Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference - Wikidata - wikimedia - TriplyDB

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

http://www.wikidata.org/entity/Q24811874

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Pol II-expressed shRNA knocks down Sod2 gene expression and causes phenotypes of the gene knockout in mice.Fibroblast growth factor signaling in skeletal development and disease In vitro and in vivo approaches to study osteocyte biology A non-specific effect associated with conditional transgene expression based on Cre-loxP strategy in mice Construction of permanently inducible miRNA-based expression vectors using site-specific recombinases Essential roles of BCCIP in mouse embryonic development and structural stability of chromosomes A construct with fluorescent indicators for conditional expression of miRNA.Inducible transgenic rat model for diabetes mellitus based on shRNA-mediated gene knockdown.Synthetic strategies for studying embryonic development.Granulin epithelin precursor: a bone morphogenic protein 2-inducible growth factor that activates Erk1/2 signaling and JunB transcription factor in chondrogenesis.Accelerating cancer modeling with RNAi and nongermline genetically engineered mouse models.Genetically engineered mouse models for drug development and preclinical trials.Extracellular matrix degradation and remodeling in development and disease GEP, a local growth factor, is critical for odontogenesis and amelogenesis.Disrupted junctional membrane complexes and hyperactive ryanodine receptors after acute junctophilin knockdown in mice Conditional transgenic mouse models: from the basics to genome-wide sets of knockouts and current studies of tissue regeneration.An RNAi-based approach to down-regulate a gene family in vivo.shRNA off-target effects in vivo: impaired endogenous siRNA expression and spermatogenic defects.RNAi-based conditional gene knockdown in mice using a U6 promoter driven vector Role of FGF/FGFR signaling in skeletal development and homeostasis: learning from mouse models.Reversible gene knockdown in mice using a tight, inducible shRNA expression system.Conditional brain-specific knockdown of MAPK using Cre/loxP regulated RNA interference.Tissue-specific and reversible RNA interference in transgenic mice.Approaches for chemically synthesized siRNA and vector-mediated RNAi.Merging fields: stem cells in neurogenesis, transplantation, and disease modeling.Tetracycline-inducible expression systems: new strategies and practices in the transgenic mouse modeling.FGF signaling in the osteoprogenitor lineage non-autonomously regulates postnatal chondrocyte proliferation and skeletal growth.The apical ectodermal ridge is a timer for generating distal limb progenitors.Loss of androgen receptor promotes adipogenesis but suppresses osteogenesis in bone marrow stromal cells.A significant increase of RNAi efficiency in human cells by the CMV enhancer with a tRNAlys promoter.Heritable and inducible gene knockdown in astrocytes or neurons in vivo by a combined lentiviral and RNAi approach.Role of FGFs/FGFRs in skeletal development and bone regeneration.LADD syndrome with glaucoma is caused by a novel gene Gene-targeted embryonic stem cells: real-time PCR assay for estimation of the number of neomycin selection cassettes.Mutations in different components of FGF signaling in LADD syndrome.Silencing of the Pink1 gene expression by conditional RNAi does not induce dopaminergic neuron death in mice.The impact of preclinical drug safety on R&D productivity.RNA interference and inhibition of MEK-ERK signaling prevent abnormal skeletal phenotypes in a mouse model of craniosynostosis

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P2860

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

http://www.wikidata.org/entity/Q24811874

description

2005 nî lūn-bûn

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2005 թուականին հրատարակուած գիտական յօդուած

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2005 թվականին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@ast

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@en

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@nl

type

label

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@ast

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@en

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@nl

prefLabel

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@ast

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@en

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@nl

P1433

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P1433

Nucleic Acids Research

P1476

Conditional knockdown of Fgfr2 in mice using Cre-LoxP induced RNA interference

@en

P2093

Chu-Xia Deng

http://www.w3.org/2001/XMLSchema#string

Cuiling Li

http://www.w3.org/2001/XMLSchema#string

Rui-Hong Wang

http://www.w3.org/2001/XMLSchema#string

Vivek Shukla

http://www.w3.org/2001/XMLSchema#string

P2860

The knockout mouse project

Fibroblast growth factor receptors from liver vary in three structural domains

Fibroblast growth factors

Cre-lox-regulated conditional RNA interference from transgenes

RNA interference is mediated by 21- and 22-nucleotide RNAs

Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells

Cre recombinase: the universal reagent for genome tailoring

Talking about a revolution: The impact of site-specific recombinases on genetic analyses in mice

Roles of FGF receptors in mammalian development and congenital diseases

Altering the genome by homologous recombination

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e102

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scholarly article

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10.1093/NAR/GNI100

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11

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33

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7755807

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15987787

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1159121

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