Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors - Wikidata - wikimedia - TriplyDB

Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors

Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors

http://www.wikidata.org/entity/Q24811719

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Endocrine-disrupting chemicals: an Endocrine Society scientific statement Neurotoxicity of persistent organic pollutants: possible mode(s) of action and further considerations Polychlorinated biphenyls disturb differentiation of normal human neural progenitor cells: clue for involvement of thyroid hormone receptors.Longitudinal assessment of PCBs and chlorinated pesticides in pregnant women from Western Canada Principles and practices of neurodevelopmental assessment in children: lessons learned from the Centers for Children's Environmental Health and Disease Prevention Research Association between Several Persistent Organic Pollutants and Thyroid Hormone Levels in Cord Blood Serum and Bloodspot of the Newborn Infants of Korea Organic halogenated contaminants in mother-fetus pairs of harbor seals (Phoca vitulina richardii) from Alaska, 2000-2002.Association of Polybrominated Diphenyl Ethers (PBDEs) and Polychlorinated Biphenyls (PCBs) with Hyperthyroidism in Domestic Felines, Sentinels for Thyroid Hormone Disruption In utero and lactational exposure to a complex mixture of polychlorinated biphenyls: toxicity in pups dependent on the Cyp1a2 and Ahr genotypes.Effects of perfluoroalkyl compounds on mRNA expression levels of thyroid hormone-responsive genes in primary cultures of avian neuronal cells.PCB-related alteration of thyroid hormones and thyroid hormone receptor gene expression in free-ranging harbor seals (Phoca vitulina)Individual polychlorinated biphenyl (PCB) congeners produce tissue- and gene-specific effects on thyroid hormone signaling during development.Alteration of rat fetal cerebral cortex development after prenatal exposure to polychlorinated biphenyls Effects of age, adipose percent, and reproduction on PCB concentrations and profiles in an extreme fasting North Pacific marine mammal.Early developmental actions of endocrine disruptors on the hypothalamus, hippocampus, and cerebral cortex.Endocrine disrupting chemicals and disease susceptibility.Ontogenetic alterations in molecular and structural correlates of dendritic growth after developmental exposure to polychlorinated biphenyls.Developmental triclosan exposure decreases maternal, fetal, and early neonatal thyroxine: a dynamic and kinetic evaluation of a putative mode-of-action Polychlorinated biphenyls 105 and 118 form thyroid hormone receptor agonists after cytochrome P4501A1 activation in rat pituitary GH3 cells Mode of action: neurotoxicity induced by thyroid hormone disruption during development--hearing loss resulting from exposure to PHAHs.Long-term, intermittent, insulin-induced hypoglycemia produces marked obesity without hyperphagia or insulin resistance: a model for weight gain with intensive insulin therapy.Assessment of sex specific endocrine disrupting effects in the prenatal and pre-pubertal rodent brain.Thyroid system-disrupting chemicals: interference with thyroid hormone binding to plasma proteins and the cellular thyroid hormone signaling pathway.Relationship of thyroid hormone levels to levels of polychlorinated biphenyls, lead, p,p'- DDE, and other toxicants in Akwesasne Mohawk youth.General background on the hypothalamic-pituitary-thyroid (HPT) axis.Current and potential rodent screens and tests for thyroid toxicants.Polychlorinated biphenyls (Aroclor 1254) do not uniformly produce agonist actions on thyroid hormone responses in the developing rat brain.Endocrine disrupting polyhalogenated organic pollutants interfere with thyroid hormone signalling in the developing brain.Thyroid-disrupting chemicals: interpreting upstream biomarkers of adverse outcomes.Receptor interactions by polybrominated diphenyl ethers versus polychlorinated biphenyls: a theoretical Structure-activity assessment.Chemical contamination and the thyroid.Exposure to endocrine disrupting chemicals and neurodevelopmental alterations.Effects of in utero exposure to 2,2',4,4',5,5'-hexachlorobiphenyl (PCB 153) on somatic growth and endocrine status in rat offspring.Early exposure to Aroclor 1254 in vivo disrupts the functional synaptic development of newborn hippocampal granule cells.Transplacental Transfer of Polychlorinated Biphenyls, Polybrominated Diphenylethers, and Organochlorine Pesticides in Ringed Seals (Pusa hispida).Comparative Analysis of Zearalenone Effects on Thyroid Receptor Alpha (TRα) and Beta (TRβ) Expression in Rat Primary Cerebellar Cell Cultures.

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Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors

http://www.wikidata.org/entity/Q24811719

description

2004 nî lūn-bûn

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2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2004 թվականի ապրիլին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Environmental Health Perspectives

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Polychlorinated biphenyls (PCB ...... d to thyroid hormone receptors

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Kelly J Gauger

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Koichi Haraguchi

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Larry W Robertson

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R Thomas Zoeller

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Ruby Bansal

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Yoshihisa Kato

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P2860

The c-erb-A gene encodes a thyroid hormone receptor

Characterization of a thyroid hormone receptor expressed in human kidney and other tissues

Transplacental thyroxine and fetal brain development

Effects of PCB exposure on neuropsychological function in children

Assessment of pre- and postnatal exposure to polychlorinated biphenyls: lessons from the Inuit Cohort Study.

The nuclear receptor superfamily: the second decade

Tissue-specific actions of thyroid hormone: insights from animal models

The partial agonist activity of antagonist-occupied steroid receptors is controlled by a novel hinge domain-binding coactivator L7/SPA and the corepressors N-CoR or SMRT

The c-erb-A protein is a high-affinity receptor for thyroid hormone

Association between serum concentrations of hexachlorobenzene and polychlorobiphenyls with thyroid hormone and liver enzymes in a sample of the general population

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10.1289/EHP.6672

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5

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112

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Hans-Joachim Lehmler

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8637767

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15064154

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1241914

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