Foxo proteins from conception to cancer.FoxO proteins: cunning concepts and considerations for the cardiovascular system.Intrinsic role of FoxO3a in the development of CD8+ T cell memory.Ectopic localization of FOXO3a protein in Lewy bodies in Lewy body dementia and Parkinson's disease Therapeutic promise and principles: metabotropic glutamate receptors A fork in the path: Developing therapeutic inroads with FoxO proteins.Regulation of Foxo-dependent transcription by post-translational modifications.The roles of FoxOs in modulation of aging by calorie restriction.TRAIL is involved in CpG ODN-mediated anti-apoptotic signals A small molecule screen in stem-cell-derived motor neurons identifies a kinase inhibitor as a candidate therapeutic for ALS.Zinc deficiency impairs the renewal of hippocampal neural stem cells in adult rats: involvement of FoxO3a activation and downstream p27(kip1) expression.

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P2860

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

http://www.wikidata.org/entity/Q24808691

description

2004 nî lūn-bûn

@nan

2004 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

@zh-hant

2004年論文

@zh-hk

2004年論文

@zh-mo

2004年論文

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2004年论文

@wuu

name

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@ast

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@en

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@nl

type

label

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@ast

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@en

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@nl

prefLabel

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@ast

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@en

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@nl

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BMC Neuroscience

P1476

Foxo3a induces motoneuron death through the Fas pathway in cooperation with JNK

@en

P2093

Brigitte Pettmann

http://www.w3.org/2001/XMLSchema#string

Christopher E Henderson

http://www.w3.org/2001/XMLSchema#string

P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Expression of the pro-apoptotic Bcl-2 family member Bim is regulated by the forkhead transcription factor FKHR-L1

Gene structure alternative splicing, and chromosomal localization of pro-apoptotic Bcl-2 relative Bim

The forkhead transcription factor FoxO regulates transcription of p27Kip1 and Bim in response to IL-2

Fas/tumor necrosis factor receptor death signaling is required for axotomy-induced death of motoneurons in vivo

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

DNA repair pathway stimulated by the forkhead transcription factor FOXO3a through the Gadd45 protein

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

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48

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scholarly article

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10.1186/1471-2202-5-48

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1

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5

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Catherine Barthélémy

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2004-11-29T00:00:00Z

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8158885

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1008457794

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15569384

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538283

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